Sammy L. Eber

26 papers receiving 1.8k citations

Peers

Sammy L. Eber
Comparison fields: 5 of 94
  • Endocrinology 323
  • Gastroenterology 115
  • Nutrition and Dietetics 342
  • Physiology 74
  • Nephrology 99
Replace F. K. Hamra with:
F. K. Hamra United States
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Citations per year

Countries citing papers authored by Sammy L. Eber

Since Specialization
Citations

This map shows the geographic impact of Sammy L. Eber's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sammy L. Eber with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sammy L. Eber more than expected).

Fields of papers citing papers by Sammy L. Eber

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sammy L. Eber. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sammy L. Eber. The network helps show where Sammy L. Eber may publish in the future.

Co-authors

The 25 scholars most cited alongside Sammy L. Eber, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Sammy L. Eber Line = papers co-authored together Sammy L. Eber links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 26 papers — load more, or switch the sort, to bring in the rest.

#Work
1 1993284
2 1992231
3
Uroguanylin treatment suppresses polyp formation in the Apc(Min/+) mouse and induces apoptosis in human colon adenocarcinoma cells via cyclic GMP.
2000182
4 199794
5 199382
6 199781
7
Distribution of heat-stable enterotoxin/guanylin receptors in the intestinal tract of man and other mammals.
199479
8 198773
9 199471
10 200067
11 199664
12 199753
13 198852
14 199750
15 199049
16 199944
17 199742
18 199537
19 199934
20 199633

About Sammy L. Eber

Sammy L. Eber is a scholar working on Endocrinology, Molecular Biology, Nutrition and Dietetics, Microbiology and Immunology, having authored 26 papers that have together received 1.8k indexed citations. Recurring topics across this work include Escherichia coli research studies (10 papers), Antimicrobial Peptides and Activities (6 papers), Invertebrate Immune Response Mechanisms (5 papers), Infant Nutrition and Health (4 papers), Metabolism, Diabetes, and Cancer (3 papers), Parathyroid Disorders and Treatments (3 papers), Viral gastroenteritis research and epidemiology (2 papers) and Protein Kinase Regulation and GTPase Signaling (2 papers). The work is most often cited by research in Endocrinology (323 citations), Gastroenterology (115 citations), Nutrition and Dietetics (342 citations), Physiology (74 citations) and Nephrology (99 citations). Sammy L. Eber has collaborated with scholars based in United States, India and Germany. Frequent co-authors include Leonard R. Forte, William J. Krause, Ronald H. Freeman, F. K. Hamra, Mark G. Currie, Kam F. Fok, David T. Chin, P. K. Thorne, Roslyn M. London and Xiaohui Fan. Their work appears in journals such as Endocrinology, American Journal of Physiology-Endocrinology and Metabolism, Proceedings of the National Academy of Sciences, Poultry Science and American Journal of Physiology-Gastrointestinal and Liver Physiology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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