Emil Gregersen
Impact in
- Neurology top 10%
- Parkinson's Disease Mechanisms and Treatments
- Neurological disorders and treatments
- Neuroinflammation and Neurodegeneration Mechanisms
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- Neuroscience and Neuropharmacology Research
- Nuclear Receptors and Signaling
Papers in
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- Parkinson's Disease Mechanisms and Treatments 7
- Neurological disorders and treatments 4
- Parkinson's Disease and Spinal Disorders 1
- Neuroinflammation and Neurodegeneration Mechanisms 1
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- Alzheimer's disease research and treatments 3
- Co-authors
- Poul Henning Jensen (7 shared papers)Cristine Betzer (5 shared papers)Rikke Hahn Kofoed (2 shared papers)Louise Berkhoudt Lassen (2 shared papers)Lasse Reimer (3 shared papers)Arne Moeller (2 shared papers)Glenda M. Halliday (2 shared papers)Tito Calí (1 shared paper)
In The Last Decade
Emil Gregersen
8 papers receiving 263 citations
Peers
Comparison fields: 5 of 64
- Neurology 168
- Neurology 45
- Cellular and Molecular Neuroscience 84
- Physiology 84
- Aging 4
Countries citing papers authored by Emil Gregersen
This map shows the geographic impact of Emil Gregersen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Emil Gregersen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Emil Gregersen more than expected).
Fields of papers citing papers by Emil Gregersen
This network shows the impact of papers produced by Emil Gregersen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Emil Gregersen. The network helps show where Emil Gregersen may publish in the future.
Co-authors
The 25 scholars most cited alongside Emil Gregersen, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2018 | 94 | |
| 2 | 2018 | 54 | |
| 3 | 2021 | 35 | |
| 4 | 2021 | 29 | |
| 5 | 2019 | 24 | |
| 6 | 2021 | 22 | |
| 7 | 2023 | 6 | |
| 8 | 2023 | 4 | |
| 9 | 2024 | 0 |
About Emil Gregersen
Emil Gregersen is a scholar working on Neurology, Physiology, Surgery, Molecular Biology and Genetics, having authored 9 papers that have together received 268 indexed citations. Recurring topics across this work include Parkinson's Disease Mechanisms and Treatments (7 papers), Neurological disorders and treatments (4 papers), Alzheimer's disease research and treatments (3 papers), Mesenchymal stem cell research (2 papers), Tissue Engineering and Regenerative Medicine (2 papers), Parkinson's Disease and Spinal Disorders (1 paper), Neuroinflammation and Neurodegeneration Mechanisms (1 paper) and Ginkgo biloba and Cashew Applications (1 paper). The work is most often cited by research in Neurology (168 citations), Neurology (45 citations), Cellular and Molecular Neuroscience (84 citations), Physiology (84 citations) and Aging (4 citations). Emil Gregersen has collaborated with scholars based in Denmark, Germany and Australia. Frequent co-authors include Poul Henning Jensen, Cristine Betzer, Rikke Hahn Kofoed, Louise Berkhoudt Lassen, Lasse Reimer, Arne Moeller, Glenda M. Halliday, Tito Calí, Susana Aznar and YuHong Fu. Their work appears in journals such as PLoS ONE, Apmis, The EMBO Journal, Journal of Neurochemistry and Frontiers in Cell and Developmental Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.