Eli Keshet
Impact in
- Cancer Research top 0.05%
- Cancer, Hypoxia, and Metabolism
- Ophthalmology top 0.1%
- Retinal Diseases and Treatments
Papers in
-
- Angiogenesis and VEGF in Cancer 35
- Congenital heart defects research 13
- RNA Research and Splicing 9
- RNA and protein synthesis mechanisms 8
-
- Cancer, Hypoxia, and Metabolism 20
- Co-authors
- Ahuva Itin (44 shared papers)Dorit Shweiki (6 shared papers)Dov Soffer (1 shared paper)Laura E. Benjamin (6 shared papers)Itzhak Hemo (6 shared papers)Yuval Dor (17 shared papers)Michal Neeman (7 shared papers)Tamar Alon (3 shared papers)
- Journals
- Proceedings of the National Academy of Sciences (10 papers)Journal of Virology (7 papers)Molecular and Cellular Biology (7 papers)Journal of Clinical Investigation (7 papers)Development (6 papers)
- Partner nations
- IsraelUnited StatesSwitzerland
In The Last Decade
Eli Keshet
119 papers receiving 24.0k citations
Eli Keshet's Hit Papers
Peers
Comparison fields: 5 of 155
- Cancer Research 6.3k
- Ophthalmology 2.1k
- Obstetrics and Gynecology 1.5k
- Molecular Biology 12.9k
- Immunology and Allergy 869
Countries citing papers authored by Eli Keshet
This map shows the geographic impact of Eli Keshet's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Eli Keshet with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Eli Keshet more than expected).
Fields of papers citing papers by Eli Keshet
This network shows the impact of papers produced by Eli Keshet. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Eli Keshet. The network helps show where Eli Keshet may publish in the future.
Co-authors
The 25 scholars most cited alongside Eli Keshet, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 120 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | Vascular endothelial growth factor induced by hypoxia may mediate hypoxia-initiated angiogenesis Hit paper breakdown → | 1992 | 4066 |
| 2 | Role of HIF-1α in hypoxia-mediated apoptosis, cell proliferation and tumour angiogenesis Hit paper breakdown → | 1998 | 2216 |
| 3 | Decidual NK cells regulate key developmental processes at the human fetal-maternal interface Hit paper breakdown → | 2006 | 1405 |
| 4 | Vascular endothelial growth factor acts as a survival factor for newly formed retinal vessels and has implications for retinopathy of prematurity Hit paper breakdown → | 1995 | 1380 |
| 5 | A plasticity window for blood vessel remodelling is defined by pericyte coverage of the preformed endothelial network and is regulated by PDGF-B and VEGF Hit paper breakdown → | 1998 | 1055 |
| 6 | Selective ablation of immature blood vessels in established human tumors follows vascular endothelial growth factor withdrawal Hit paper breakdown → | 1999 | 1038 |
| 7 | VEGF-Induced Adult Neovascularization: Recruitment, Retention, and Role of Accessory Cells Hit paper breakdown → | 2006 | 963 |
| 8 | Development of retinal vasculature is mediated by hypoxia-induced vascular endothelial growth factor (VEGF) expression by neuroglia Hit paper breakdown → | 1995 | 882 |
| 9 | Loss of HIF-2α and inhibition of VEGF impair fetal lung maturation, whereas treatment with VEGF prevents fatal respiratory distress in premature mice Hit paper breakdown → | 2002 | 620 |
| 10 | Induction of vascular endothelial growth factor expression by hypoxia and by glucose deficiency in multicell spheroids: implications for tumor angiogenesis. Hit paper breakdown → | 1995 | 527 |
| 11 | 2002 | 488 | |
| 12 | 1993 | 473 | |
| 13 | 1997 | 446 | |
| 14 | 1997 | 443 | |
| 15 | 1998 | 436 | |
| 16 | 1995 | 409 | |
| 17 | Hypoxia-induced expression of vascular endothelial growth factor by retinal cells is a common factor in neovascularizing ocular diseases. | 1995 | 380 |
| 18 | 1994 | 374 | |
| 19 | 2002 | 329 | |
| 20 | 1990 | 329 |
About Eli Keshet
Eli Keshet is a scholar working on Molecular Biology, Cancer Research, Genetics, Cellular and Molecular Neuroscience and Surgery, having authored 120 papers that have together received 25.1k indexed citations. Recurring topics across this work include Angiogenesis and VEGF in Cancer (35 papers), Cancer, Hypoxia, and Metabolism (20 papers), Congenital heart defects research (13 papers), RNA Research and Splicing (9 papers), Axon Guidance and Neuronal Signaling (9 papers), Virus-based gene therapy research (8 papers), RNA and protein synthesis mechanisms (8 papers) and Neurogenesis and neuroplasticity mechanisms (7 papers). The work is most often cited by research in Cancer Research (6.3k citations), Ophthalmology (2.1k citations), Obstetrics and Gynecology (1.5k citations), Molecular Biology (12.9k citations) and Immunology and Allergy (869 citations). Eli Keshet has collaborated with scholars based in Israel, United States and Switzerland. Frequent co-authors include Ahuva Itin, Dorit Shweiki, Dov Soffer, Laura E. Benjamin, Itzhak Hemo, Yuval Dor, Michal Neeman, Tamar Alon, Hadassah Gnessin and Rinat Abramovitch. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Virology, Molecular and Cellular Biology, Journal of Clinical Investigation and Development.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.