Jacqueline C. Mitchell

3.4k citations
42 papers · 2.4k · 1 hit paper · h-index 22

Impact in

  • Neurology top 0.5%
    • Amyotrophic Lateral Sclerosis Research
    • Parkinson's Disease Mechanisms and Treatments
  • Genetics top 1%
    • Neurogenetic and Muscular Disorders Research

Papers in

Co-authors
Christopher E. Shaw (12 shared papers)Christopher C.J. Miller (13 shared papers)Caroline Vance (9 shared papers)Boris Rogelj (9 shared papers)Emma L. Scotter (6 shared papers)Gema Vizcay‐Barrena (3 shared papers)Kurt J. De Vos (3 shared papers)Sébastien Paillusson (3 shared papers)
Journals
Human Molecular Genetics (4 papers)Acta Neuropathologica Communications (3 papers)Journal of Neuroendocrinology (2 papers)Aging Cell (2 papers)Brain (2 papers)
Partner nations
United KingdomUnited StatesIreland

In The Last Decade

Jacqueline C. Mitchell

40 papers receiving 2.4k citations

Jacqueline C. Mitchell's Hit Papers

ER–mitochondria associations are regulated by the VAPB–PTPIP51 interaction and are disrupted by ALS/FTD-associated TDP-43 2014 · 505 citations
5050+4+8Years since publication100200300400500
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if any of the following hold:

  • it has ≥500 total citations;
  • it reaches ≥1.5× the top-1% citation threshold for papers in the same subfield and year (the threshold is the minimum needed to enter the top 1%, not the average within it);
  • it reaches the top citation threshold in at least one of its specific research topics.
  1. 2014 Nature Communications

Peers

Jacqueline C. Mitchell
Comparison fields: 5 of 93
  • Neurology 1.2k
  • Genetics 595
  • Behavioral Neuroscience 118
  • Cell Biology 436
  • Neurology 208
Replace M. Galbiati with:
M. Galbiati Italy
P. Rusmini Italy
Claudia Crosio Italy
Elio Messi Italy
V. Crippa Italy
Jacqueline A. Sluijs Netherlands
Tomohiro Kabuta Japan
Louis J. DeGennaro United States
Alyson Peel United States
Sozos Ch. Papasozomenos United States
Jacqueline C. Mitchell relative to M. Galbiati Italy M. Galbiati's profile →
Citations per field
00.5×1.5×2.0×
M. Galbiati · 1×
Citations per year

Countries citing papers authored by Jacqueline C. Mitchell

Since Specialization
Citations

This map shows the geographic impact of Jacqueline C. Mitchell's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Jacqueline C. Mitchell with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Jacqueline C. Mitchell more than expected).

Fields of papers citing papers by Jacqueline C. Mitchell

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Jacqueline C. Mitchell. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Jacqueline C. Mitchell. The network helps show where Jacqueline C. Mitchell may publish in the future.

Co-authors

The 25 scholars most cited alongside Jacqueline C. Mitchell, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Jacqueline C. Mitchell Line = papers co-authored together Jacqueline C. Mitchell links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 42 papers — load more, or switch the sort, to bring in the rest.

#Work
1
ER–mitochondria associations are regulated by the VAPB–PTPIP51 interaction and are disrupted by ALS/FTD-associated TDP-43
Nature Communications ·Radu Stoica, Kurt J. De Vos, Sébastien Paillusson, Sarah B. Mueller, Rosa M. Sancho, Kwok‐Fai Lau, Gema Vizcay‐Barrena, Wen-Lang Lin, Ya-Fei Xu, Jada Lewis, Dennis W. Dickson, Leonard Petrucelli, Jacqueline C. Mitchell, Christopher E. Shaw, Christopher C.J. Miller
Hit paper breakdown →		2014505
2
ALS/FTD‐associated FUS activates GSK‐3β to disrupt the VAPB–PTPIP51 interaction and ER–mitochondria associations
EMBO Reports ·Radu Stoica, Sébastien Paillusson, Patricia Gómez‐Suaga, Jacqueline C. Mitchell, Dawn H. W. Lau, Emma H. Gray, Rosa M. Sancho, Gema Vizcay‐Barrena, Kurt J. De Vos, Christopher E. Shaw, Diane P. Hanger, Wendy Noble, Christopher C.J. Miller
2016224
3
Overexpression of human wild-type FUS causes progressive motor neuron degeneration in an age- and dose-dependent fashion
Acta Neuropathologica ·Jacqueline C. Mitchell, Philip McGoldrick, Caroline Vance, Tibor Hortobágyi, Jemeen Sreedharan, Boris Rogelj, Elizabeth L. Tudor, Bradley Smith, Christian Klasen, Christopher C.J. Miller, Jonathan D. Cooper, Linda Greensmith, Christopher E. Shaw
2012212
4
Differential roles of the ubiquitin proteasome system (UPS) and autophagy in the clearance of soluble and aggregated TDP-43 species
Journal of Cell Science ·Emma L. Scotter, Caroline Vance, Agnes L. Nishimura, Youn‐Bok Lee, Han-Jou Chen, Hazel Urwin, Valentina Sardone, Jacqueline C. Mitchell, Boris Rogelj, David C. Rubinsztein, Christopher E. Shaw
2014212
5
ALS mutant FUS disrupts nuclear localization and sequesters wild-type FUS within cytoplasmic stress granules
Human Molecular Genetics ·Caroline Vance, Emma L. Scotter, Agnes L. Nishimura, Claire Troakes, Jacqueline C. Mitchell, Claudia Kathe, Hazel Urwin, Catherine Manser, Christopher C.J. Miller, Tibor Hortobágyi, Mike Dragunow, Boris Rogelj, Christopher E. Shaw
2013198
6
The heat shock response plays an important role in TDP-43 clearance: evidence for dysfunction in amyotrophic lateral sclerosis
Brain ·Han-Jou Chen, Jacqueline C. Mitchell, Sergey S. Novoselov, Jack W. Miller, Agnes L. Nishimura, Emma L. Scotter, Caroline Vance, Michael E. Cheetham, Christopher E. Shaw
2016132
7
Novel mutations support a role for Profilin 1 in the pathogenesis of ALS
Neurobiology of Aging ·Bradley Smith, Caroline Vance, Emma L. Scotter, Claire Troakes, Chun Hao Wong, Simon Topp, Satomi Maekawa, Andrew King, Jacqueline C. Mitchell, Karan Lund, Ammar Al‐Chalabi, Nicola Ticozzi, Vincenzo Silani, Peter C. Sapp, Robert H. Brown, John E. Landers, Safa Al‐Sarraj, Christopher E. Shaw
201484
8
Wild type human TDP-43 potentiates ALS-linked mutant TDP-43 driven progressive motor and cortical neuron degeneration with pathological features of ALS
Acta Neuropathologica Communications ·Jacqueline C. Mitchell, Remy Constable, Eva So, Caroline Vance, Emma L. Scotter, Leanne Glover, Tibor Hortobágyi, Eveline S. Arnold, Shuo‐Chien Ling, Melissa McAlonis, Sandrine Da Cruz, Magda Polymenidou, Lino Tessarolo, Don W. Cleveland, Christopher E. Shaw
201583
9
The X11/Mint family of adaptor proteins
Brain Research Reviews ·Boris Rogelj, Jacqueline C. Mitchell, Christopher C.J. Miller, Declan M. McLoughlin
200672
10
Mitochondrial abnormalities and disruption of the neuromuscular junction precede the clinical phenotype and motor neuron loss in hFUSWT transgenic mice
Human Molecular Genetics ·Eva So, Jacqueline C. Mitchell, Caroline Memmi, George Chennell, Gema Vizcay‐Barrena, Leanne Allison, Christopher E. Shaw, Caroline Vance
201767
11
The Effect of Oestradiol and Progesterone on Hypoglycaemic Stress‐Induced Suppression of Pulsatile Luteinizing Hormone Release and on Corticotropin‐Releasing Hormone mRNA Expression in the Rat
Journal of Neuroendocrinology ·X. F. Li, Jacqueline C. Mitchell, S. A. Wood, Clive W. Coen, Stafford L. Lightman, (unknown)
200365
12
Amyotrophic lateral sclerosis mutant vesicle-associated membrane protein-associated protein-B transgenic mice develop TAR-DNA-binding protein-43 pathology
Neuroscience ·Elizabeth L. Tudor, Clare Galtrey, Michael S. Perkinton, Kwok‐Fai Lau, Kurt J. De Vos, Jacqueline C. Mitchell, Steven Ackerley, Tibor Hortobágyi, E. Vámos, P. Nigel Leigh, Christian Klasen, Declan M. McLoughlin, Christopher E. Shaw, Christopher C.J. Miller
201063
13
Disruption of ER‐mitochondria tethering and signalling in C9orf72‐associated amyotrophic lateral sclerosis and frontotemporal dementia
Aging Cell ·Patricia Gómez‐Suaga, Gábor M. Mórotz, Andrea Markovinović, Sandra M. Martín‐Guerrero, Elisavet Preza, Natalia Arias, Keith Mayl, Afra Aabdien, Vesela Gesheva, Agnes L. Nishimura, Ambra Annibali, Youn‐Bok Lee, Jacqueline C. Mitchell, Selina Wray, Christopher E. Shaw, Wendy Noble, Christopher C.J. Miller
202252
14
Ultraviolet-mediated antibiotic activity of species of Compositae caused by polyacetylenic compounds.
PubMed ·Towers Gh, Wat Ck, Graham Ea, Bandoni Rj, Chan Gq, Jacqueline C. Mitchell, Lam J
197849
15
Stress-Induced Suppression of the Gonadotropin-Releasing Hormone Pulse Generator in the Female Rat: A Novel Neural Action for Calcitonin Gene-Related Peptide
Endocrinology ·Xiao Feng Li, James E. Bowe, Jacqueline C. Mitchell, Susan D. Brain, Stafford L. Lightman, (unknown)
200446
16
The Role of the Locus Coeruleus in Corticotropin-Releasing Hormone and Stress-Induced Suppression of Pulsatile Luteinizing Hormone Secretion in the Female Rat
Endocrinology ·Jacqueline C. Mitchell, X. F. Li, Leigh Breen, Jean‐Christophe Thalabard, (unknown)
200444
17
Tauopathy induced by low level expression of a human brain-derived tau fragment in mice is rescued by phenylbutyrate
Brain ·Marie K. Bondulich, Tong Guo, Christopher Meehan, John Manion, T. Rodriguez Martin, Jacqueline C. Mitchell, Tibor Hortobágyi, Natalia Yankova, Virginie Stygelbout, Jean‐Pierre Brion, Wendy Noble, Diane P. Hanger
201640
18
Differential Effects of Repeated Restraint Stress on Pulsatile Lutenizing Hormone Secretion in Female Fischer, Lewis and Wistar Rats
Journal of Neuroendocrinology ·X. F. Li, Justin A. Edward, Jacqueline C. Mitchell, Bei Shao, Jonathan Bowes, Clive W. Coen, Stafford L. Lightman, (unknown)
200440
19
Pancreatic exocrine function in severe human chronic renal failure.
Gut ·Chung Owyang, L. J. Miller, Eugene P. DiMagno, Jacqueline C. Mitchell, V. L. W. Go
198235
20
X11  rescues memory and long-term potentiation deficits in Alzheimer's disease APPswe Tg2576 mice
Human Molecular Genetics ·Jacqueline C. Mitchell, Ben Ariff, Darran Yates, Kwok‐Fai Lau, M. S. Perkinton, Boris Rogelj, John D. Stephenson, Christopher C.J. Miller, Declan M. McLoughlin
200930

About Jacqueline C. Mitchell

Jacqueline C. Mitchell is a scholar working on Neurology, Molecular Biology, Physiology, Genetics and Cellular and Molecular Neuroscience, having authored 42 papers that have together received 2.4k indexed citations. Recurring topics across this work include Amyotrophic Lateral Sclerosis Research (18 papers), Neurogenetic and Muscular Disorders Research (9 papers), Alzheimer's disease research and treatments (8 papers), Endoplasmic Reticulum Stress and Disease (4 papers), Hypothalamic control of reproductive hormones (4 papers), Mitochondrial Function and Pathology (4 papers), Stress Responses and Cortisol (4 papers) and Nuclear Receptors and Signaling (3 papers). The work is most often cited by research in Neurology (1.2k citations), Genetics (595 citations), Behavioral Neuroscience (118 citations), Cell Biology (436 citations) and Neurology (208 citations). Jacqueline C. Mitchell has collaborated with scholars based in United Kingdom, United States and Ireland. Frequent co-authors include Christopher E. Shaw, Christopher C.J. Miller, Caroline Vance, Boris Rogelj, Emma L. Scotter, Gema Vizcay‐Barrena, Kurt J. De Vos, Sébastien Paillusson, Agnes L. Nishimura and Radu Stoica. Their work appears in journals such as Human Molecular Genetics, Acta Neuropathologica Communications, Journal of Neuroendocrinology, Aging Cell and Brain.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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