Thomas E. Lloyd
Impact in
- Neurology top 0.5%
- Amyotrophic Lateral Sclerosis Research
- Cellular and Molecular Neuroscience top 0.5%
- Hereditary Neurological Disorders
- Genetic Neurodegenerative Diseases
Papers in
- Neurology 35
- Amyotrophic Lateral Sclerosis Research 25
-
- Hereditary Neurological Disorders 18
- Genetic Neurodegenerative Diseases 12
- Neurobiology and Insect Physiology Research 9
- Co-authors
- Andrew Lee Mammen (30 shared papers)Hugo Jozef Bellen (10 shared papers)Lisa Christopher‐Stine (24 shared papers)Mark N. Wu (8 shared papers)J. Paul Taylor (4 shared papers)Yi Zhou (2 shared papers)Iago Pinal‐Fernandez (20 shared papers)Julie J. Paik (13 shared papers)
- Journals
- Neurology (8 papers)Annals of the Rheumatic Diseases (7 papers)Cell (6 papers)Annals of Neurology (6 papers)Neuron (6 papers)
- Partner nations
- United StatesUnited KingdomSpain
In The Last Decade
Thomas E. Lloyd
131 papers receiving 7.9k citations
Thomas E. Lloyd's Hit Papers
Peers
Comparison fields: 5 of 128
- Neurology 2.0k
- Cellular and Molecular Neuroscience 2.0k
- Genetics 1.1k
- Cell Biology 1.4k
- Aging 127
Countries citing papers authored by Thomas E. Lloyd
This map shows the geographic impact of Thomas E. Lloyd's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas E. Lloyd with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas E. Lloyd more than expected).
Fields of papers citing papers by Thomas E. Lloyd
This network shows the impact of papers produced by Thomas E. Lloyd. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas E. Lloyd. The network helps show where Thomas E. Lloyd may publish in the future.
Co-authors
The 25 scholars most cited alongside Thomas E. Lloyd, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 135 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | The C9orf72 repeat expansion disrupts nucleocytoplasmic transport Hit paper breakdown → | 2015 | 795 |
| 2 | 2002 | 387 | |
| 3 | 2018 | 302 | |
| 4 | 2014 | 291 | |
| 5 | 2017 | 275 | |
| 6 | 2002 | 275 | |
| 7 | 2012 | 216 | |
| 8 | 2014 | 192 | |
| 9 | 2012 | 182 | |
| 10 | 1999 | 175 | |
| 11 | 2019 | 173 | |
| 12 | 2011 | 172 | |
| 13 | 2018 | 171 | |
| 14 | 2016 | 153 | |
| 15 | 2013 | 145 | |
| 16 | 2016 | 144 | |
| 17 | 2000 | 144 | |
| 18 | 2020 | 130 | |
| 19 | 2020 | 129 | |
| 20 | 2021 | 124 |
About Thomas E. Lloyd
Thomas E. Lloyd is a scholar working on Neurology, Cellular and Molecular Neuroscience, Epidemiology, Genetics and Cell Biology, having authored 135 papers that have together received 8.1k indexed citations. Recurring topics across this work include Inflammatory Myopathies and Dermatomyositis (51 papers), Amyotrophic Lateral Sclerosis Research (25 papers), Neurogenetic and Muscular Disorders Research (21 papers), Hereditary Neurological Disorders (18 papers), Cellular transport and secretion (13 papers), Genetic Neurodegenerative Diseases (12 papers), Neurobiology and Insect Physiology Research (9 papers) and Muscle Physiology and Disorders (8 papers). The work is most often cited by research in Neurology (2.0k citations), Cellular and Molecular Neuroscience (2.0k citations), Genetics (1.1k citations), Cell Biology (1.4k citations) and Aging (127 citations). Thomas E. Lloyd has collaborated with scholars based in United States, United Kingdom and Spain. Frequent co-authors include Andrew Lee Mammen, Hugo Jozef Bellen, Lisa Christopher‐Stine, Mark N. Wu, J. Paul Taylor, Yi Zhou, Iago Pinal‐Fernandez, Julie J. Paik, Katherine S. Pak and Giuseppa Pennetta. Their work appears in journals such as Neurology, Annals of the Rheumatic Diseases, Cell, Annals of Neurology and Neuron.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.