Beth Levine
Impact in
- Epidemiology top 0.01%
- Autophagy in Disease and Therapy
- Aging top 0.05%
- Genetics, Aging, and Longevity in Model Organisms
Papers in
- Epidemiology 156
- Autophagy in Disease and Therapy 151
- Herpesvirus Infections and Treatments 4
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- Cell death mechanisms and regulation 20
- Co-authors
- Guido Kroemer (12 shared papers)Noboru Mizushima (9 shared papers)Daniel J. Klionsky (6 shared papers)Xiaonan Dong (7 shared papers)Herbert W. Virgin (13 shared papers)Ana María Cuervo (3 shared papers)Tamotsu Yoshimori (2 shared papers)Vojo P. Deretic (6 shared papers)
- Journals
- Autophagy (31 papers)Cell (16 papers)Nature (14 papers)Journal of Virology (13 papers)Journal of Clinical Investigation (12 papers)
- Partner nations
- United StatesFranceJapan
In The Last Decade
Beth Levine
220 papers receiving 111.5k citations
Beth Levine's Hit Papers
Peers
Comparison fields: 5 of 180
- Epidemiology 56.6k
- Aging 1.8k
- Molecular Biology 23.2k
- Immunology 6.8k
- Cell Biology 4k
Countries citing papers authored by Beth Levine
This map shows the geographic impact of Beth Levine's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Beth Levine with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Beth Levine more than expected).
Fields of papers citing papers by Beth Levine
This network shows the impact of papers produced by Beth Levine. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Beth Levine. The network helps show where Beth Levine may publish in the future.
Co-authors
The 25 scholars most cited alongside Beth Levine, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 221 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | Autophagy in the Pathogenesis of Disease Hit paper breakdown → | 2008 | 6118 |
| 2 | Autophagy fights disease through cellular self-digestion Hit paper breakdown → | 2008 | 5661 |
| 3 | Molecular mechanisms of cell death: recommendations of the Nomenclature Committee on Cell Death 2018 Hit paper breakdown → | 2018 | 5362 |
| 4 | Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) Hit paper breakdown → | 2016 | 4680 |
| 5 | Autophagy and Viruses: Adversaries or Allies? Hit paper breakdown → | 2013 | 4068 |
| 6 | Methods in Mammalian Autophagy Research Hit paper breakdown → | 2010 | 4010 |
| 7 | Guidelines for the use and interpretation of assays for monitoring autophagy Hit paper breakdown → | 2012 | 3543 |
| 8 | Development by Self-Digestion Hit paper breakdown → | 2004 | 3294 |
| 9 | Bcl-2 Antiapoptotic Proteins Inhibit Beclin 1-Dependent Autophagy Hit paper breakdown → | 2005 | 3074 |
| 10 | Autophagy and the Integrated Stress Response Hit paper breakdown → | 2010 | 2983 |
| 11 | Induction of autophagy and inhibition of tumorigenesis by beclin 1 Hit paper breakdown → | 1999 | 2888 |
| 12 | Autophagy in immunity and inflammation Hit paper breakdown → | 2011 | 2823 |
| 13 | Biological Functions of Autophagy Genes: A Disease Perspective Hit paper breakdown → | 2019 | 2287 |
| 14 | Autophagy in Human Health and Disease Hit paper breakdown → | 2013 | 2244 |
| 15 | Elevated Circulating Levels of Tumor Necrosis Factor in Severe Chronic Heart Failure Hit paper breakdown → | 1990 | 2225 |
| 16 | Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition) 1 Hit paper breakdown → | 2021 | 2067 |
| 17 | Guidelines for the use and interpretation of assays for monitoring autophagy in higher eukaryotes Hit paper breakdown → | 2008 | 1996 |
| 18 | Promotion of tumorigenesis by heterozygous disruption of the beclin 1 autophagy gene Hit paper breakdown → | 2003 | 1992 |
| 19 | Autophagy in cell death: an innocent convict? Hit paper breakdown → | 2005 | 1470 |
| 20 | Molecular definitions of autophagy and related processes Hit paper breakdown → | 2017 | 1371 |
About Beth Levine
Beth Levine is a scholar working on Epidemiology, Molecular Biology, Public Health, Environmental and Occupational Health, Aging and Immunology, having authored 221 papers that have together received 113.6k indexed citations. Recurring topics across this work include Autophagy in Disease and Therapy (151 papers), Cell death mechanisms and regulation (20 papers), Mosquito-borne diseases and control (18 papers), interferon and immune responses (6 papers), Tuberculosis Research and Epidemiology (5 papers), Genetics, Aging, and Longevity in Model Organisms (5 papers), Herpesvirus Infections and Treatments (4 papers) and Infectious Encephalopathies and Encephalitis (4 papers). The work is most often cited by research in Epidemiology (56.6k citations), Aging (1.8k citations), Molecular Biology (23.2k citations), Immunology (6.8k citations) and Cell Biology (4k citations). Beth Levine has collaborated with scholars based in United States, France and Japan. Frequent co-authors include Guido Kroemer, Noboru Mizushima, Daniel J. Klionsky, Xiaonan Dong, Herbert W. Virgin, Ana María Cuervo, Tamotsu Yoshimori, Vojo P. Deretic, Guillermo Mariño and Milton Packer. Their work appears in journals such as Autophagy, Cell, Nature, Journal of Virology and Journal of Clinical Investigation.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.