Peter Sabbatini

3.8k citations
29 papers · 3.1k · 1 hit paper · h-index 20

Impact in

  • Oncology top 2%
    • Cancer-related Molecular Pathways
    • CAR-T cell therapy research
    • Cancer Research and Treatments

Papers in

    • RNA Interference and Gene Delivery 4
    • Ubiquitin and proteasome pathways 3
    • Cancer-related Molecular Pathways 10

Peter Sabbatini

28 papers receiving 3.0k citations

Peter Sabbatini's Hit Papers

The adenovirus E1A proteins induce apoptosis, which is inhibited by the E1B 19-kDa and Bcl-2 proteins. 1992 · 566 citations
5660+11+22Years since publication100200300400500

Peers

Peter Sabbatini
Comparison fields: 5 of 94
  • Oncology 1.4k
  • Biotechnology 369
  • Genetics 930
  • Molecular Biology 2.0k
  • Immunology 555
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Thomas Chittenden United States
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Citations per field
00.5×1.7×
Thomas Chittenden · 1×
Citations per year

Countries citing papers authored by Peter Sabbatini

Since Specialization
Citations

This map shows the geographic impact of Peter Sabbatini's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Peter Sabbatini with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Peter Sabbatini more than expected).

Fields of papers citing papers by Peter Sabbatini

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Peter Sabbatini. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Peter Sabbatini. The network helps show where Peter Sabbatini may publish in the future.

Co-authors

The 25 scholars most cited alongside Peter Sabbatini, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Peter Sabbatini Line = papers co-authored together Peter Sabbatini links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 29 papers — load more, or switch the sort, to bring in the rest.

#Work
1
The adenovirus E1A proteins induce apoptosis, which is inhibited by the E1B 19-kDa and Bcl-2 proteins.
Hit paper breakdown →
1992566
2 1992315
3 1996295
4 1997243
5 1991216
6 1995198
7 1992171
8 1996165
9 1995163
10 1999121
11 200986
12 199581
13 201981
14 201569
15 200962
16
Interleukin 1 beta converting enzyme-like proteases are essential for p53-mediated transcriptionally dependent apoptosis.
199757
17 201455
18 200241
19 201830
20 200827

About Peter Sabbatini

Peter Sabbatini is a scholar working on Molecular Biology, Oncology, Genetics, Genetics and Hematology, having authored 29 papers that have together received 3.1k indexed citations. Recurring topics across this work include Cancer-related Molecular Pathways (10 papers), Virus-based gene therapy research (7 papers), Multiple Myeloma Research and Treatments (5 papers), Chronic Lymphocytic Leukemia Research (5 papers), RNA Interference and Gene Delivery (4 papers), Monoclonal and Polyclonal Antibodies Research (4 papers), Growth Hormone and Insulin-like Growth Factors (4 papers) and Ubiquitin and proteasome pathways (3 papers). The work is most often cited by research in Oncology (1.4k citations), Biotechnology (369 citations), Genetics (930 citations), Molecular Biology (2.0k citations) and Immunology (555 citations). Peter Sabbatini has collaborated with scholars based in United States, Netherlands and Germany. Frequent co-authors include Eileen White, Lakshmi Rao, Jeonghoon Han, David M. Hockenbery, S J Korsmeyer, William S.M. Wold, L R Gooding, Frank McCormick, George C. Prendergast and Daitoku Sakamuro. Their work appears in journals such as Blood, Molecular and Cellular Biology, Clinical Cancer Research, Genes & Development and Journal of Virology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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