Judith Campisi
Impact in
- Aging top 0.01%
- Genetics, Aging, and Longevity in Model Organisms
- Physiology top 0.01%
- Telomeres, Telomerase, and Senescence
Papers in
- Physiology 203
- Telomeres, Telomerase, and Senescence 186
- Aging 60
- Genetics, Aging, and Longevity in Model Organisms 60
- Co-authors
- Pierre‐Yves Desprez (30 shared papers)Ana Krtolica (13 shared papers)Fabrizio d’Adda di Fagagna (1 shared paper)Jean‐Philippe Coppé (14 shared papers)Françis Rodier (22 shared papers)Adam Freund (8 shared papers)Marco Demaria (27 shared papers)Christopher K. Patil (8 shared papers)
- Journals
- Aging Cell (27 papers)Aging (18 papers)Journal of Biological Chemistry (14 papers)Proceedings of the National Academy of Sciences (13 papers)Molecular and Cellular Biology (13 papers)
- Partner nations
- United StatesNetherlandsCanada
In The Last Decade
Judith Campisi
348 papers receiving 80.4k citations
Judith Campisi's Hit Papers
Peers
Comparison fields: 5 of 196
- Aging 8.3k
- Physiology 34.1k
- Geriatrics and Gerontology 2.3k
- Immunology 14.1k
- Cancer Research 8.9k
Countries citing papers authored by Judith Campisi
This map shows the geographic impact of Judith Campisi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Judith Campisi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Judith Campisi more than expected).
Fields of papers citing papers by Judith Campisi
This network shows the impact of papers produced by Judith Campisi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Judith Campisi. The network helps show where Judith Campisi may publish in the future.
Co-authors
The 25 scholars most cited alongside Judith Campisi, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 353 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | The Senescence-Associated Secretory Phenotype: The Dark Side of Tumor Suppression Hit paper breakdown → | 2010 | 4140 |
| 2 | Cellular senescence: when bad things happen to good cells Hit paper breakdown → | 2007 | 3598 |
| 3 | Chronic inflammation in the etiology of disease across the life span Hit paper breakdown → | 2019 | 3539 |
| 4 | Cellular Senescence: Defining a Path Forward Hit paper breakdown → | 2019 | 2366 |
| 5 | Aging, Cellular Senescence, and Cancer Hit paper breakdown → | 2012 | 2321 |
| 6 | Geroscience: Linking Aging to Chronic Disease Hit paper breakdown → | 2014 | 2013 |
| 7 | Senescent Cells, Tumor Suppression, and Organismal Aging: Good Citizens, Bad Neighbors Hit paper breakdown → | 2005 | 1936 |
| 8 | Persistent DNA damage signalling triggers senescence-associated inflammatory cytokine secretion Hit paper breakdown → | 2009 | 1823 |
| 9 | Four faces of cellular senescence Hit paper breakdown → | 2011 | 1641 |
| 10 | An Essential Role for Senescent Cells in Optimal Wound Healing through Secretion of PDGF-AA Hit paper breakdown → | 2014 | 1637 |
| 11 | Clearance of senescent cells by ABT263 rejuvenates aged hematopoietic stem cells in mice Hit paper breakdown → | 2015 | 1495 |
| 12 | Cellular senescence and the senescent secretory phenotype: therapeutic opportunities Hit paper breakdown → | 2013 | 1444 |
| 13 | Senescent fibroblasts promote epithelial cell growth and tumorigenesis: A link between cancer and aging Hit paper breakdown → | 2001 | 1391 |
| 14 | Protocols to detect senescence-associated beta-galactosidase (SA-βgal) activity, a biomarker of senescent cells in culture and in vivo Hit paper breakdown → | 2009 | 1369 |
| 15 | Local clearance of senescent cells attenuates the development of post-traumatic osteoarthritis and creates a pro-regenerative environment Hit paper breakdown → | 2017 | 1222 |
| 16 | Targeted Apoptosis of Senescent Cells Restores Tissue Homeostasis in Response to Chemotoxicity and Aging Hit paper breakdown → | 2017 | 1167 |
| 17 | Reversal of human cellular senescence: roles of the p53 and p16 pathways Hit paper breakdown → | 2003 | 1163 |
| 18 | Inflammatory networks during cellular senescence: causes and consequences Hit paper breakdown → | 2010 | 1124 |
| 19 | Cellular Senescence Promotes Adverse Effects of Chemotherapy and Cancer Relapse Hit paper breakdown → | 2016 | 1086 |
| 20 | From discoveries in ageing research to therapeutics for healthy ageing Hit paper breakdown → | 2019 | 1021 |
About Judith Campisi
Judith Campisi is a scholar working on Physiology, Aging, Molecular Biology, Cancer Research and Immunology, having authored 353 papers that have together received 82.1k indexed citations. Recurring topics across this work include Telomeres, Telomerase, and Senescence (186 papers), Genetics, Aging, and Longevity in Model Organisms (60 papers), DNA Repair Mechanisms (49 papers), Cancer-related Molecular Pathways (40 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (30 papers), Advanced biosensing and bioanalysis techniques (22 papers), Epigenetics and DNA Methylation (19 papers) and Genomics and Chromatin Dynamics (16 papers). The work is most often cited by research in Aging (8.3k citations), Physiology (34.1k citations), Geriatrics and Gerontology (2.3k citations), Immunology (14.1k citations) and Cancer Research (8.9k citations). Judith Campisi has collaborated with scholars based in United States, Netherlands and Canada. Frequent co-authors include Pierre‐Yves Desprez, Ana Krtolica, Fabrizio d’Adda di Fagagna, Jean‐Philippe Coppé, Françis Rodier, Adam Freund, Marco Demaria, Christopher K. Patil, Simon Melov and Christopher D. Wiley. Their work appears in journals such as Aging Cell, Aging, Journal of Biological Chemistry, Proceedings of the National Academy of Sciences and Molecular and Cellular Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.