John Hardy
Impact in
- Neurology top 0.01%
- Parkinson's Disease Mechanisms and Treatments
- Neuroinflammation and Neurodegeneration Mechanisms
- Neurological diseases and metabolism
- Physiology top 0.01%
- Alzheimer's disease research and treatments
Papers in
-
- Prion Diseases and Protein Misfolding 76
- Bioinformatics and Genomic Networks 74
- Physiology 339
- Alzheimer's disease research and treatments 296
- Co-authors
- Dennis J. Selkoe (3 shared papers)Gerald A. Higgins (1 shared paper)Andrew Singleton (104 shared papers)David Allsop (5 shared papers)Andrew J. Lees (40 shared papers)Tamás Révész (32 shared papers)Jonathan M. Schott (15 shared papers)Henry Houlden (75 shared papers)
- Journals
- Neurobiology of Aging (53 papers)Movement Disorders (50 papers)Alzheimer s & Dementia (27 papers)Annals of Neurology (27 papers)Journal of Neurochemistry (26 papers)
- Partner nations
- United KingdomUnited StatesSweden
In The Last Decade
John Hardy
883 papers receiving 99.9k citations
John Hardy's Hit Papers
Peers
Comparison fields: 5 of 220
- Neurology 17.7k
- Physiology 51.4k
- Neurology 22.2k
- Biological Psychiatry 3.4k
- Cellular and Molecular Neuroscience 21.4k
Countries citing papers authored by John Hardy
This map shows the geographic impact of John Hardy's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John Hardy with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John Hardy more than expected).
Fields of papers citing papers by John Hardy
This network shows the impact of papers produced by John Hardy. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John Hardy. The network helps show where John Hardy may publish in the future.
Co-authors
The 25 scholars most cited alongside John Hardy, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 903 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | The Amyloid Hypothesis of Alzheimer's Disease: Progress and Problems on the Road to Therapeutics Hit paper breakdown → | 2002 | 11531 |
| 2 | Alzheimer's Disease: The Amyloid Cascade Hypothesis Hit paper breakdown → | 1992 | 5901 |
| 3 | The amyloid hypothesis of Alzheimer's disease at 25 years Hit paper breakdown → | 2016 | 4599 |
| 4 | Segregation of a missense mutation in the amyloid precursor protein gene with familial Alzheimer's disease Hit paper breakdown → | 1991 | 3699 |
| 5 | Amyloid deposition as the central event in the aetiology of Alzheimer's disease Hit paper breakdown → | 1991 | 2039 |
| 6 | Alzheimer's disease Hit paper breakdown → | 2017 | 1944 |
| 7 | Parkinson's disease Hit paper breakdown → | 2009 | 1633 |
| 8 | Aβ peptide vaccination prevents memory loss in an animal model of Alzheimer's disease Hit paper breakdown → | 2000 | 1310 |
| 9 | Enhanced Neurofibrillary Degeneration in Transgenic Mice Expressing Mutant Tau and APP Hit paper breakdown → | 2001 | 1292 |
| 10 | Increased amyloid-β42(43) in brains of mice expressing mutant presenilin 1 Hit paper breakdown → | 1996 | 1253 |
| 11 | The Amyloid-β Pathway in Alzheimer’s Disease Hit paper breakdown → | 2021 | 1235 |
| 12 | Amyloid, the presenilins and Alzheimer's disease Hit paper breakdown → | 1997 | 1183 |
| 13 | Accelerated Alzheimer-type phenotype in transgenic mice carrying both mutant amyloid precursor protein and presenilin 1 transgenes Hit paper breakdown → | 1998 | 1151 |
| 14 | Neurofibrillary tangles, amyotrophy and progressive motor disturbance in mice expressing mutant (P301L) tau protein Hit paper breakdown → | 2000 | 1112 |
| 15 | Toxic Proteins in Neurodegenerative Disease Hit paper breakdown → | 2002 | 1040 |
| 16 | Early-onset Alzheimer's disease caused by mutations at codon 717 of the β-amyloid precursor protein gene Hit paper breakdown → | 1991 | 1011 |
| 17 | Endoproteolysis of Presenilin 1 and Accumulation of Processed Derivatives In Vivo Hit paper breakdown → | 1996 | 889 |
| 18 | Functional brain abnormalities in young adults at genetic risk for late-onset Alzheimer's dementia Hit paper breakdown → | 2003 | 830 |
| 19 | Neuropathological assessment of Parkinson's disease: refining the diagnostic criteria Hit paper breakdown → | 2009 | 734 |
| 20 | Imputation of sequence variants for identification of genetic risks for Parkinson's disease: a meta-analysis of genome-wide association studies Hit paper breakdown → | 2011 | 695 |
About John Hardy
John Hardy is a scholar working on Molecular Biology, Physiology, Neurology, Cellular and Molecular Neuroscience and Neurology, having authored 903 papers that have together received 103.0k indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (296 papers), Parkinson's Disease Mechanisms and Treatments (190 papers), Neurological diseases and metabolism (118 papers), Nuclear Receptors and Signaling (85 papers), Prion Diseases and Protein Misfolding (76 papers), Bioinformatics and Genomic Networks (74 papers), Genetic Associations and Epidemiology (56 papers) and Amyotrophic Lateral Sclerosis Research (50 papers). The work is most often cited by research in Neurology (17.7k citations), Physiology (51.4k citations), Neurology (22.2k citations), Biological Psychiatry (3.4k citations) and Cellular and Molecular Neuroscience (21.4k citations). John Hardy has collaborated with scholars based in United Kingdom, United States and Sweden. Frequent co-authors include Dennis J. Selkoe, Gerald A. Higgins, Andrew Singleton, David Allsop, Andrew J. Lees, Tamás Révész, Jonathan M. Schott, Henry Houlden, Chris Lane and Karen Duff. Their work appears in journals such as Neurobiology of Aging, Movement Disorders, Alzheimer s & Dementia, Annals of Neurology and Journal of Neurochemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.