Yoshitake Maeda
Impact in
- Hematology top 10%
- Platelet Disorders and Treatments
Papers in
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- Glycosylation and Glycoproteins Research 7
- Protein purification and stability 4
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- Monoclonal and Polyclonal Antibodies Research 7
- Co-authors
- Tadashi Ueda (4 shared papers)Hidenori Yamada (5 shared papers)Taiji Imoto (3 shared papers)Ryota Kuroki (9 shared papers)Taro Tamada (6 shared papers)Eijiro Honjo (4 shared papers)Matsujiro Ishibashi (3 shared papers)Tomoyuki Okamoto (3 shared papers)
In The Last Decade
Yoshitake Maeda
19 papers receiving 561 citations
Peers
Comparison fields: 5 of 84
- Hematology 79
- Immunology 140
- Oncology 132
- Molecular Biology 359
- Radiology, Nuclear Medicine and Imaging 103
Countries citing papers authored by Yoshitake Maeda
This map shows the geographic impact of Yoshitake Maeda's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yoshitake Maeda with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yoshitake Maeda more than expected).
Fields of papers citing papers by Yoshitake Maeda
This network shows the impact of papers produced by Yoshitake Maeda. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yoshitake Maeda. The network helps show where Yoshitake Maeda may publish in the future.
Co-authors
The 25 scholars most cited alongside Yoshitake Maeda, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2009 | 130 | |
| 2 | 2006 | 98 | |
| 3 | 2004 | 68 | |
| 4 | 1995 | 66 | |
| 5 | 1996 | 63 | |
| 6 | 2005 | 26 | |
| 7 | 1993 | 24 | |
| 8 | 1996 | 22 | |
| 9 | 1994 | 13 | |
| 10 | 2004 | 12 | |
| 11 | 2002 | 10 | |
| 12 | 2011 | 9 | |
| 13 | 2010 | 9 | |
| 14 | 2013 | 7 | |
| 15 | 2000 | 6 | |
| 16 | 2004 | 5 | |
| 17 | 2005 | 5 | |
| 18 | 2016 | 3 | |
| 19 | 2008 | 3 | |
| 20 | 2019 | 1 |
About Yoshitake Maeda
Yoshitake Maeda is a scholar working on Molecular Biology, Radiology, Nuclear Medicine and Imaging, Immunology, Hematology and Materials Chemistry, having authored 20 papers that have together received 580 indexed citations. Recurring topics across this work include Glycosylation and Glycoproteins Research (7 papers), Monoclonal and Polyclonal Antibodies Research (7 papers), Protein purification and stability (4 papers), Enzyme Structure and Function (4 papers), Platelet Disorders and Treatments (3 papers), Chemokine receptors and signaling (3 papers), Immune Response and Inflammation (3 papers) and T-cell and B-cell Immunology (2 papers). The work is most often cited by research in Hematology (79 citations), Immunology (140 citations), Oncology (132 citations), Molecular Biology (359 citations) and Radiology, Nuclear Medicine and Imaging (103 citations). Yoshitake Maeda has collaborated with scholars based in Japan, Germany and Russia. Frequent co-authors include Tadashi Ueda, Hidenori Yamada, Taiji Imoto, Ryota Kuroki, Taro Tamada, Eijiro Honjo, Matsujiro Ishibashi, Tomoyuki Okamoto, Masao Tokunaga and Hiroshi Koga. Their work appears in journals such as Protein Engineering Design and Selection, The Journal of Biochemistry, Protein Science, Proceedings of the National Academy of Sciences and FEBS Journal.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.