David Lyden
Impact in
- Cancer Research top 0.05%
- MicroRNA in disease regulation
- Cancer, Hypoxia, and Metabolism
- Oncology top 0.1%
- Cancer Cells and Metastasis
Papers in
-
- Extracellular vesicles in disease 26
- Angiogenesis and VEGF in Cancer 17
- Oncology 30
- Cancer Cells and Metastasis 18
- Co-authors
- Shahin Rafii (22 shared papers)Bethan Psaila (7 shared papers)Héctor Peinado (7 shared papers)Rosandra N. Kaplan (9 shared papers)Beate Heissig (7 shared papers)Robert Benezra (6 shared papers)Koichi Hattori (6 shared papers)Sérgio Dias (6 shared papers)
- Journals
- Cancer Research (9 papers)Pediatric Blood & Cancer (6 papers)Nature Medicine (5 papers)PLoS ONE (4 papers)Nature reviews. Cancer (4 papers)
- Partner nations
- United StatesPortugalJapan
In The Last Decade
David Lyden
102 papers receiving 21.3k citations
David Lyden's Hit Papers
Peers
Comparison fields: 5 of 153
- Cancer Research 5.7k
- Oncology 6.3k
- Genetics 2.3k
- Molecular Biology 11.9k
- Immunology and Allergy 873
Countries citing papers authored by David Lyden
This map shows the geographic impact of David Lyden's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Lyden with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Lyden more than expected).
Fields of papers citing papers by David Lyden
This network shows the impact of papers produced by David Lyden. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Lyden. The network helps show where David Lyden may publish in the future.
Co-authors
The 25 scholars most cited alongside David Lyden, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 104 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | Extracellular Vesicles in Cancer: Cell-to-Cell Mediators of Metastasis Hit paper breakdown → | 2016 | 1514 |
| 2 | Impaired recruitment of bone-marrow–derived endothelial and hematopoietic precursor cells blocks tumor angiogenesis and growth Hit paper breakdown → | 2001 | 1480 |
| 3 | Pre-metastatic niches: organ-specific homes for metastases Hit paper breakdown → | 2017 | 1387 |
| 4 | Recruitment of Stem and Progenitor Cells from the Bone Marrow Niche Requires MMP-9 Mediated Release of Kit-Ligand Hit paper breakdown → | 2002 | 1368 |
| 5 | Therapeutic stem and progenitor cell transplantation for organ vascularization and regeneration Hit paper breakdown → | 2003 | 1287 |
| 6 | Tumor Response to Radiotherapy Regulated by Endothelial Cell Apoptosis Hit paper breakdown → | 2003 | 1240 |
| 7 | The metastatic niche: adapting the foreign soil Hit paper breakdown → | 2009 | 984 |
| 8 | Exosome-Mediated Metastasis: Communication from a Distance Hit paper breakdown → | 2019 | 983 |
| 9 | The perivascular niche regulates breast tumour dormancy Hit paper breakdown → | 2013 | 856 |
| 10 | Id1 and Id3 are required for neurogenesis, angiogenesis and vascularization of tumour xenografts Hit paper breakdown → | 1999 | 736 |
| 11 | CD133 expression is not restricted to stem cells, and both CD133+ and CD133– metastatic colon cancer cells initiate tumors Hit paper breakdown → | 2008 | 730 |
| 12 | Inductive angiocrine signals from sinusoidal endothelium are required for liver regeneration Hit paper breakdown → | 2010 | 618 |
| 13 | Chemokine-mediated interaction of hematopoietic progenitors with the bone marrow vascular niche is required for thrombopoiesis Hit paper breakdown → | 2003 | 583 |
| 14 | Vascular Endothelial Growth Factor and Angiopoietin-1 Stimulate Postnatal Hematopoiesis by Recruitment of Vasculogenic and Hematopoietic Stem Cells Hit paper breakdown → | 2001 | 532 |
| 15 | Vascular and haematopoietic stem cells: novel targets for anti-angiogenesis therapy? Hit paper breakdown → | 2002 | 525 |
| 16 | The secreted factors responsible for pre-metastatic niche formation: Old sayings and new thoughts Hit paper breakdown → | 2011 | 520 |
| 17 | 2002 | 490 | |
| 18 | 2006 | 483 | |
| 19 | Asymmetric-flow field-flow fractionation technology for exomere and small extracellular vesicle separation and characterization Hit paper breakdown → | 2019 | 379 |
| 20 | 2004 | 302 |
About David Lyden
David Lyden is a scholar working on Molecular Biology, Oncology, Cancer Research, Genetics and Immunology, having authored 104 papers that have together received 21.6k indexed citations. Recurring topics across this work include Extracellular vesicles in disease (26 papers), Cancer Cells and Metastasis (18 papers), Angiogenesis and VEGF in Cancer (17 papers), MicroRNA in disease regulation (10 papers), Mesenchymal stem cell research (7 papers), Glioma Diagnosis and Treatment (7 papers), Immune cells in cancer (6 papers) and Cell Adhesion Molecules Research (6 papers). The work is most often cited by research in Cancer Research (5.7k citations), Oncology (6.3k citations), Genetics (2.3k citations), Molecular Biology (11.9k citations) and Immunology and Allergy (873 citations). David Lyden has collaborated with scholars based in United States, Portugal and Japan. Frequent co-authors include Shahin Rafii, Bethan Psaila, Héctor Peinado, Rosandra N. Kaplan, Beate Heissig, Robert Benezra, Koichi Hattori, Sérgio Dias, Haiying Zhang and Neil R. Hackett. Their work appears in journals such as Cancer Research, Pediatric Blood & Cancer, Nature Medicine, PLoS ONE and Nature reviews. Cancer.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.