Eva Kling

1.4k citations
12 papers · 217 · h-index 8

Impact in

    • MicroRNA in disease regulation
    • Cancer-related molecular mechanisms research
    • Glioma Diagnosis and Treatment

Papers in

    • Epigenetics and DNA Methylation 5
    • RNA modifications and cancer 3
    • Kruppel-like factors research 2
    • RNA and protein synthesis mechanisms 2
    • Peroxisome Proliferator-Activated Receptors 2
    • MicroRNA in disease regulation 1

Eva Kling

12 papers receiving 213 citations

Peers

Eva Kling
Comparison fields: 5 of 55
  • Cancer Research 60
  • Genetics 37
  • Molecular Biology 166
  • Immunology 30
  • Cell Biology 17
Replace Yu Shen with:
Yu Shen China
Gretchen M. Alicea United States
Yawen Pan China
Roza Berhanu Lemma Norway
Federica Begalli Italy
In‐Young Hwang South Korea
Michaela Jackson United States
Montserrat Puiggròs Spain
M. Shaked-Rabi United Kingdom
Robin Steinhaus Germany
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Citations per field
00.5×6.7×
Yu Shen · 1×
Citations per year

Countries citing papers authored by Eva Kling

Since Specialization
Citations

This map shows the geographic impact of Eva Kling's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eva Kling with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eva Kling more than expected).

Fields of papers citing papers by Eva Kling

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Eva Kling. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eva Kling. The network helps show where Eva Kling may publish in the future.

Co-authors

The 25 scholars most cited alongside Eva Kling, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Eva Kling Line = papers co-authored together Eva Kling links everyone, so they are left out of the graph.

All Works

12 of 12 papers shown
#Work
1 202158
2 201730
3 201729
4 200828
5 200619
6 201619
7 201615
8 20229
9 20225
10 20183
11 20241
12 20181

About Eva Kling

Eva Kling is a scholar working on Molecular Biology, Cancer Research, Genetics, Immunology and Clinical Biochemistry, having authored 12 papers that have together received 217 indexed citations. Recurring topics across this work include Epigenetics and DNA Methylation (5 papers), RNA modifications and cancer (3 papers), Kruppel-like factors research (2 papers), RNA and protein synthesis mechanisms (2 papers), Peroxisome Proliferator-Activated Receptors (2 papers), Glioma Diagnosis and Treatment (2 papers), interferon and immune responses (1 paper) and MicroRNA in disease regulation (1 paper). The work is most often cited by research in Cancer Research (60 citations), Genetics (37 citations), Molecular Biology (166 citations), Immunology (30 citations) and Cell Biology (17 citations). Eva Kling has collaborated with scholars based in Germany, Sweden and Italy. Frequent co-authors include Maria Stella Carro, Roberto Ferrarese, Sven Nelander, Thomas Unterkircher, Dieter Henrik Heiland, Fangping Dai, Astrid Weyerbrock, Massimo Squatrito, Carolina Marques and Gaetano Gargiulo. Their work appears in journals such as Molecular Cancer Research, iScience, Oncotarget, Communications Biology and Life Science Alliance.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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