Mark R. Morris

4.0k citations
57 papers · 2.6k · h-index 31

Impact in

Papers in

Mark R. Morris

55 papers receiving 2.6k citations

Peers

Mark R. Morris
Comparison fields: 5 of 123
  • Cancer Research 587
  • Pulmonary and Respiratory Medicine 783
  • Molecular Biology 1.5k
  • Oncology 240
  • Biomaterials 97
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Citations per year

Countries citing papers authored by Mark R. Morris

Since Specialization
Citations

This map shows the geographic impact of Mark R. Morris's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark R. Morris with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark R. Morris more than expected).

Fields of papers citing papers by Mark R. Morris

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mark R. Morris. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark R. Morris. The network helps show where Mark R. Morris may publish in the future.

Co-authors

The 25 scholars most cited alongside Mark R. Morris, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Mark R. Morris Line = papers co-authored together Mark R. Morris links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 57 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2008248
2 2010166
3 2010134
4
Identification of cyclin D1 and other novel targets for the von Hippel-Lindau tumor suppressor gene by expression array analysis and investigation of cyclin D1 genotype as a modifier in von Hippel-Lindau disease.
2002125
5 2001104
6 200597
7 201897
8 201696
9 201395
10 200387
11 200883
12 201578
13 201373
14 200068
15 200965
16 200860
17 201057
18 200456
19 201550
20
Mutation analysis of hypoxia-inducible factors HIF1A and HIF2A in renal cell carcinoma.
200949

About Mark R. Morris

Mark R. Morris is a scholar working on Molecular Biology, Pulmonary and Respiratory Medicine, Cancer Research, Surgery and Immunology, having authored 57 papers that have together received 2.6k indexed citations. Recurring topics across this work include Epigenetics and DNA Methylation (16 papers), Cancer, Hypoxia, and Metabolism (11 papers), Renal cell carcinoma treatment (9 papers), Renal and related cancers (8 papers), Cystic Fibrosis Research Advances (6 papers), RNA modifications and cancer (6 papers), Cancer-related gene regulation (5 papers) and Neutrophil, Myeloperoxidase and Oxidative Mechanisms (4 papers). The work is most often cited by research in Cancer Research (587 citations), Pulmonary and Respiratory Medicine (783 citations), Molecular Biology (1.5k citations), Oncology (240 citations) and Biomaterials (97 citations). Mark R. Morris has collaborated with scholars based in United Kingdom, France and United States. Frequent co-authors include Farida Latif, Eamonn R. Maher, Dean Gentle, Christopher J. Ricketts, Dewi Astuti, Takeshi Kishida, Masahiro Yao, Emma R. Woodward, Noel W. Clarke and Maurice B. Hallett. Their work appears in journals such as Oncogene, Neuro-Oncology, Genes Chromosomes and Cancer, Epigenetics and British Journal of Cancer.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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