Beáta Barsvári
Impact in
- Behavioral Neuroscience top 5%
- Stress Responses and Cortisol
- Biological Psychiatry top 10%
- Tryptophan and brain disorders
Papers in
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- Stress Responses and Cortisol 5
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- Neuroendocrine regulation and behavior 4
- Co-authors
- József Haller (8 shared papers)Éva Mikics (5 shared papers)Boglárka Barsy (3 shared papers)S. Goldberg (1 shared paper)L.V. Panlilio (1 shared paper)István Barna (1 shared paper)Sevil Yaşar (1 shared paper)Máté Tóth (3 shared papers)
- Journals
- Biological Psychiatry (1 paper)Psychoneuroendocrinology (1 paper)Neuropharmacology (1 paper)Psychopharmacology (1 paper)Hormones and Behavior (1 paper)
- Partner nations
- HungaryUnited States
In The Last Decade
Beáta Barsvári
8 papers receiving 420 citations
Peers
Comparison fields: 5 of 62
- Behavioral Neuroscience 151
- Biological Psychiatry 35
- Cellular and Molecular Neuroscience 158
- Pharmacology 135
- Social Psychology 178
Countries citing papers authored by Beáta Barsvári
This map shows the geographic impact of Beáta Barsvári's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Beáta Barsvári with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Beáta Barsvári more than expected).
Fields of papers citing papers by Beáta Barsvári
This network shows the impact of papers produced by Beáta Barsvári. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Beáta Barsvári. The network helps show where Beáta Barsvári may publish in the future.
Co-authors
The 18 scholars most cited alongside Beáta Barsvári, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2009 | 138 | |
| 2 | 2005 | 112 | |
| 3 | 2012 | 53 | |
| 4 | 2015 | 29 | |
| 5 | 2007 | 29 | |
| 6 | 2005 | 27 | |
| 7 | 2014 | 25 | |
| 8 | 2010 | 13 |
About Beáta Barsvári
Beáta Barsvári is a scholar working on Behavioral Neuroscience, Social Psychology, Experimental and Cognitive Psychology, Cognitive Neuroscience and Pharmacology, having authored 8 papers that have together received 426 indexed citations. Recurring topics across this work include Stress Responses and Cortisol (5 papers), Neuroendocrine regulation and behavior (4 papers), Memory and Neural Mechanisms (2 papers), Evolutionary Psychology and Human Behavior (2 papers), Cannabis and Cannabinoid Research (1 paper), Child and Adolescent Psychosocial and Emotional Development (1 paper), Hormonal and reproductive studies (1 paper) and Anxiety, Depression, Psychometrics, Treatment, Cognitive Processes (1 paper). The work is most often cited by research in Behavioral Neuroscience (151 citations), Biological Psychiatry (35 citations), Cellular and Molecular Neuroscience (158 citations), Pharmacology (135 citations) and Social Psychology (178 citations). Beáta Barsvári has collaborated with scholars based in Hungary and United States. Frequent co-authors include József Haller, Éva Mikics, Boglárka Barsy, S. Goldberg, L.V. Panlilio, István Barna, Sevil Yaşar, Máté Tóth, László Bíró and Áron Tulogdi. Their work appears in journals such as Biological Psychiatry, Psychoneuroendocrinology, Neuropharmacology, Psychopharmacology and Hormones and Behavior.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.