Ralph Schulz
Impact in
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- Chronic Lymphocytic Leukemia Research
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- Hematopoietic Stem Cell Transplantation
Papers in
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- Immune Cell Function and Interaction 4
- T-cell and B-cell Immunology 3
- Galectins and Cancer Biology 2
- Immunodeficiency and Autoimmune Disorders 2
- Immunotherapy and Immune Responses 1
- Genetics 6
- Chronic Lymphocytic Leukemia Research 6
- Co-authors
- Martina Seiffert (7 shared papers)Stephan Stilgenbauer (3 shared papers)B Dörken (1 shared paper)S Cayeux (1 shared paper)Philipp M. Roessner (2 shared papers)Antonio Pezzutto (1 shared paper)M Körbling (1 shared paper)Bola S. Hanna (2 shared papers)
- Journals
- Blood (3 papers)British Journal of Haematology (1 paper)Leukemia (1 paper)HemaSphere (1 paper)Haematologica (1 paper)
- Partner nations
- GermanySpainUnited States
In The Last Decade
Ralph Schulz
8 papers receiving 121 citations
Peers
Comparison fields: 5 of 30
- Genetics 55
- Hematology 32
- Immunology 58
- Pathology and Forensic Medicine 33
- Oncology 35
Countries citing papers authored by Ralph Schulz
This map shows the geographic impact of Ralph Schulz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ralph Schulz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ralph Schulz more than expected).
Fields of papers citing papers by Ralph Schulz
This network shows the impact of papers produced by Ralph Schulz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ralph Schulz. The network helps show where Ralph Schulz may publish in the future.
Co-authors
The 25 scholars most cited alongside Ralph Schulz, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2019 | 38 | |
| 2 | 2020 | 32 | |
| 3 | 1989 | 32 | |
| 4 | 2019 | 13 | |
| 5 | 2018 | 4 | |
| 6 | 2016 | 2 | |
| 7 | 2018 | 1 | |
| 8 | 2019 | 1 | |
| 9 | Development of selective tolerance to particular types of opiate receptors. | 1982 | 0 |
About Ralph Schulz
Ralph Schulz is a scholar working on Immunology, Genetics, Molecular Biology, Nephrology and Pathology and Forensic Medicine, having authored 9 papers that have together received 123 indexed citations. Recurring topics across this work include Chronic Lymphocytic Leukemia Research (6 papers), Immune Cell Function and Interaction (4 papers), T-cell and B-cell Immunology (3 papers), Galectins and Cancer Biology (2 papers), Immunodeficiency and Autoimmune Disorders (2 papers), Acute Lymphoblastic Leukemia research (1 paper), Immunotherapy and Immune Responses (1 paper) and Pharmacological Receptor Mechanisms and Effects (1 paper). The work is most often cited by research in Genetics (55 citations), Hematology (32 citations), Immunology (58 citations), Pathology and Forensic Medicine (33 citations) and Oncology (35 citations). Ralph Schulz has collaborated with scholars based in Germany, Spain and United States. Frequent co-authors include Martina Seiffert, Stephan Stilgenbauer, B Dörken, S Cayeux, Philipp M. Roessner, Antonio Pezzutto, M Körbling, Bola S. Hanna, Dolors Colomer and Peter Lichter. Their work appears in journals such as Blood, British Journal of Haematology, Leukemia, HemaSphere and Haematologica.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.