John J. Halliday

993 citations
9 papers · 742 · 1 hit paper · h-index 7

Impact in

  • Genetics top 5%
    • Glioma Diagnosis and Treatment
    • MicroRNA in disease regulation
    • Cancer, Hypoxia, and Metabolism
    • Cancer-related molecular mechanisms research

Papers in

    • RNA and protein synthesis mechanisms 2
    • Polyamine Metabolism and Applications 1
    • Drug Transport and Resistance Mechanisms 3

John J. Halliday

8 papers receiving 729 citations

John J. Halliday's Hit Papers

Osteopontin-CD44 Signaling in the Glioma Perivascular Niche Enhances Cancer Stem Cell Phenotypes and Promotes Aggressive Tumor Growth 2014 · 414 citations
4140+4+8Years since publication100200300400

Peers

John J. Halliday
Comparison fields: 5 of 64
  • Genetics 279
  • Cancer Research 191
  • Oncology 211
  • Cell Biology 88
  • Immunology 107
Replace Demirkan Gursel with:
Demirkan Gursel United States
Dieter Lemke Germany
Yeri Lee South Korea
Tushar Tomar Netherlands
Jérôme Kroonen Belgium
German G. Gomez United States
Ivan Radovanovic Switzerland
Muhammad Jamal United States
Sara Abbadi United States
Sakellarios Zairis United States
John J. Halliday relative to Demirkan Gursel United States Demirkan Gursel's profile →
Citations per field
00.5×1.5×
Demirkan Gursel · 1×
Citations per year

Countries citing papers authored by John J. Halliday

Since Specialization
Citations

This map shows the geographic impact of John J. Halliday's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John J. Halliday with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John J. Halliday more than expected).

Fields of papers citing papers by John J. Halliday

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John J. Halliday. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John J. Halliday. The network helps show where John J. Halliday may publish in the future.

Co-authors

The 25 scholars most cited alongside John J. Halliday, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with John J. Halliday Line = papers co-authored together John J. Halliday links everyone, so they are left out of the graph.

All Works

9 of 9 papers shown
#Work
1
Osteopontin-CD44 Signaling in the Glioma Perivascular Niche Enhances Cancer Stem Cell Phenotypes and Promotes Aggressive Tumor Growth
Hit paper breakdown →
2014414
2 2014138
3 201397
4 200824
5 197524
6 201021
7 201220
8 20104
9 19810

About John J. Halliday

John J. Halliday is a scholar working on Molecular Biology, Oncology, Genetics, Nutrition and Dietetics and Infectious Diseases, having authored 9 papers that have together received 742 indexed citations. Recurring topics across this work include Drug Transport and Resistance Mechanisms (3 papers), Trace Elements in Health (2 papers), Glioma Diagnosis and Treatment (2 papers), RNA and protein synthesis mechanisms (2 papers), Polyamine Metabolism and Applications (1 paper), Neurogenesis and neuroplasticity mechanisms (1 paper), Amino Acid Enzymes and Metabolism (1 paper) and Liver physiology and pathology (1 paper). The work is most often cited by research in Genetics (279 citations), Cancer Research (191 citations), Oncology (211 citations), Cell Biology (88 citations) and Immunology (107 citations). John J. Halliday has collaborated with scholars based in United States and South Africa. Frequent co-authors include Kenneth L. Pitter, Jason T. Huse, Eric C. Holland, Elin Ekström, Nduka Amankulor, Alexander Pietras, Jillian L. Werbeck, Amanda Katz, Karim Y. Helmy and Tatsuya Ozawa. Their work appears in journals such as Journal of Biological Chemistry, Cell stem cell, Analytical Biochemistry, Proceedings of the National Academy of Sciences and Brain Pathology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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