George Perry
Impact in
- Biological Psychiatry top 0.02%
- Tryptophan and brain disorders
- Physiology top 0.01%
- Alzheimer's disease research and treatments
Papers in
- Physiology 503
- Alzheimer's disease research and treatments 468
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- Mitochondrial Function and Pathology 109
- Prion Diseases and Protein Misfolding 59
- Co-authors
- Mark A. Smith (251 shared papers)Xiongwei Zhu (237 shared papers)Akihiko Nunomura (97 shared papers)Lawrence M. Sayre (35 shared papers)Hyoung‐gon Lee (107 shared papers)Rudy J. Castellani (113 shared papers)Xinglong Wang (49 shared papers)Paula I. Moreira (76 shared papers)
- Journals
- Journal of Alzheimer s Disease (76 papers)Journal of Neuropathology & Experimental Neurology (34 papers)Journal of Neurochemistry (31 papers)Brain Research (29 papers)Current Alzheimer Research (20 papers)
- Partner nations
- United StatesJapanPortugal
In The Last Decade
George Perry
888 papers receiving 67.4k citations
George Perry's Hit Papers
Peers
Comparison fields: 5 of 208
- Biological Psychiatry 4.1k
- Physiology 36.7k
- Neurology 10.4k
- Pharmacology 10.1k
- Aging 915
Countries citing papers authored by George Perry
This map shows the geographic impact of George Perry's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by George Perry with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites George Perry more than expected).
Fields of papers citing papers by George Perry
This network shows the impact of papers produced by George Perry. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by George Perry. The network helps show where George Perry may publish in the future.
Co-authors
The 25 scholars most cited alongside George Perry, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 905 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | Oxidative Damage Is the Earliest Event in Alzheimer Disease Hit paper breakdown → | 2001 | 1522 |
| 2 | Mitochondrial Abnormalities in Alzheimer's Disease Hit paper breakdown → | 2001 | 1120 |
| 3 | Iron accumulation in Alzheimer disease is a source of redox-generated free radicals Hit paper breakdown → | 1997 | 1116 |
| 4 | The Amyloid-β Pathway in Alzheimer’s Disease Hit paper breakdown → | 2021 | 1099 |
| 5 | Oxidative stress and mitochondrial dysfunction in Alzheimer's disease Hit paper breakdown → | 2013 | 1064 |
| 6 | Widespread Peroxynitrite-Mediated Damage in Alzheimer’s Disease Hit paper breakdown → | 1997 | 1044 |
| 7 | Impaired Balance of Mitochondrial Fission and Fusion in Alzheimer's Disease Hit paper breakdown → | 2009 | 1005 |
| 8 | 4‐Hydroxynonenal‐Derived Advanced Lipid Peroxidation End Products Are Increased in Alzheimer's Disease Hit paper breakdown → | 1997 | 843 |
| 9 | Mitochondria dysfunction in the pathogenesis of Alzheimer’s disease: recent advances Hit paper breakdown → | 2020 | 832 |
| 10 | Oxidative stress in Alzheimer’s disease Hit paper breakdown → | 2000 | 724 |
| 11 | Oxidative Stress and Neurotoxicity Hit paper breakdown → | 2007 | 690 |
| 12 | RNA Oxidation Is a Prominent Feature of Vulnerable Neurons in Alzheimer’s Disease Hit paper breakdown → | 1999 | 614 |
| 13 | Advanced Maillard reaction end products are associated with Alzheimer disease pathology. Hit paper breakdown → | 1994 | 608 |
| 14 | Mitochondrial dysfunction is a trigger of Alzheimer's disease pathophysiology Hit paper breakdown → | 2009 | 585 |
| 15 | Parkinson's Disease Is Associated with Oxidative Damage to Cytoplasmic DNA and RNA in Substantia Nigra Neurons Hit paper breakdown → | 1999 | 513 |
| 16 | Ubiquitin is detected in neurofibrillary tangles and senile plaque neurites of Alzheimer disease brains. Hit paper breakdown → | 1987 | 512 |
| 17 | Chemistry and Biochemistry of Oxidative Stress in Neurodegenerative Disease Hit paper breakdown → | 2001 | 508 |
| 18 | 2006 | 478 | |
| 19 | 2003 | 477 | |
| 20 | 1998 | 469 |
About George Perry
George Perry is a scholar working on Physiology, Molecular Biology, Pharmacology, Cellular and Molecular Neuroscience and Neurology, having authored 905 papers that have together received 69.0k indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (468 papers), Cholinesterase and Neurodegenerative Diseases (123 papers), Mitochondrial Function and Pathology (109 papers), Trace Elements in Health (67 papers), Neuroscience and Neuropharmacology Research (65 papers), Prion Diseases and Protein Misfolding (59 papers), Parkinson's Disease Mechanisms and Treatments (55 papers) and Computational Drug Discovery Methods (45 papers). The work is most often cited by research in Biological Psychiatry (4.1k citations), Physiology (36.7k citations), Neurology (10.4k citations), Pharmacology (10.1k citations) and Aging (915 citations). George Perry has collaborated with scholars based in United States, Japan and Portugal. Frequent co-authors include Mark A. Smith, Xiongwei Zhu, Akihiko Nunomura, Lawrence M. Sayre, Hyoung‐gon Lee, Rudy J. Castellani, Xinglong Wang, Paula I. Moreira, Peggy L.R. Harris and Sandra L. Siedlak. Their work appears in journals such as Journal of Alzheimer s Disease, Journal of Neuropathology & Experimental Neurology, Journal of Neurochemistry, Brain Research and Current Alzheimer Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.