William E. Wixted

696 citations
10 papers · 574 · h-index 9

Impact in

    • Genomics, phytochemicals, and oxidative stress
    • RNA Interference and Gene Delivery
    • Glutathione Transferases and Polymorphisms
    • Protein Degradation and Inhibitors
    • Apelin-related biomedical research

Papers in

William E. Wixted

10 papers receiving 560 citations

Peers

William E. Wixted
Comparison fields: 5 of 71
  • Molecular Biology 378
  • Pharmacology 84
  • Organic Chemistry 90
  • Endocrinology, Diabetes and Metabolism 42
  • Biological Psychiatry 6
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Citations per field
00.5×5×10×14.7×
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Citations per year

Countries citing papers authored by William E. Wixted

Since Specialization
Citations

This map shows the geographic impact of William E. Wixted's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by William E. Wixted with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites William E. Wixted more than expected).

Fields of papers citing papers by William E. Wixted

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by William E. Wixted. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by William E. Wixted. The network helps show where William E. Wixted may publish in the future.

Co-authors

The 25 scholars most cited alongside William E. Wixted, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with William E. Wixted Line = papers co-authored together William E. Wixted links everyone, so they are left out of the graph.

All Works

10 of 10 papers shown
#Work
1 2016251
2 201592
3 200457
4 200546
5 200345
6 202128
7 201718
8 200417
9 201516
10 20094

About William E. Wixted

William E. Wixted is a scholar working on Endocrinology, Diabetes and Metabolism, Pharmacology, Molecular Biology, Microbiology and Clinical Biochemistry, having authored 10 papers that have together received 574 indexed citations. Recurring topics across this work include Genomics, phytochemicals, and oxidative stress (4 papers), Cardiovascular, Neuropeptides, and Oxidative Stress Research (2 papers), Hormonal Regulation and Hypertension (2 papers), Synthesis and Biological Evaluation (2 papers), Apelin-related biomedical research (2 papers), RNA Interference and Gene Delivery (1 paper), Nitrogen and Sulfur Effects on Brassica (1 paper) and Antimicrobial Peptides and Activities (1 paper). The work is most often cited by research in Molecular Biology (378 citations), Pharmacology (84 citations), Organic Chemistry (90 citations), Endocrinology, Diabetes and Metabolism (42 citations) and Biological Psychiatry (6 citations). William E. Wixted has collaborated with scholars based in United States, United Kingdom and France. Frequent co-authors include James J. Foley, Peter T. Buckley, Henry M. Sarau, Christopher L. Herold, David J. Behm, Stephen A. Douglas, Nigel J. Gooderham, Timothy W. Gant, Paul A. Wilson and Stephen A. Hughes. Their work appears in journals such as British Journal of Pharmacology, Journal of Medicinal Chemistry, Journal of Pharmacology and Experimental Therapeutics, Toxicology in Vitro and Nucleic Acids Research.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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