Thomas Son

519 citations
8 papers · 110 · h-index 4

Impact in

Papers in

    • HER2/EGFR in Cancer Research 2
    • CAR-T cell therapy research 2
    • Cancer Immunotherapy and Biomarkers 2
    • Cancer Treatment and Pharmacology 1
    • Receptor Mechanisms and Signaling 2

Thomas Son

7 papers receiving 108 citations

Peers

Thomas Son
Comparison fields: 5 of 30
  • Cellular and Molecular Neuroscience 58
  • Biological Psychiatry 4
  • Endocrinology, Diabetes and Metabolism 19
  • Pharmacology 18
  • Molecular Biology 68
Replace Charlotte Avet with:
Charlotte Avet Canada
Elisabetta Amadori Italy
Louis Dwomoh United Kingdom
Luke Jehu United Kingdom
Anne Kinhult Ståhlbom Sweden
Yuchen Xu China
Tara Skelly United States
Portia Chipendo United States
Juan C. Troncoso United States
Cristiana Artiola Italy
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Citations per field
00.5×
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Citations per year

Countries citing papers authored by Thomas Son

Since Specialization
Citations

This map shows the geographic impact of Thomas Son's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas Son with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas Son more than expected).

Fields of papers citing papers by Thomas Son

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas Son. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas Son. The network helps show where Thomas Son may publish in the future.

Co-authors

The 25 scholars most cited alongside Thomas Son, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Thomas Son Line = papers co-authored together Thomas Son links everyone, so they are left out of the graph.

All Works

8 of 8 papers shown
#Work
1 200960
2 200722
3 200418
4 20243
5 20203
6 20162
7 20241
8 20221

About Thomas Son

Thomas Son is a scholar working on Oncology, Molecular Biology, Radiology, Nuclear Medicine and Imaging, Immunology and Cellular and Molecular Neuroscience, having authored 8 papers that have together received 110 indexed citations. Recurring topics across this work include Immunotherapy and Immune Responses (3 papers), Prostate Cancer Treatment and Research (2 papers), HER2/EGFR in Cancer Research (2 papers), CAR-T cell therapy research (2 papers), Receptor Mechanisms and Signaling (2 papers), Monoclonal and Polyclonal Antibodies Research (2 papers), Cancer Immunotherapy and Biomarkers (2 papers) and Cancer Treatment and Pharmacology (1 paper). The work is most often cited by research in Cellular and Molecular Neuroscience (58 citations), Biological Psychiatry (4 citations), Endocrinology, Diabetes and Metabolism (19 citations), Pharmacology (18 citations) and Molecular Biology (68 citations). Thomas Son has collaborated with scholars based in United States and Sweden. Frequent co-authors include Robert E. Davis, Stefania Risso Bradley, Hans H. Schiffer, Tracy A. Spalding, Ethan S. Burstein, Ali Tabatabaei, Thomas Ott, Derek Nguyen, Linda Ohrmund and Krista McFarland. Their work appears in journals such as Cancer Research, Journal of Pharmacology and Experimental Therapeutics, The Journal of Steroid Biochemistry and Molecular Biology and Neuropharmacology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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