Manuel Serrano
Impact in
- Aging top 0.01%
- Genetics, Aging, and Longevity in Model Organisms
- Geriatrics and Gerontology top 0.02%
- Sirtuins and Resveratrol in Medicine
Papers in
-
- Pluripotent Stem Cells Research 34
- Epigenetics and DNA Methylation 32
- CRISPR and Genetic Engineering 24
- RNA modifications and cancer 20
- Ubiquitin and proteasome pathways 18
- Physiology 81
- Telomeres, Telomerase, and Senescence 62
- Co-authors
- Marı́a A. Blasco (26 shared papers)Guido Kroemer (4 shared papers)Carlos López-Otı́n (3 shared papers)Linda Partridge (2 shared papers)Manuel Collado (32 shared papers)David Beach (12 shared papers)Daniel Muñoz‐Espín (9 shared papers)Gregory J. Hannon (2 shared papers)
- Journals
- Oncogene (16 papers)Cell Cycle (12 papers)Cancer Research (12 papers)Aging Cell (12 papers)Nature (9 papers)
- Partner nations
- SpainUnited StatesUnited Kingdom
In The Last Decade
Manuel Serrano
271 papers receiving 54.7k citations
Manuel Serrano's Hit Papers
Peers
Comparison fields: 5 of 194
- Aging 5.4k
- Geriatrics and Gerontology 2.9k
- Physiology 16.4k
- Oncology 12.4k
- Molecular Biology 30.8k
Countries citing papers authored by Manuel Serrano
This map shows the geographic impact of Manuel Serrano's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Manuel Serrano with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Manuel Serrano more than expected).
Fields of papers citing papers by Manuel Serrano
This network shows the impact of papers produced by Manuel Serrano. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Manuel Serrano. The network helps show where Manuel Serrano may publish in the future.
Co-authors
The 25 scholars most cited alongside Manuel Serrano, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 276 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | The Hallmarks of Aging Hit paper breakdown → | 2013 | 11136 |
| 2 | Oncogenic ras Provokes Premature Cell Senescence Associated with Accumulation of p53 and p16INK4a Hit paper breakdown → | 1997 | 4219 |
| 3 | Hallmarks of aging: An expanding universe Hit paper breakdown → | 2023 | 3385 |
| 4 | A new regulatory motif in cell-cycle control causing specific inhibition of cyclin D/CDK4 Hit paper breakdown → | 1993 | 3151 |
| 5 | Cellular senescence: from physiology to pathology Hit paper breakdown → | 2014 | 2110 |
| 6 | Role of the INK4a Locus in Tumor Suppression and Cell Mortality Hit paper breakdown → | 1996 | 1377 |
| 7 | Cellular Senescence in Cancer and Aging Hit paper breakdown → | 2007 | 1375 |
| 8 | Senescence in premalignant tumours Hit paper breakdown → | 2005 | 1242 |
| 9 | Programmed Cell Senescence during Mammalian Embryonic Development Hit paper breakdown → | 2013 | 1129 |
| 10 | A p16 INK4a -Insensitive CDK4 Mutant Targeted by Cytolytic T Lymphocytes in a Human Melanoma Hit paper breakdown → | 1995 | 956 |
| 11 | Senescence in tumours: evidence from mice and humans Hit paper breakdown → | 2009 | 886 |
| 12 | A p53-mediated DNA damage response limits reprogramming to ensure iPS cell genomic integrity Hit paper breakdown → | 2009 | 851 |
| 13 | The common biology of cancer and ageing Hit paper breakdown → | 2007 | 845 |
| 14 | Sirt1 protects against high-fat diet-induced metabolic damage Hit paper breakdown → | 2008 | 806 |
| 15 | The Ink4/Arf locus is a barrier for iPS cell reprogramming Hit paper breakdown → | 2009 | 804 |
| 16 | Premature senescence involving p53 and p16 is activated in response to constitutive MEK/MAPK mitogenic signaling Hit paper breakdown → | 1998 | 796 |
| 17 | Sirt1 improves healthy ageing and protects from metabolic syndrome-associated cancer Hit paper breakdown → | 2010 | 526 |
| 18 | GLP-1 Agonism Stimulates Brown Adipose Tissue Thermogenesis and Browning Through Hypothalamic AMPK Hit paper breakdown → | 2014 | 496 |
| 19 | 1994 | 493 | |
| 20 | Tissue damage and senescence provide critical signals for cellular reprogramming in vivo Hit paper breakdown → | 2016 | 466 |
About Manuel Serrano
Manuel Serrano is a scholar working on Molecular Biology, Physiology, Oncology, Cancer Research and Genetics, having authored 276 papers that have together received 55.8k indexed citations. Recurring topics across this work include Cancer-related Molecular Pathways (68 papers), Telomeres, Telomerase, and Senescence (62 papers), Pluripotent Stem Cells Research (34 papers), Epigenetics and DNA Methylation (32 papers), CRISPR and Genetic Engineering (24 papers), Genetics, Aging, and Longevity in Model Organisms (21 papers), RNA modifications and cancer (20 papers) and Ubiquitin and proteasome pathways (18 papers). The work is most often cited by research in Aging (5.4k citations), Geriatrics and Gerontology (2.9k citations), Physiology (16.4k citations), Oncology (12.4k citations) and Molecular Biology (30.8k citations). Manuel Serrano has collaborated with scholars based in Spain, United States and United Kingdom. Frequent co-authors include Marı́a A. Blasco, Guido Kroemer, Carlos López-Otı́n, Linda Partridge, Manuel Collado, David Beach, Daniel Muñoz‐Espín, Gregory J. Hannon, Athena W. Lin and Scott Lowe. Their work appears in journals such as Oncogene, Cell Cycle, Cancer Research, Aging Cell and Nature.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.