Bart De Strooper

80.1k citations
440 papers · 63.8k · 24 hit papers · h-index 132

Impact in

  • Physiology top 0.01%
    • Alzheimer's disease research and treatments
  • Neurology top 0.01%
    • Neuroinflammation and Neurodegeneration Mechanisms

Papers in

    • Alzheimer's disease research and treatments 240
    • Neuroinflammation and Neurodegeneration Mechanisms 50

Bart De Strooper

432 papers receiving 62.6k citations

Bart De Strooper's Hit Papers

Xenografted human microglia display diverse transcriptomic states in response to Alzheimer’s disease-related amyloid-β pathology 2024 · 90 citations
900+4+9Years since publication50010001.5k2.0k2.5k

Peers

Bart De Strooper
Comparison fields: 5 of 183
  • Physiology 30.4k
  • Neurology 8.7k
  • Biological Psychiatry 2.2k
  • Cellular and Molecular Neuroscience 10.4k
  • Developmental Neuroscience 2.1k
Replace Christian Haass with:
Christian Haass Germany
Rudolph E. Tanzi United States
Valina L. Dawson United States
Isidró Ferrer Spain
Stuart A. Lipton United States
M. Flint Beal United States
Takaomi C. Saido Japan
Ted M. Dawson United States
Frank M. LaFerla United States
Ralph A. Nixon United States
Bart De Strooper relative to Christian Haass Germany Christian Haass's profile →
Citations per field
00.5×1.5×
Christian Haass · 1×
Citations per year

Countries citing papers authored by Bart De Strooper

Since Specialization
Citations

This map shows the geographic impact of Bart De Strooper's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bart De Strooper with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bart De Strooper more than expected).

Fields of papers citing papers by Bart De Strooper

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bart De Strooper. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bart De Strooper. The network helps show where Bart De Strooper may publish in the future.

Co-authors

The 25 scholars most cited alongside Bart De Strooper, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Bart De Strooper Line = papers co-authored together Bart De Strooper links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 440 papers — load more, or switch the sort, to bring in the rest.

#Work
1
Alzheimer's disease
Hit paper breakdown →
20162650
2
The amyloid cascade hypothesis for Alzheimer's disease: an appraisal for the development of therapeutics
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20111858
3
A presenilin-1-dependent γ-secretase-like protease mediates release of Notch intracellular domain
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19991803
4
Reactive astrocyte nomenclature, definitions, and future directions
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20211721
5
The toxic Aβ oligomer and Alzheimer's disease: an emperor in need of clothes
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20121689
6
Deficiency of presenilin-1 inhibits the normal cleavage of amyloid precursor protein
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19981544
7
The Cellular Phase of Alzheimer’s Disease
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20161464
8
OPA1 Controls Apoptotic Cristae Remodeling Independently from Mitochondrial Fusion
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20061390
9
Microglia states and nomenclature: A field at its crossroads
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20221302
10
Cholesterol depletion inhibits the generation of β-amyloid in hippocampal neurons
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19981037
11
Loss of microRNA cluster miR-29a/b-1 in sporadic Alzheimer's disease correlates with increased BACE1/β-secretase expression
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20081016
12
Aph-1, Pen-2, and Nicastrin with Presenilin Generate an Active γ-Secretase Complex
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2003833
13
The secretases: enzymes with therapeutic potential in Alzheimer disease
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2010682
14
The Major Risk Factors for Alzheimer’s Disease: Age, Sex, and Genes Modulate the Microglia Response to Aβ Plaques
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2019637
15
Spatial Transcriptomics and In Situ Sequencing to Study Alzheimer’s Disease
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2020636
16 2006615
17 2006594
18 2006591
19
APP mouse models for Alzheimer's disease preclinical studies
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2017574
20 2005569

About Bart De Strooper

Bart De Strooper is a scholar working on Physiology, Neurology, Cellular and Molecular Neuroscience, Molecular Biology and Cell Biology, having authored 440 papers that have together received 63.8k indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (240 papers), Cholinesterase and Neurodegenerative Diseases (53 papers), Neuroinflammation and Neurodegeneration Mechanisms (50 papers), Computational Drug Discovery Methods (43 papers), Cellular transport and secretion (37 papers), Nuclear Receptors and Signaling (36 papers), Neuroscience and Neuropharmacology Research (36 papers) and Prion Diseases and Protein Misfolding (30 papers). The work is most often cited by research in Physiology (30.4k citations), Neurology (8.7k citations), Biological Psychiatry (2.2k citations), Cellular and Molecular Neuroscience (10.4k citations) and Developmental Neuroscience (2.1k citations). Bart De Strooper has collaborated with scholars based in Belgium, United States and United Kingdom. Frequent co-authors include Eric H. Karran, Wim G. Annaert, Paul Säftig, Katleen Craessaerts, Iryna Benilova, Sébastien S. Hébert, Marc H. Mercken, Lutgarde Serneels, Stephen Salloway and Dieter Hartmann. Their work appears in journals such as Journal of Biological Chemistry, Alzheimer s & Dementia, Neuron, Cell and The EMBO Journal.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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