Edel Kavanagh
Impact in
- Neurology top 1%
- Neuroinflammation and Neurodegeneration Mechanisms
- Biological Psychiatry top 5%
- Tryptophan and brain disorders
Papers in
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- Cell death mechanisms and regulation 7
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- Neuroinflammation and Neurodegeneration Mechanisms 7
- Co-authors
- Bertrand Joseph (12 shared papers)Miguel Ángel Burguillos (8 shared papers)José L. Venero (7 shared papers)Jens Füllgrabe (1 shared paper)Nabil Hajji (2 shared papers)Annette Persson (3 shared papers)Elisabet Englund (3 shared papers)Tomas Deierborg (3 shared papers)
In The Last Decade
Edel Kavanagh
19 papers receiving 1.7k citations
Peers
Comparison fields: 5 of 94
- Neurology 568
- Biological Psychiatry 98
- Developmental Neuroscience 95
- Immunology 461
- Molecular Biology 858
Countries citing papers authored by Edel Kavanagh
This map shows the geographic impact of Edel Kavanagh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Edel Kavanagh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Edel Kavanagh more than expected).
Fields of papers citing papers by Edel Kavanagh
This network shows the impact of papers produced by Edel Kavanagh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Edel Kavanagh. The network helps show where Edel Kavanagh may publish in the future.
Co-authors
The 25 scholars most cited alongside Edel Kavanagh, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2011 | 478 | |
| 2 | 2015 | 278 | |
| 3 | 2020 | 261 | |
| 4 | 2011 | 216 | |
| 5 | 2011 | 80 | |
| 6 | 2016 | 74 | |
| 7 | 2014 | 61 | |
| 8 | 2017 | 55 | |
| 9 | 2014 | 46 | |
| 10 | 2008 | 36 | |
| 11 | 2015 | 26 | |
| 12 | 2006 | 24 | |
| 13 | 2016 | 20 | |
| 14 | 2012 | 18 | |
| 15 | 2012 | 15 | |
| 16 | 2013 | 13 | |
| 17 | 2022 | 8 | |
| 18 | 2013 | 3 | |
| 19 | 2023 | 1 |
About Edel Kavanagh
Edel Kavanagh is a scholar working on Molecular Biology, Neurology, Cellular and Molecular Neuroscience, Immunology and Cell Biology, having authored 19 papers that have together received 1.7k indexed citations. Recurring topics across this work include Neuroinflammation and Neurodegeneration Mechanisms (7 papers), Cell death mechanisms and regulation (7 papers), Nerve injury and regeneration (3 papers), Nuclear Receptors and Signaling (3 papers), Cancer-related Molecular Pathways (3 papers), Inflammation biomarkers and pathways (2 papers), Endoplasmic Reticulum Stress and Disease (2 papers) and Adenosine and Purinergic Signaling (2 papers). The work is most often cited by research in Neurology (568 citations), Biological Psychiatry (98 citations), Developmental Neuroscience (95 citations), Immunology (461 citations) and Molecular Biology (858 citations). Edel Kavanagh has collaborated with scholars based in Sweden, Spain and Ireland. Frequent co-authors include Bertrand Joseph, Miguel Ángel Burguillos, José L. Venero, Jens Füllgrabe, Nabil Hajji, Annette Persson, Elisabet Englund, Tomas Deierborg, Albert García-Quintanilla and Josefina Cano. Their work appears in journals such as Cell Death and Disease, SLAS DISCOVERY, Acta Neuropathologica Communications, Aging and Frontiers in Cell and Developmental Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.