Frank McCormick
Impact in
- Molecular Biology top 0.01%
- Protein Kinase Regulation and GTPase Signaling
- PI3K/AKT/mTOR signaling in cancer
- Wnt/β-catenin signaling in development and cancer
- Melanoma and MAPK Pathways
- Cancer-related gene regulation
- Cell Biology top 0.02%
- Cellular transport and secretion
Papers in
-
- Protein Kinase Regulation and GTPase Signaling 125
- PI3K/AKT/mTOR signaling in cancer 48
- Melanoma and MAPK Pathways 47
- Cell death mechanisms and regulation 32
- Wnt/β-catenin signaling in development and cancer 25
- Oncology 119
- Cancer-related Molecular Pathways 67
- Co-authors
- Osamu Tetsu (16 shared papers)Henry R. Bourne (5 shared papers)D.A.R. Sanders (2 shared papers)Mark S. Boguski (1 shared paper)Charles J. Sherr (1 shared paper)Meg Trahey (8 shared papers)Simon J. Cook (6 shared papers)Gideon Bollag (12 shared papers)
- Journals
- Proceedings of the National Academy of Sciences (28 papers)Molecular and Cellular Biology (27 papers)Journal of Biological Chemistry (25 papers)Science (19 papers)Oncogene (15 papers)
- Partner nations
- United StatesUnited KingdomJapan
In The Last Decade
Frank McCormick
377 papers receiving 56.5k citations
Frank McCormick's Hit Papers
Peers
Comparison fields: 5 of 184
- Molecular Biology 41.1k
- Cell Biology 9.1k
- Oncology 13.4k
- Cancer Research 5.1k
- Immunology and Allergy 1.9k
Countries citing papers authored by Frank McCormick
This map shows the geographic impact of Frank McCormick's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Frank McCormick with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Frank McCormick more than expected).
Fields of papers citing papers by Frank McCormick
This network shows the impact of papers produced by Frank McCormick. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Frank McCormick. The network helps show where Frank McCormick may publish in the future.
Co-authors
The 25 scholars most cited alongside Frank McCormick, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 384 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | β-Catenin regulates expression of cyclin D1 in colon carcinoma cells Hit paper breakdown → | 1999 | 3118 |
| 2 | The GTPase superfamily: conserved structure and molecular mechanism Hit paper breakdown → | 1991 | 2848 |
| 3 | The GTPase superfamily: a conserved switch for diverse cell functions Hit paper breakdown → | 1990 | 1955 |
| 4 | Proteins regulating Ras and its relatives Hit paper breakdown → | 1993 | 1630 |
| 5 | An Adenovirus Mutant That Replicates Selectively in p53- Deficient Human Tumor Cells Hit paper breakdown → | 1996 | 1398 |
| 6 | RAS Proteins and Their Regulators in Human Disease Hit paper breakdown → | 2017 | 1305 |
| 7 | Drug-tolerant persister cancer cells are vulnerable to GPX4 inhibition Hit paper breakdown → | 2017 | 1255 |
| 8 | The RB and p53 pathways in cancer Hit paper breakdown → | 2002 | 1245 |
| 9 | Dual Role of Phosphatidylinositol-3,4,5-trisphosphate in the Activation of Protein Kinase B Hit paper breakdown → | 1997 | 1041 |
| 10 | A Cytoplasmic Protein Stimulates Normal N- ras p21 GTPase, But Does Not Affect Oncogenic Mutants Hit paper breakdown → | 1987 | 1040 |
| 11 | Protein Kinase B Kinases That Mediate Phosphatidylinositol 3,4,5-Trisphosphate-Dependent Activation of Protein Kinase B Hit paper breakdown → | 1998 | 915 |
| 12 | Inhibition by cAMP of Ras-Dependent Activation of Raf Hit paper breakdown → | 1993 | 846 |
| 13 | Two G Protein Oncogenes in Human Endocrine Tumors Hit paper breakdown → | 1990 | 829 |
| 14 | An essential role for Rac in Ras transformation Hit paper breakdown → | 1995 | 792 |
| 15 | The GAP-related domain of the neurofibromatosis type 1 gene product interacts with ras p21 Hit paper breakdown → | 1990 | 787 |
| 16 | ONYX-015, an E1B gene-attenuated adenovirus, causes tumor-specific cytolysis and antitumoral efficacy that can be augmented by standard chemotherapeutic agents Hit paper breakdown → | 1997 | 746 |
| 17 | RAS-targeted therapies: is the undruggable drugged? Hit paper breakdown → | 2020 | 729 |
| 18 | Wiskott–Aldrich Syndrome Protein, a Novel Effector for the GTPase CDC42Hs, Is Implicated in Actin Polymerization Hit paper breakdown → | 1996 | 725 |
| 19 | Phosphorylation of GAP and GAP-associated proteins by transforming and mitogenic tyrosine kinases Hit paper breakdown → | 1990 | 690 |
| 20 | Targeting RAF kinases for cancer therapy: BRAF-mutated melanoma and beyond Hit paper breakdown → | 2014 | 628 |
About Frank McCormick
Frank McCormick is a scholar working on Molecular Biology, Oncology, Cell Biology, Genetics and Neurology, having authored 384 papers that have together received 57.9k indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (125 papers), Cancer-related Molecular Pathways (67 papers), PI3K/AKT/mTOR signaling in cancer (48 papers), Melanoma and MAPK Pathways (47 papers), Virus-based gene therapy research (37 papers), Cell death mechanisms and regulation (32 papers), Neurofibromatosis and Schwannoma Cases (26 papers) and Wnt/β-catenin signaling in development and cancer (25 papers). The work is most often cited by research in Molecular Biology (41.1k citations), Cell Biology (9.1k citations), Oncology (13.4k citations), Cancer Research (5.1k citations) and Immunology and Allergy (1.9k citations). Frank McCormick has collaborated with scholars based in United States, United Kingdom and Japan. Frequent co-authors include Osamu Tetsu, Henry R. Bourne, D.A.R. Sanders, Mark S. Boguski, Charles J. Sherr, Meg Trahey, Simon J. Cook, Gideon Bollag, Marc Symons and Dwight V. Nissley. Their work appears in journals such as Proceedings of the National Academy of Sciences, Molecular and Cellular Biology, Journal of Biological Chemistry, Science and Oncogene.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.