Raphael Escaig

600 citations
7 papers · 145 · h-index 5

Impact in

  • Hematology top 10%
    • Platelet Disorders and Treatments
    • Blood Coagulation and Thrombosis Mechanisms
    • Venous Thromboembolism Diagnosis and Management

Papers in

Raphael Escaig

6 papers receiving 145 citations

Peers

Raphael Escaig
Comparison fields: 5 of 47
  • Hematology 66
  • Internal Medicine 19
  • Immunology 35
  • Immunology and Allergy 5
  • Oncology 21
Replace Daphne N. Pariser with:
Daphne N. Pariser United States
Patrick Thomas United Kingdom
Iván Murrieta-Álvarez Mexico
Joana Batista United Kingdom
Sue Chandraratne Germany
Antonio Lugini Italy
Marie Favier France
Sara Gamba Italy
SJ Koppelman
Jean-Michel Micléa France
Raphael Escaig relative to Daphne N. Pariser United States Daphne N. Pariser's profile →
Citations per field
00.5×
Daphne N. Pariser · 1×
Citations per year

Countries citing papers authored by Raphael Escaig

Since Specialization
Citations

This map shows the geographic impact of Raphael Escaig's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Raphael Escaig with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Raphael Escaig more than expected).

Fields of papers citing papers by Raphael Escaig

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Raphael Escaig. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Raphael Escaig. The network helps show where Raphael Escaig may publish in the future.

Co-authors

The 25 scholars most cited alongside Raphael Escaig, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Raphael Escaig Line = papers co-authored together Raphael Escaig links everyone, so they are left out of the graph.

All Works

7 of 7 papers shown
#Work
1 202260
2 202337
3 202232
4 20239
5 20216
6 20221
7 20260

About Raphael Escaig

Raphael Escaig is a scholar working on Hematology, Pulmonary and Respiratory Medicine, Immunology, Molecular Biology and Genetics, having authored 7 papers that have together received 145 indexed citations. Recurring topics across this work include Platelet Disorders and Treatments (4 papers), Blood Coagulation and Thrombosis Mechanisms (2 papers), Blood properties and coagulation (2 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (2 papers), Cell Adhesion Molecules Research (1 paper), Chronic Myeloid Leukemia Treatments (1 paper), Myeloproliferative Neoplasms: Diagnosis and Treatment (1 paper) and Atherosclerosis and Cardiovascular Diseases (1 paper). The work is most often cited by research in Hematology (66 citations), Internal Medicine (19 citations), Immunology (35 citations), Immunology and Allergy (5 citations) and Oncology (21 citations). Raphael Escaig has collaborated with scholars based in Germany, Hungary and Austria. Frequent co-authors include Leo Nicolai, Rainer Kaiser, Johanna Erber, Steffen Maßberg, Michael Lorenz, Florian Gaertner, Afra Anjum, Konstantin Stark, Anna Titova and Stefan Kääb. Their work appears in journals such as Blood, EMBO Molecular Medicine, Haematologica and Frontiers in Cardiovascular Medicine.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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