Christopher E. Shaw
Impact in
- Neurology top 0.01%
- Amyotrophic Lateral Sclerosis Research
- Neuroinflammation and Neurodegeneration Mechanisms
- Physiology top 0.1%
- Alzheimer's disease research and treatments
Papers in
- Neurology 255
- Amyotrophic Lateral Sclerosis Research 251
-
- Genetic Neurodegenerative Diseases 6
- Hereditary Neurological Disorders 6
- Neuropeptides and Animal Physiology 6
- Co-authors
- Ammar Al‐Chalabi (84 shared papers)Christopher C.J. Miller (24 shared papers)P. Nigel Leigh (45 shared papers)Boris Rogelj (22 shared papers)Caroline A. Vance (32 shared papers)Steven Ackerley (12 shared papers)P. Nigel Leigh (29 shared papers)Agnes Lumi Nishimura (23 shared papers)
- Journals
- Brain (18 papers)Neurobiology of Aging (17 papers)Journal of Neurology Neurosurgery & Psychiatry (15 papers)Amyotrophic Lateral Sclerosis and Frontotemporal Degeneration (15 papers)Human Molecular Genetics (14 papers)
- Partner nations
- United KingdomUnited StatesNetherlands
In The Last Decade
Christopher E. Shaw
329 papers receiving 43.3k citations
Christopher E. Shaw's Hit Papers
Peers
Comparison fields: 5 of 180
- Neurology 22.3k
- Physiology 7.8k
- Neurology 2.4k
- Psychiatry and Mental health 3.7k
- Cellular and Molecular Neuroscience 2.1k
Countries citing papers authored by Christopher E. Shaw
This map shows the geographic impact of Christopher E. Shaw's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Christopher E. Shaw with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Christopher E. Shaw more than expected).
Fields of papers citing papers by Christopher E. Shaw
This network shows the impact of papers produced by Christopher E. Shaw. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Christopher E. Shaw. The network helps show where Christopher E. Shaw may publish in the future.
Co-authors
The 25 scholars most cited alongside Christopher E. Shaw, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 332 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's disease Hit paper breakdown → | 2009 | 2884 |
| 2 | TDP-43 Mutations in Familial and Sporadic Amyotrophic Lateral Sclerosis Hit paper breakdown → | 2008 | 2233 |
| 3 | Mutations in FUS, an RNA Processing Protein, Cause Familial Amyotrophic Lateral Sclerosis Type 6 Hit paper breakdown → | 2009 | 2165 |
| 4 | Genetic meta-analysis of diagnosed Alzheimer’s disease identifies new risk loci and implicates Aβ, tau, immunity and lipid processing Hit paper breakdown → | 2019 | 2140 |
| 5 | Common variants at ABCA7, MS4A6A/MS4A4E, EPHA1, CD33 and CD2AP are associated with Alzheimer's disease Hit paper breakdown → | 2011 | 1664 |
| 6 | New insights into the genetic etiology of Alzheimer’s disease and related dementias Hit paper breakdown → | 2022 | 1531 |
| 7 | Mutations in prion-like domains in hnRNPA2B1 and hnRNPA1 cause multisystem proteinopathy and ALS Hit paper breakdown → | 2013 | 1216 |
| 8 | Characterizing the RNA targets and position-dependent splicing regulation by TDP-43 Hit paper breakdown → | 2011 | 944 |
| 9 | Exome sequencing in amyotrophic lateral sclerosis identifies risk genes and pathways Hit paper breakdown → | 2015 | 807 |
| 10 | Rare coding variants in PLCG2, ABI3, and TREM2 implicate microglial-mediated innate immunity in Alzheimer's disease Hit paper breakdown → | 2017 | 764 |
| 11 | VEGF is a modifier of amyotrophic lateral sclerosis in mice and humans and protects motoneurons against ischemic death Hit paper breakdown → | 2003 | 743 |
| 12 | Evidence of widespread cerebral microglial activation in amyotrophic lateral sclerosis: an [11C](R)-PK11195 positron emission tomography study Hit paper breakdown → | 2004 | 635 |
| 13 | Genetic Evidence Implicates the Immune System and Cholesterol Metabolism in the Aetiology of Alzheimer's Disease Hit paper breakdown → | 2010 | 551 |
| 14 | Axonal Transport of TDP-43 mRNA Granules Is Impaired by ALS-Causing Mutations Hit paper breakdown → | 2014 | 546 |
| 15 | Genome-Wide Analyses Identify KIF5A as a Novel ALS Gene Hit paper breakdown → | 2018 | 539 |
| 16 | ER–mitochondria associations are regulated by the VAPB–PTPIP51 interaction and are disrupted by ALS/FTD-associated TDP-43 Hit paper breakdown → | 2014 | 527 |
| 17 | 2011 | 483 | |
| 18 | Genome-wide association analyses identify new risk variants and the genetic architecture of amyotrophic lateral sclerosis Hit paper breakdown → | 2016 | 457 |
| 19 | Gain of Toxicity from ALS/FTD-Linked Repeat Expansions in C9ORF72 Is Alleviated by Antisense Oligonucleotides Targeting GGGGCC-Containing RNAs Hit paper breakdown → | 2016 | 450 |
| 20 | 2004 | 431 |
About Christopher E. Shaw
Christopher E. Shaw is a scholar working on Neurology, Cellular and Molecular Neuroscience, Physiology, Developmental Neuroscience and Molecular Biology, having authored 332 papers that have together received 44.2k indexed citations. Recurring topics across this work include Amyotrophic Lateral Sclerosis Research (251 papers), Alzheimer's disease research and treatments (18 papers), Genetic Neurodegenerative Diseases (6 papers), Hereditary Neurological Disorders (6 papers), Neuropeptides and Animal Physiology (6 papers), Skin and Cellular Biology Research (5 papers), RNA Research and Splicing (5 papers) and Neurogenesis and neuroplasticity mechanisms (5 papers). The work is most often cited by research in Neurology (22.3k citations), Physiology (7.8k citations), Neurology (2.4k citations), Psychiatry and Mental health (3.7k citations) and Cellular and Molecular Neuroscience (2.1k citations). Christopher E. Shaw has collaborated with scholars based in United Kingdom, United States and Netherlands. Frequent co-authors include Ammar Al‐Chalabi, Christopher C.J. Miller, P. Nigel Leigh, Boris Rogelj, Caroline A. Vance, Steven Ackerley, P. Nigel Leigh, Agnes Lumi Nishimura, Martin R. Turner and Bradley N. Smith. Their work appears in journals such as Brain, Neurobiology of Aging, Journal of Neurology Neurosurgery & Psychiatry, Amyotrophic Lateral Sclerosis and Frontotemporal Degeneration and Human Molecular Genetics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.