Daniel H. Geschwind
Impact in
- Developmental Neuroscience top 0.01%
- Neurogenesis and neuroplasticity mechanisms
- Cognitive Neuroscience top 0.05%
- Autism Spectrum Disorder Research
Papers in
-
- Congenital heart defects research 36
- Genetics 136
- Genetics and Neurodevelopmental Disorders 92
- Genomic variations and chromosomal abnormalities 38
- Co-authors
- Brett S. Abrahams (12 shared papers)Steve Horvath (17 shared papers)Giovanni Coppola (80 shared papers)Pat Levitt (5 shared papers)Donna M. Werling (6 shared papers)Neelroop Parikshak (22 shared papers)Michael C. Oldham (14 shared papers)Harley I. Kornblum (20 shared papers)
- Journals
- Neuron (26 papers)Proceedings of the National Academy of Sciences (17 papers)Journal of Neuroscience (15 papers)Molecular Psychiatry (14 papers)Cell (14 papers)
- Partner nations
- United StatesUnited KingdomGermany
In The Last Decade
Daniel H. Geschwind
423 papers receiving 50.5k citations
Daniel H. Geschwind's Hit Papers
Peers
Comparison fields: 5 of 197
- Developmental Neuroscience 5.0k
- Cognitive Neuroscience 12.9k
- Biological Psychiatry 1.2k
- Cellular and Molecular Neuroscience 8.8k
- Neurology 3.8k
Countries citing papers authored by Daniel H. Geschwind
This map shows the geographic impact of Daniel H. Geschwind's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Daniel H. Geschwind with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Daniel H. Geschwind more than expected).
Fields of papers citing papers by Daniel H. Geschwind
This network shows the impact of papers produced by Daniel H. Geschwind. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Daniel H. Geschwind. The network helps show where Daniel H. Geschwind may publish in the future.
Co-authors
The 25 scholars most cited alongside Daniel H. Geschwind, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 430 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | Dentate Granule Cell Neurogenesis Is Increased by Seizures and Contributes to Aberrant Network Reorganization in the Adult Rat Hippocampus Hit paper breakdown → | 1997 | 1531 |
| 2 | Cancerous stem cells can arise from pediatric brain tumors Hit paper breakdown → | 2003 | 1430 |
| 3 | Transcriptomic analysis of autistic brain reveals convergent molecular pathology Hit paper breakdown → | 2011 | 1362 |
| 4 | Advances in autism genetics: on the threshold of a new neurobiology Hit paper breakdown → | 2008 | 1303 |
| 5 | Functional cortical neurons and astrocytes from human pluripotent stem cells in 3D culture Hit paper breakdown → | 2015 | 1111 |
| 6 | The Central Nervous System and the Gut Microbiome Hit paper breakdown → | 2016 | 1074 |
| 7 | Autism spectrum disorders: developmental disconnection syndromes Hit paper breakdown → | 2007 | 1051 |
| 8 | Neuroprotective effects of brain-derived neurotrophic factor in rodent and primate models of Alzheimer's disease Hit paper breakdown → | 2009 | 845 |
| 9 | Sex differences in autism spectrum disorders Hit paper breakdown → | 2013 | 834 |
| 10 | Absence of CNTNAP2 Leads to Epilepsy, Neuronal Migration Abnormalities, and Core Autism-Related Deficits Hit paper breakdown → | 2011 | 814 |
| 11 | Selenium Drives a Transcriptional Adaptive Program to Block Ferroptosis and Treat Stroke Hit paper breakdown → | 2019 | 804 |
| 12 | Integrative Functional Genomic Analyses Implicate Specific Molecular Pathways and Circuits in Autism Hit paper breakdown → | 2013 | 697 |
| 13 | Linkage, Association, and Gene-Expression Analyses Identify CNTNAP2 as an Autism-Susceptibility Gene Hit paper breakdown → | 2008 | 605 |
| 14 | Functional organization of the transcriptome in human brain Hit paper breakdown → | 2008 | 555 |
| 15 | Advancing the understanding of autism disease mechanisms through genetics Hit paper breakdown → | 2016 | 553 |
| 16 | Evidence for α-synuclein prions causing multiple system atrophy in humans with parkinsonism Hit paper breakdown → | 2015 | 550 |
| 17 | 2006 | 490 | |
| 18 | 2011 | 468 | |
| 19 | A Highly Conserved Program of Neuronal Microexons Is Misregulated in Autistic Brains Hit paper breakdown → | 2014 | 460 |
| 20 | 2008 | 459 |
About Daniel H. Geschwind
Daniel H. Geschwind is a scholar working on Molecular Biology, Genetics, Cognitive Neuroscience, Cellular and Molecular Neuroscience and Physiology, having authored 430 papers that have together received 51.3k indexed citations. Recurring topics across this work include Genetics and Neurodevelopmental Disorders (92 papers), Autism Spectrum Disorder Research (83 papers), Neurogenesis and neuroplasticity mechanisms (42 papers), Alzheimer's disease research and treatments (40 papers), Genomic variations and chromosomal abnormalities (38 papers), Congenital heart defects research (36 papers), Genetic Neurodegenerative Diseases (27 papers) and Neuroinflammation and Neurodegeneration Mechanisms (26 papers). The work is most often cited by research in Developmental Neuroscience (5.0k citations), Cognitive Neuroscience (12.9k citations), Biological Psychiatry (1.2k citations), Cellular and Molecular Neuroscience (8.8k citations) and Neurology (3.8k citations). Daniel H. Geschwind has collaborated with scholars based in United States, United Kingdom and Germany. Frequent co-authors include Brett S. Abrahams, Steve Horvath, Giovanni Coppola, Pat Levitt, Donna M. Werling, Neelroop Parikshak, Michael C. Oldham, Harley I. Kornblum, Rita M. Cantor and Jennifer K. Lowe. Their work appears in journals such as Neuron, Proceedings of the National Academy of Sciences, Journal of Neuroscience, Molecular Psychiatry and Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.