James E. Bradner

54.9k citations
218 papers · 29.3k · 17 hit papers · h-index 87

Impact in

  • Hematology top 0.05%
    • Multiple Myeloma Research and Treatments
    • Protein Degradation and Inhibitors
    • Ubiquitin and proteasome pathways
    • Histone Deacetylase Inhibitors Research
    • Epigenetics and DNA Methylation
    • Genomics and Chromatin Dynamics
    • RNA Research and Splicing

Papers in

    • Protein Degradation and Inhibitors 140
    • Histone Deacetylase Inhibitors Research 64
    • Ubiquitin and proteasome pathways 60
    • Genomics and Chromatin Dynamics 24
    • Epigenetics and DNA Methylation 18
    • Multiple Myeloma Research and Treatments 43
    • Acute Myeloid Leukemia Research 16

James E. Bradner

212 papers receiving 28.9k citations

James E. Bradner's Hit Papers

Plasticity in binding confers selectivity in ligand-induced protein degradation 2018 · 446 citations
4460+5+11Years since publication4008001.2k

Peers

James E. Bradner
Comparison fields: 5 of 157
  • Hematology 5.4k
  • Molecular Biology 23.5k
  • Oncology 5.8k
  • Cancer Research 2.1k
  • Genetics 1.2k
Replace Nathanael S. Gray with:
Nathanael S. Gray United States
Andrew L. Kung United States
Victoria M. Richon United States
Craig M. Crews United States
Ricky W. Johnstone Australia
Kristian Helin Denmark
E. Premkumar Reddy United States
Saverio Minucci Italy
Kevan M. Shokat United States
Martine F. Roussel United States
James E. Bradner relative to Nathanael S. Gray United States Nathanael S. Gray's profile →
Citations per field
00.5×1.6×
Nathanael S. Gray · 1×
Citations per year

Countries citing papers authored by James E. Bradner

Since Specialization
Citations

This map shows the geographic impact of James E. Bradner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James E. Bradner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James E. Bradner more than expected).

Fields of papers citing papers by James E. Bradner

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by James E. Bradner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James E. Bradner. The network helps show where James E. Bradner may publish in the future.

Co-authors

The 25 scholars most cited alongside James E. Bradner, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with James E. Bradner Line = papers co-authored together James E. Bradner links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 218 papers — load more, or switch the sort, to bring in the rest.

#Work
1
RNAi screen identifies Brd4 as a therapeutic target in acute myeloid leukaemia
Hit paper breakdown →
20111447
2
Phthalimide conjugation as a strategy for in vivo target protein degradation
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20151281
3
HDAC2 negatively regulates memory formation and synaptic plasticity
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20091280
4
The Myeloma Drug Lenalidomide Promotes the Cereblon-Dependent Destruction of Ikaros Proteins
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20131132
5
Transcriptional Amplification in Tumor Cells with Elevated c-Myc
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20121130
6
Transcriptional Addiction in Cancer
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2017774
7
Coactivation of Receptor Tyrosine Kinases Affects the Response of Tumor Cells to Targeted Therapies
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2007705
8
YY1 Is a Structural Regulator of Enhancer-Promoter Loops
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2017689
9
The dTAG system for immediate and target-specific protein degradation
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2018682
10
Direct inhibition of the NOTCH transcription factor complex
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2009621
11
PF00299804, an Irreversible Pan-ERBB Inhibitor, Is Effective in Lung Cancer Models with EGFR and ERBB2 Mutations that Are Resistant to Gefitinib
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2007604
12
Chemical phylogenetics of histone deacetylases
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2010584
13
Discovery and Characterization of Super-Enhancer-Associated Dependencies in Diffuse Large B Cell Lymphoma
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2013560
14 2005475
15
Targeting MYCN in Neuroblastoma by BET Bromodomain Inhibition
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2013465
16 2014464
17
Plasticity in binding confers selectivity in ligand-induced protein degradation
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2018446
18
Pharmacological perturbation of CDK9 using selective CDK9 inhibition or degradation
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2017412
19 2015362
20 2015355

About James E. Bradner

James E. Bradner is a scholar working on Molecular Biology, Hematology, Oncology, Organic Chemistry and Immunology, having authored 218 papers that have together received 29.3k indexed citations. Recurring topics across this work include Protein Degradation and Inhibitors (140 papers), Histone Deacetylase Inhibitors Research (64 papers), Ubiquitin and proteasome pathways (60 papers), Multiple Myeloma Research and Treatments (43 papers), Genomics and Chromatin Dynamics (24 papers), Peptidase Inhibition and Analysis (20 papers), Epigenetics and DNA Methylation (18 papers) and Acute Myeloid Leukemia Research (16 papers). The work is most often cited by research in Hematology (5.4k citations), Molecular Biology (23.5k citations), Oncology (5.8k citations), Cancer Research (2.1k citations) and Genetics (1.2k citations). James E. Bradner has collaborated with scholars based in United States, China and United Kingdom. Frequent co-authors include Richard A. Young, Jun Qi, Dennis L. Buckley, Denes Hnisz, Joshiawa Paulk, Georg E. Winter, Justin M. Roberts, Ralph Mazitschek, Charles Y. Lin and Tong Ihn Lee. Their work appears in journals such as Blood, Cancer Research, Proceedings of the National Academy of Sciences, Cell and Nature Chemical Biology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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