Thomas Comptdaer
Impact in
- Neurology top 5%
- Parkinson's Disease Mechanisms and Treatments
- Neuroinflammation and Neurodegeneration Mechanisms
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- Cellular transport and secretion
- Microtubule and mitosis dynamics
- Endoplasmic Reticulum Stress and Disease
Papers in
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- Parkinson's Disease Mechanisms and Treatments 6
- Neuroinflammation and Neurodegeneration Mechanisms 1
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- Alzheimer's disease research and treatments 3
- Co-authors
- Marie‐Christine Chartier‐Harlin (6 shared papers)Eugénie Mutez (5 shared papers)Martin Figeac (3 shared papers)A. Destée (5 shared papers)Karim Bélarbi (2 shared papers)Aurélie Duflot (2 shared papers)Christel Vanbesien-Mailliot (2 shared papers)Frédéric Leprêtre (2 shared papers)
In The Last Decade
Thomas Comptdaer
13 papers receiving 346 citations
Peers
Comparison fields: 5 of 40
- Neurology 184
- Neurology 29
- Cell Biology 37
- Physiology 51
- Endocrine and Autonomic Systems 6
Countries citing papers authored by Thomas Comptdaer
This map shows the geographic impact of Thomas Comptdaer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Comptdaer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Comptdaer more than expected).
Fields of papers citing papers by Thomas Comptdaer
This network shows the impact of papers produced by Thomas Comptdaer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Comptdaer. The network helps show where Thomas Comptdaer may publish in the future.
Co-authors
The 25 scholars most cited alongside Thomas Comptdaer, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2018 | 92 | |
| 2 | 2020 | 58 | |
| 3 | 2013 | 55 | |
| 4 | 2019 | 26 | |
| 5 | 2022 | 23 | |
| 6 | 2018 | 19 | |
| 7 | 2022 | 18 | |
| 8 | 2016 | 15 | |
| 9 | 2021 | 15 | |
| 10 | 2023 | 13 | |
| 11 | 2022 | 6 | |
| 12 | 2024 | 5 | |
| 13 | 2015 | 1 |
About Thomas Comptdaer
Thomas Comptdaer is a scholar working on Neurology, Physiology, Neurology, Molecular Biology and Cellular and Molecular Neuroscience, having authored 13 papers that have together received 346 indexed citations. Recurring topics across this work include Parkinson's Disease Mechanisms and Treatments (6 papers), Alzheimer's disease research and treatments (3 papers), RNA and protein synthesis mechanisms (1 paper), Neuroinflammation and Neurodegeneration Mechanisms (1 paper), Genomics and Chromatin Dynamics (1 paper), Mosquito-borne diseases and control (1 paper), Genetic Neurodegenerative Diseases (1 paper) and DNA Repair Mechanisms (1 paper). The work is most often cited by research in Neurology (184 citations), Neurology (29 citations), Cell Biology (37 citations), Physiology (51 citations) and Endocrine and Autonomic Systems (6 citations). Thomas Comptdaer has collaborated with scholars based in France, Belgium and Spain. Frequent co-authors include Marie‐Christine Chartier‐Harlin, Eugénie Mutez, Martin Figeac, A. Destée, Karim Bélarbi, Aurélie Duflot, Christel Vanbesien-Mailliot, Frédéric Leprêtre, Céline Villenet and Séverine Bleuse. Their work appears in journals such as Neurobiology of Disease, Frontiers in Cell and Developmental Biology, Cancers, Nutrients and Brain Pathology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.