Michael Whiteside

818 citations
8 papers · 676 · h-index 7

Impact in

Papers in

    • Neuroinflammation and Neurodegeneration Mechanisms 3
    • Barrier Structure and Function Studies 1
    • Immune Response and Inflammation 2
    • Galectins and Cancer Biology 1

Michael Whiteside

8 papers receiving 660 citations

Peers

Michael Whiteside
Comparison fields: 5 of 73
  • Biological Psychiatry 115
  • Behavioral Neuroscience 134
  • Neurology 254
  • Endocrine and Autonomic Systems 76
  • Immunology 219
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Citations per field
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Citations per year

Countries citing papers authored by Michael Whiteside

Since Specialization
Citations

This map shows the geographic impact of Michael Whiteside's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael Whiteside with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael Whiteside more than expected).

Fields of papers citing papers by Michael Whiteside

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael Whiteside. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael Whiteside. The network helps show where Michael Whiteside may publish in the future.

Co-authors

The 11 scholars most cited alongside Michael Whiteside, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Michael Whiteside Line = papers co-authored together Michael Whiteside links everyone, so they are left out of the graph.

All Works

8 of 8 papers shown
#Work
1 1998274
2 1998137
3 1997136
4 199973
5 202120
6 201017
7 200016
8
Induction of inhibitory factor kBa mRNA in the central nervous system after peripheral lipopolysaccharide administration: An in situ hybridization histochemistry study in the rat (endotoxinycytokineynuclear factor kByblood-brain barrieryglia)
19973

About Michael Whiteside

Michael Whiteside is a scholar working on Neurology, Immunology, Surgery, Molecular Biology and Epidemiology, having authored 8 papers that have together received 676 indexed citations. Recurring topics across this work include Neuroinflammation and Neurodegeneration Mechanisms (3 papers), Trypanosoma species research and implications (2 papers), Receptor Mechanisms and Signaling (2 papers), Calcium signaling and nucleotide metabolism (2 papers), Immune Response and Inflammation (2 papers), Barrier Structure and Function Studies (1 paper), Eicosanoids and Hypertension Pharmacology (1 paper) and Galectins and Cancer Biology (1 paper). The work is most often cited by research in Biological Psychiatry (115 citations), Behavioral Neuroscience (134 citations), Neurology (254 citations), Endocrine and Autonomic Systems (76 citations) and Immunology (219 citations). Michael Whiteside has collaborated with scholars based in United States and Sweden. Frequent co-authors include Miles Herkenham, Ning Quan, Jama Mhlanga, Krister Kristensson, Allison N. McCoy, Clyde A. Helms, Emily N. Vinson, Louis E. DeFrate, Tally Lassiter and Melissa Scribani. Their work appears in journals such as Neuroscience, The American Journal of Sports Medicine, The Journal of Comparative Neurology, Proceedings of the National Academy of Sciences and Brain Research.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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