Mark A. Smith
Impact in
- Biological Psychiatry top 0.01%
- Tryptophan and brain disorders
- Physiology top 0.01%
- Alzheimer's disease research and treatments
Papers in
- Physiology 351
- Alzheimer's disease research and treatments 314
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- Mitochondrial Function and Pathology 79
- Co-authors
- George Perry (251 shared papers)Xiongwei Zhu (226 shared papers)Akihiko Nunomura (88 shared papers)Lawrence M. Sayre (33 shared papers)Hyoung‐gon Lee (119 shared papers)Rudy J. Castellani (94 shared papers)Peggy L.R. Harris (46 shared papers)Paula I. Moreira (62 shared papers)
- Journals
- Journal of Alzheimer s Disease (37 papers)Journal of Neurochemistry (31 papers)Brain Research (22 papers)Annals of the New York Academy of Sciences (15 papers)Free Radical Biology and Medicine (15 papers)
- Partner nations
- United StatesJapanUnited Kingdom
In The Last Decade
Mark A. Smith
759 papers receiving 65.0k citations
Mark A. Smith's Hit Papers
Peers
Comparison fields: 5 of 218
- Biological Psychiatry 3.9k
- Physiology 28.6k
- Neurology 7.5k
- Behavioral Neuroscience 2.5k
- Pharmacology 8.5k
Countries citing papers authored by Mark A. Smith
This map shows the geographic impact of Mark A. Smith's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark A. Smith with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark A. Smith more than expected).
Fields of papers citing papers by Mark A. Smith
This network shows the impact of papers produced by Mark A. Smith. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark A. Smith. The network helps show where Mark A. Smith may publish in the future.
Co-authors
The 25 scholars most cited alongside Mark A. Smith, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 767 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | Towards a Better Understanding of Context and Context-Awareness Hit paper breakdown → | 1999 | 1993 |
| 2 | Iron accumulation in Alzheimer disease is a source of redox-generated free radicals Hit paper breakdown → | 1997 | 1183 |
| 3 | Mitochondrial Abnormalities in Alzheimer's Disease Hit paper breakdown → | 2001 | 1181 |
| 4 | Impaired Balance of Mitochondrial Fission and Fusion in Alzheimer's Disease Hit paper breakdown → | 2009 | 1058 |
| 5 | Cardioprotective Effect of Diazoxide and Its Interaction With Mitochondrial ATP-Sensitive K+Channels Hit paper breakdown → | 1997 | 877 |
| 6 | Oxidative stress in Alzheimer’s disease Hit paper breakdown → | 2000 | 756 |
| 7 | Oxidative Stress and Neurotoxicity Hit paper breakdown → | 2007 | 733 |
| 8 | Advanced Maillard reaction end products are associated with Alzheimer disease pathology. Hit paper breakdown → | 1994 | 655 |
| 9 | RNA Oxidation Is a Prominent Feature of Vulnerable Neurons in Alzheimer’s Disease Hit paper breakdown → | 1999 | 646 |
| 10 | Mitochondrial dysfunction is a trigger of Alzheimer's disease pathophysiology Hit paper breakdown → | 2009 | 609 |
| 11 | Parkinson's Disease Is Associated with Oxidative Damage to Cytoplasmic DNA and RNA in Substantia Nigra Neurons Hit paper breakdown → | 1999 | 552 |
| 12 | Leptin inhibits hypothalamic neurons by activation of ATP-sensitive potassium channels Hit paper breakdown → | 1997 | 548 |
| 13 | Chemistry and Biochemistry of Oxidative Stress in Neurodegenerative Disease Hit paper breakdown → | 2001 | 538 |
| 14 | Involvement of Oxidative Stress in Alzheimer Disease Hit paper breakdown → | 2006 | 512 |
| 15 | Glycated tau protein in Alzheimer disease: a mechanism for induction of oxidant stress. Hit paper breakdown → | 1994 | 508 |
| 16 | Metal Binding and Oxidation of Amyloid-β within Isolated Senile Plaque Cores: Raman Microscopic Evidence Hit paper breakdown → | 2003 | 504 |
| 17 | 1998 | 498 | |
| 18 | 2000 | 487 | |
| 19 | 2010 | 438 | |
| 20 | 2007 | 433 |
About Mark A. Smith
Mark A. Smith is a scholar working on Physiology, Molecular Biology, Cellular and Molecular Neuroscience, Pharmacology and Nutrition and Dietetics, having authored 767 papers that have together received 67.2k indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (314 papers), Cholinesterase and Neurodegenerative Diseases (94 papers), Mitochondrial Function and Pathology (79 papers), Neuroscience and Neuropharmacology Research (70 papers), Trace Elements in Health (51 papers), Tryptophan and brain disorders (40 papers), Parkinson's Disease Mechanisms and Treatments (37 papers) and Stress Responses and Cortisol (36 papers). The work is most often cited by research in Biological Psychiatry (3.9k citations), Physiology (28.6k citations), Neurology (7.5k citations), Behavioral Neuroscience (2.5k citations) and Pharmacology (8.5k citations). Mark A. Smith has collaborated with scholars based in United States, Japan and United Kingdom. Frequent co-authors include George Perry, Xiongwei Zhu, Akihiko Nunomura, Lawrence M. Sayre, Hyoung‐gon Lee, Rudy J. Castellani, Peggy L.R. Harris, Paula I. Moreira, Arun K. Raina and Gemma Casadesús. Their work appears in journals such as Journal of Alzheimer s Disease, Journal of Neurochemistry, Brain Research, Annals of the New York Academy of Sciences and Free Radical Biology and Medicine.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.