T L Deckwerth

2.0k citations
10 papers · 1.7k · h-index 10

Impact in

Papers in

    • Cell death mechanisms and regulation 2
    • Nicotinic Acetylcholine Receptors Study 2
    • Angiogenesis and VEGF in Cancer 1
    • Ion channel regulation and function 1
    • Neuroscience and Neuropharmacology Research 6
    • Nerve injury and regeneration 2
    • Photoreceptor and optogenetics research 2

T L Deckwerth

10 papers receiving 1.7k citations

Peers

T L Deckwerth
Comparison fields: 5 of 95
  • Cellular and Molecular Neuroscience 882
  • Developmental Neuroscience 187
  • Molecular Biology 1.2k
  • Cell Biology 221
  • Neurology 83
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Citations per field
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Citations per year

Countries citing papers authored by T L Deckwerth

Since Specialization
Citations

This map shows the geographic impact of T L Deckwerth's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by T L Deckwerth with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites T L Deckwerth more than expected).

Fields of papers citing papers by T L Deckwerth

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by T L Deckwerth. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by T L Deckwerth. The network helps show where T L Deckwerth may publish in the future.

Co-authors

The 21 scholars most cited alongside T L Deckwerth, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with T L Deckwerth Line = papers co-authored together T L Deckwerth links everyone, so they are left out of the graph.

All Works

10 of 10 papers shown
#Work
1 1993481
2 1988299
3 1993284
4 1996198
5 1995142
6
Suppression of developmental retinal cell death but not of photoreceptor degeneration in Bax-deficient mice.
1998108
7 198967
8 199559
9 200337
10 199030

About T L Deckwerth

T L Deckwerth is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience, Pharmacology, Genetics and Oncology, having authored 10 papers that have together received 1.7k indexed citations. Recurring topics across this work include Neuroscience and Neuropharmacology Research (6 papers), Nerve injury and regeneration (2 papers), Cell death mechanisms and regulation (2 papers), Photoreceptor and optogenetics research (2 papers), Nicotinic Acetylcholine Receptors Study (2 papers), Angiogenesis and VEGF in Cancer (1 paper), Coagulation, Bradykinin, Polyphosphates, and Angioedema (1 paper) and Ion channel regulation and function (1 paper). The work is most often cited by research in Cellular and Molecular Neuroscience (882 citations), Developmental Neuroscience (187 citations), Molecular Biology (1.2k citations), Cell Biology (221 citations) and Neurology (83 citations). T L Deckwerth has collaborated with scholars based in United States and Germany. Frequent co-authors include Eugene M. Johnson, Reinhard Jahn, Peter R. Maycox, Johannes Hell, Patricia A. Lampe, Brenda D. Shivers, Mohanish Deshmukh, John P. Vasilakos, César Sanz-Rodrı́guez and EM Johnson. Their work appears in journals such as The Journal of Cell Biology, Journal of Biological Chemistry, Journal of Neuroscience, Journal of the Neurological Sciences and Annual Review of Neuroscience.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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