Joaquín Botta

16 papers receiving 422 citations

Peers

Joaquín Botta
Comparison fields: 5 of 66
  • Endocrine and Autonomic Systems 36
  • Endocrinology, Diabetes and Metabolism 86
  • Cellular and Molecular Neuroscience 97
  • Molecular Biology 242
  • Physiology 13
Replace Isabelle Van Liefde with:
Isabelle Van Liefde Belgium
Sung‐Jun Han United States
Amanda J. Wheal United Kingdom
Stefanie Blättermann Germany
Morihiro Mitsuya Japan
Lihua Wang‐Eckhardt Germany
Yuji Shinjo Japan
Linshan Xie China
Sanaz Darbalaei China
Aaron M. D’Antona United States
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Citations per year

Countries citing papers authored by Joaquín Botta

Since Specialization
Citations

This map shows the geographic impact of Joaquín Botta's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Joaquín Botta with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Joaquín Botta more than expected).

Fields of papers citing papers by Joaquín Botta

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Joaquín Botta. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Joaquín Botta. The network helps show where Joaquín Botta may publish in the future.

Co-authors

The 25 scholars most cited alongside Joaquín Botta, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Joaquín Botta Line = papers co-authored together Joaquín Botta links everyone, so they are left out of the graph.

All Works

16 of 16 papers shown
#Work
1 202166
2 198365
3 201941
4 201739
5 201733
6 202031
7 198929
8 202025
9 201324
10 201924
11 198519
12 202012
13 20199
14 19879
15 19903
16 20173

About Joaquín Botta

Joaquín Botta is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology, Cell Biology, Physiology and Organic Chemistry, having authored 16 papers that have together received 432 indexed citations. Recurring topics across this work include Receptor Mechanisms and Signaling (5 papers), Hemoglobin structure and function (5 papers), Erythrocyte Function and Pathophysiology (4 papers), Neurotransmitter Receptor Influence on Behavior (3 papers), Neuropeptides and Animal Physiology (3 papers), Neonatal Health and Biochemistry (2 papers), Monoclonal and Polyclonal Antibodies Research (2 papers) and Mass Spectrometry Techniques and Applications (2 papers). The work is most often cited by research in Endocrine and Autonomic Systems (36 citations), Endocrinology, Diabetes and Metabolism (86 citations), Cellular and Molecular Neuroscience (97 citations), Molecular Biology (242 citations) and Physiology (13 citations). Joaquín Botta has collaborated with scholars based in United Kingdom, Argentina and Spain. Frequent co-authors include Ricardo N. Farı́as, Peter J. McCormick, Roberto D. Morero, Diego de Mendoza, Saher A. Shaikh, Paul Thomas, Lesley A. Howell, Vadivel Prabahar, Bellinda Benhamú and Oksana Degtjarik. Their work appears in journals such as Journal of Biological Chemistry, Progress in molecular biology and translational science, Life Science Alliance, Biochemical and Biophysical Research Communications and European Journal of Neuroscience.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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