Michael K. Lee

26.0k citations
155 papers · 20.2k · 11 hit papers · h-index 61

Impact in

  • Neurology top 0.02%
    • Parkinson's Disease Mechanisms and Treatments
    • Amyotrophic Lateral Sclerosis Research
    • Neurological diseases and metabolism
  • Physiology top 0.05%
    • Alzheimer's disease research and treatments

Papers in

    • Parkinson's Disease Mechanisms and Treatments 43
    • Neurological diseases and metabolism 18
    • Amyotrophic Lateral Sclerosis Research 16
    • Neurological disorders and treatments 12
    • Prion Diseases and Protein Misfolding 13

Michael K. Lee

144 papers receiving 19.8k citations

Michael K. Lee's Hit Papers

Increased Expression of α-Synuclein Reduces Neurotransmitter Release by Inhibiting Synaptic Vesicle Reclustering after Endocytosis 2010 · 842 citations
8420+12+24Years since publication4008001.2k

Peers

Michael K. Lee
Comparison fields: 5 of 158
  • Neurology 7.8k
  • Neurology 3.0k
  • Physiology 7.3k
  • Cellular and Molecular Neuroscience 5.4k
  • Genetics 1.9k
Replace Philip C. Wong with:
Philip C. Wong United States
David Borchelt United States
Robert J. Ferrante United States
Masato Hasegawa Japan
Jada Lewis United States
Lee J. Martin United States
Mark Cookson United States
Henry Houlden United Kingdom
Vernice Jackson‐Lewis United States
Edward Rockenstein United States
Michael K. Lee relative to Philip C. Wong United States Philip C. Wong's profile →
Citations per field
00.5×1.5×1.8×
Philip C. Wong · 1×
Citations per year

Countries citing papers authored by Michael K. Lee

Since Specialization
Citations

This map shows the geographic impact of Michael K. Lee's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael K. Lee with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael K. Lee more than expected).

Fields of papers citing papers by Michael K. Lee

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael K. Lee. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael K. Lee. The network helps show where Michael K. Lee may publish in the future.

Co-authors

The 25 scholars most cited alongside Michael K. Lee, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Michael K. Lee Line = papers co-authored together Michael K. Lee links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 155 papers — load more, or switch the sort, to bring in the rest.

#Work
1
Mutant presenilins specifically elevate the levels of the 42 residue β-amyloid peptide in vivo: evidence for augmentation of a 42-specific γ secretase
Hit paper breakdown →
20031274
2
Familial Alzheimer's Disease–Linked Presenilin 1 Variants Elevate Aβ1–42/1–40 Ratio In Vitro and In Vivo
Hit paper breakdown →
19961227
3
An adverse property of a familial ALS-linked SOD1 mutation causes motor neuron disease characterized by vacuolar degeneration of mitochondria
Hit paper breakdown →
19951182
4
ALS-Linked SOD1 Mutant G85R Mediates Damage to Astrocytes and Promotes Rapidly Progressive Disease with SOD1-Containing Inclusions
Hit paper breakdown →
19971087
5
Endoproteolysis of Presenilin 1 and Accumulation of Processed Derivatives In Vivo
Hit paper breakdown →
1996863
6
Increased Expression of α-Synuclein Reduces Neurotransmitter Release by Inhibiting Synaptic Vesicle Reclustering after Endocytosis
Hit paper breakdown →
2010842
7
Accelerated Amyloid Deposition in the Brains of Transgenic Mice Coexpressing Mutant Presenilin 1 and Amyloid Precursor Proteins
Hit paper breakdown →
1997832
8
Human α-synuclein-harboring familial Parkinson's disease-linked Ala-53 → Thr mutation causes neurodegenerative disease with α-synuclein aggregation in transgenic mice
Hit paper breakdown →
2002660
9
Altered Reactivity of Superoxide Dismutase in Familial Amyotrophic Lateral Sclerosis
Hit paper breakdown →
1996586
10
The expression and posttranslational modification of a neuron‐specific β‐tubulin isotype during chick embryogenesis
Hit paper breakdown →
1990540
11
Parkinson's Disease α-Synuclein Transgenic Mice Develop Neuronal Mitochondrial Degeneration and Cell Death
Hit paper breakdown →
2006535
12 1994497
13 1999448
14 2005374
15 1996373
16 2007355
17 1993323
18 2012318
19 1996302
20 1996300

About Michael K. Lee

Michael K. Lee is a scholar working on Neurology, Molecular Biology, Cellular and Molecular Neuroscience, Physiology and Neurology, having authored 155 papers that have together received 20.2k indexed citations. Recurring topics across this work include Parkinson's Disease Mechanisms and Treatments (43 papers), Alzheimer's disease research and treatments (35 papers), Neurological diseases and metabolism (18 papers), Amyotrophic Lateral Sclerosis Research (16 papers), Nuclear Receptors and Signaling (16 papers), Nerve injury and regeneration (15 papers), Prion Diseases and Protein Misfolding (13 papers) and Neurological disorders and treatments (12 papers). The work is most often cited by research in Neurology (7.8k citations), Neurology (3.0k citations), Physiology (7.3k citations), Cellular and Molecular Neuroscience (5.4k citations) and Genetics (1.9k citations). Michael K. Lee has collaborated with scholars based in United States, South Korea and Germany. Frequent co-authors include Don W. Cleveland, Donald L. Price, David Borchelt, Neal G. Copeland, Sangram S. Sisodia, Nancy A. Jenkins, Juan C. Troncoso, Philip C. Wong, Ted M. Dawson and Hilda H. Slunt. Their work appears in journals such as Proceedings of the National Academy of Sciences, Neuron, Journal of Neuroscience, Human Molecular Genetics and Neurobiology of Disease.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact