Michaël Maes
Impact in
- Biological Psychiatry top 0.01%
- Tryptophan and brain disorders
- Behavioral Neuroscience top 0.01%
- Stress Responses and Cortisol
Papers in
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- Tryptophan and brain disorders 444
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- Stress Responses and Cortisol 259
- Co-authors
- Michael Berk (123 shared papers)Marta Kubera (75 shared papers)Eugène Bosmans (60 shared papers)George Anderson (63 shared papers)Herbert Y. Meltzer (51 shared papers)Gerwyn Morris (61 shared papers)Simon Scharpé (73 shared papers)Günter Kenis (42 shared papers)
In The Last Decade
Michaël Maes
1.0k papers receiving 61.6k citations
Michaël Maes's Hit Papers
Peers
Comparison fields: 5 of 199
- Biological Psychiatry 25.4k
- Behavioral Neuroscience 14.8k
- Psychiatry and Mental health 10.8k
- Neurology 5.1k
- Endocrine and Autonomic Systems 1.9k
Countries citing papers authored by Michaël Maes
This map shows the geographic impact of Michaël Maes's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michaël Maes with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michaël Maes more than expected).
Fields of papers citing papers by Michaël Maes
This network shows the impact of papers produced by Michaël Maes. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michaël Maes. The network helps show where Michaël Maes may publish in the future.
Co-authors
The 25 scholars most cited alongside Michaël Maes, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 1.0k papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | Peripheral cytokine and chemokine alterations in depression: a meta‐analysis of 82 studies Hit paper breakdown → | 2017 | 1099 |
| 2 | So depression is an inflammatory disease, but where does the inflammation come from? Hit paper breakdown → | 2013 | 1062 |
| 3 | Cytokines and major depression Hit paper breakdown → | 2005 | 954 |
| 4 | Pathways underlying neuroprogression in bipolar disorder: Focus on inflammation, oxidative stress and neurotrophic factors Hit paper breakdown → | 2010 | 940 |
| 5 | A review on the oxidative and nitrosative stress (O&NS) pathways in major depression and their possible contribution to the (neuro)degenerative processes in that illness Hit paper breakdown → | 2010 | 938 |
| 6 | Evidence for an immune response in major depression: A review and hypothesis Hit paper breakdown → | 1995 | 803 |
| 7 | The inflammatory & neurodegenerative (I&ND) hypothesis of depression: leads for future research and new drug developments in depression Hit paper breakdown → | 2008 | 724 |
| 8 | Mechanistic explanations how cell-mediated immune activation, inflammation and oxidative and nitrosative stress pathways and their sequels and concomitants play a role in the pathophysiology of unipolar depression Hit paper breakdown → | 2011 | 677 |
| 9 | THE EFFECTS OF PSYCHOLOGICAL STRESS ON HUMANS: INCREASED PRODUCTION OF PRO-INFLAMMATORY CYTOKINES AND Th1-LIKE RESPONSE IN STRESS-INDUCED ANXIETY Hit paper breakdown → | 1998 | 634 |
| 10 | INCREASED SERUM IL-6 AND IL-1 RECEPTOR ANTAGONIST CONCENTRATIONS IN MAJOR DEPRESSION AND TREATMENT RESISTANT DEPRESSION Hit paper breakdown → | 1997 | 573 |
| 11 | The new ‘5-HT’ hypothesis of depression: Cell-mediated immune activation induces indoleamine 2,3-dioxygenase, which leads to lower plasma tryptophan and an increased synthesis of detrimental tryptophan catabolites (TRYCATs), both of which contribute to the onset of depression Hit paper breakdown → | 2010 | 567 |
| 12 | Increased plasma concentrations of interleukin-6, soluble interleukin-6, soluble interleukin-2 and transferrin receptor in major depression Hit paper breakdown → | 1995 | 518 |
| 13 | Major Depression and Activation of The Inflammatory Response System Hit paper breakdown → | 1999 | 507 |
| 14 | 2012 | 490 | |
| 15 | The gut-brain barrier in major depression: intestinal mucosal dysfunction with an increased translocation of LPS from gram negative enterobacteria (leaky gut) plays a role in the inflammatory pathophysiology of depression. | 2008 | 481 |
| 16 | 2010 | 446 | |
| 17 | 2012 | 421 | |
| 18 | 2013 | 393 | |
| 19 | 2002 | 380 | |
| 20 | 2004 | 379 |
About Michaël Maes
Michaël Maes is a scholar working on Biological Psychiatry, Behavioral Neuroscience, Psychiatry and Mental health, Molecular Biology and Pharmacology, having authored 1.0k papers that have together received 63.6k indexed citations. Recurring topics across this work include Tryptophan and brain disorders (444 papers), Stress Responses and Cortisol (259 papers), Fibromyalgia and Chronic Fatigue Syndrome Research (92 papers), Bipolar Disorder and Treatment (78 papers), Treatment of Major Depression (54 papers), Neuroinflammation and Neurodegeneration Mechanisms (42 papers), Mental Health Research Topics (38 papers) and Neuroendocrine regulation and behavior (37 papers). The work is most often cited by research in Biological Psychiatry (25.4k citations), Behavioral Neuroscience (14.8k citations), Psychiatry and Mental health (10.8k citations), Neurology (5.1k citations) and Endocrine and Autonomic Systems (1.9k citations). Michaël Maes has collaborated with scholars based in Thailand, Australia and Belgium. Frequent co-authors include Michael Berk, Marta Kubera, Eugène Bosmans, George Anderson, Herbert Y. Meltzer, Gerwyn Morris, Simon Scharpé, Günter Kenis, Marieke Wichers and André F. Carvalho. Their work appears in journals such as Journal of Affective Disorders, Molecular Neurobiology, Psychiatry Research, Progress in Neuro-Psychopharmacology and Biological Psychiatry and Acta Psychiatrica Scandinavica.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.