Michael E. Greenberg

113.1k citations
272 papers · 99.7k · 41 hit papers · h-index 138

Impact in

Papers in

    • Neuroscience and Neuropharmacology Research 59
    • Nerve injury and regeneration 18
    • Axon Guidance and Neuronal Signaling 15
    • Genomics and Chromatin Dynamics 43
    • Protein Kinase Regulation and GTPase Signaling 38

Michael E. Greenberg

269 papers receiving 97k citations

Michael E. Greenberg's Hit Papers

Sleep Loss Can Cause Death through Accumulation of Reactive Oxygen Species in the Gut 2020 · 408 citations
4080+8+16Years since publication1k2k3k

Peers

Michael E. Greenberg
Comparison fields: 5 of 176
  • Developmental Neuroscience 6k
  • Cellular and Molecular Neuroscience 21.5k
  • Molecular Biology 39.1k
  • Neurology 4.3k
  • Aging 869
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Citations per field
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Citations per year

Countries citing papers authored by Michael E. Greenberg

Since Specialization
Citations

This map shows the geographic impact of Michael E. Greenberg's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael E. Greenberg with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael E. Greenberg more than expected).

Fields of papers citing papers by Michael E. Greenberg

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael E. Greenberg. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael E. Greenberg. The network helps show where Michael E. Greenberg may publish in the future.

Co-authors

The 25 scholars most cited alongside Michael E. Greenberg, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Michael E. Greenberg Line = papers co-authored together Michael E. Greenberg links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 272 papers — load more, or switch the sort, to bring in the rest.

#Work
1
Akt Promotes Cell Survival by Phosphorylating and Inhibiting a Forkhead Transcription Factor
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19995667
2
Akt Phosphorylation of BAD Couples Survival Signals to the Cell-Intrinsic Death Machinery
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19974949
3
Opposing Effects of ERK and JNK-p38 MAP Kinases on Apoptosis
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19954902
4
Microglia Sculpt Postnatal Neural Circuits in an Activity and Complement-Dependent Manner
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20123236
5
Stress-Dependent Regulation of FOXO Transcription Factors by the SIRT1 Deacetylase
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20042859
6
Stimulation of 3T3 cells induces transcription of the c-fos proto-oncogene
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19842822
7
Regulation of Neuronal Survival by the Serine-Threonine Protein Kinase Akt
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19972202
8
The regulation and function of c-fos and other immediate early genes in the nervous system
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19902167
9
Widespread transcription at neuronal activity-regulated enhancers
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20101873
10
CREB: A Stimulus-Induced Transcription Factor Activated by A Diverse Array of Extracellular Signals
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19991850
11
Cell Survival Promoted by the Ras-MAPK Signaling Pathway by Transcription-Dependent and -Independent Mechanisms
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19991684
12
A brain-specific microRNA regulates dendritic spine development
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20061549
13
Ca2+ Influx Regulates BDNF Transcription by a CREB Family Transcription Factor-Dependent Mechanism
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19981377
14
Huntingtin Acts in the Nucleus to Induce Apoptosis but Death Does Not Correlate with the Formation of Intranuclear Inclusions
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19981361
15
CREB: a Ca 2+ -Regulated Transcription Factor Phosphorylated by Calmodulin-Dependent Kinases
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19911349
16
Calcium Signaling in Neurons: Molecular Mechanisms and Cellular Consequences
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19951257
17
Exercise Induces Hippocampal BDNF through a PGC-1α/FNDC5 Pathway
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20131196
18
Coupling of the RAS-MAPK Pathway to Gene Activation by RSK2, a Growth Factor-Regulated CREB Kinase
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19961107
19
Transcription-dependent and -independent control of neuronal survival by the PI3K–Akt signaling pathway
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20011057
20
Derepression of BDNF Transcription Involves Calcium-Dependent Phosphorylation of MeCP2
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20031055

About Michael E. Greenberg

Michael E. Greenberg is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology, Developmental Neuroscience, Genetics and Cognitive Neuroscience, having authored 272 papers that have together received 99.7k indexed citations. Recurring topics across this work include Neuroscience and Neuropharmacology Research (59 papers), Genomics and Chromatin Dynamics (43 papers), Protein Kinase Regulation and GTPase Signaling (38 papers), Genetics and Neurodevelopmental Disorders (32 papers), Neurogenesis and neuroplasticity mechanisms (21 papers), Nerve injury and regeneration (18 papers), Axon Guidance and Neuronal Signaling (15 papers) and Memory and Neural Mechanisms (10 papers). The work is most often cited by research in Developmental Neuroscience (6k citations), Cellular and Molecular Neuroscience (21.5k citations), Molecular Biology (39.1k citations), Neurology (4.3k citations) and Aging (869 citations). Michael E. Greenberg has collaborated with scholars based in United States, Canada and Germany. Frequent co-authors include Edward Benjamin Ziff, Morgan Sheng, Sandeep Robert Datta, Anne Brunet, Azad Bonni, Linda S. Hu, David D. Ginty, Adam J. Shaywitz, Zhengui Xia and Anirvan Ghosh. Their work appears in journals such as Neuron, Science, Cell, Proceedings of the National Academy of Sciences and Nature.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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