Paz Einat
Impact in
- Cancer Research top 1%
- MicroRNA in disease regulation
- Cancer-related molecular mechanisms research
- Cancer, Hypoxia, and Metabolism
- Molecular Biology top 5%
- Circular RNAs in diseases
- RNA modifications and cancer
- RNA Research and Splicing
- PI3K/AKT/mTOR signaling in cancer
- Muscle Physiology and Disorders
Papers in
-
- Cancer, Hypoxia, and Metabolism 3
-
- CRISPR and Genetic Engineering 2
- Mitochondrial Function and Pathology 2
- Cancer-related gene regulation 2
- Co-authors
- Rami Skaliter (6 shared papers)Shlomit Gilad (2 shared papers)Isaac Bentwich (1 shared paper)Ranit Aharonov (1 shared paper)Zvi Bentwich (1 shared paper)Amir Avniel (1 shared paper)Omer Barad (1 shared paper)Eti Meiri (1 shared paper)
- Journals
- Molecular and Cellular Biology (2 papers)Oncogene (2 papers)Investigative Ophthalmology & Visual Science (1 paper)Nature Genetics (1 paper)Journal of Endocrinology (1 paper)
- Partner nations
- IsraelUnited StatesFrance
In The Last Decade
Paz Einat
18 papers receiving 3.7k citations
Paz Einat's Hit Papers
Peers
Comparison fields: 5 of 120
- Cancer Research 1.5k
- Molecular Biology 2.5k
- Endocrine and Autonomic Systems 141
- Aging 27
- Cell Biology 216
Countries citing papers authored by Paz Einat
This map shows the geographic impact of Paz Einat's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Paz Einat with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Paz Einat more than expected).
Fields of papers citing papers by Paz Einat
This network shows the impact of papers produced by Paz Einat. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Paz Einat. The network helps show where Paz Einat may publish in the future.
Co-authors
The 25 scholars most cited alongside Paz Einat, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | Identification of hundreds of conserved and nonconserved human microRNAs Hit paper breakdown → | 2005 | 1547 |
| 2 | 2002 | 497 | |
| 3 | 1998 | 434 | |
| 4 | 2002 | 324 | |
| 5 | 1989 | 200 | |
| 6 | 2003 | 157 | |
| 7 | 2000 | 111 | |
| 8 | 2004 | 110 | |
| 9 | 1983 | 82 | |
| 10 | 2014 | 78 | |
| 11 | Homeobox genes in mouse development. | 1991 | 73 |
| 12 | 2003 | 70 | |
| 13 | Rare variants of ATM and risk for Hodgkin's disease and radiation-associated breast cancers. | 2002 | 38 |
| 14 | 2008 | 37 | |
| 15 | 2003 | 18 | |
| 16 | 2006 | 10 | |
| 17 | 1996 | 6 | |
| 18 | 1990 | 6 | |
| 19 | 2014 | 0 |
About Paz Einat
Paz Einat is a scholar working on Cancer Research, Molecular Biology, Nutrition and Dietetics, Endocrine and Autonomic Systems and Biochemistry, having authored 19 papers that have together received 3.8k indexed citations. Recurring topics across this work include Cancer, Hypoxia, and Metabolism (3 papers), Animal Genetics and Reproduction (2 papers), Lipid metabolism and biosynthesis (2 papers), CRISPR and Genetic Engineering (2 papers), Biochemical Analysis and Sensing Techniques (2 papers), melanin and skin pigmentation (2 papers), Mitochondrial Function and Pathology (2 papers) and Cancer-related gene regulation (2 papers). The work is most often cited by research in Cancer Research (1.5k citations), Molecular Biology (2.5k citations), Endocrine and Autonomic Systems (141 citations), Aging (27 citations) and Cell Biology (216 citations). Paz Einat has collaborated with scholars based in Israel, United States and France. Frequent co-authors include Rami Skaliter, Shlomit Gilad, Isaac Bentwich, Ranit Aharonov, Zvi Bentwich, Amir Avniel, Omer Barad, Eti Meiri, Yael Spector and Eilon Sharon. Their work appears in journals such as Molecular and Cellular Biology, Oncogene, Investigative Ophthalmology & Visual Science, Nature Genetics and Journal of Endocrinology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.