P. Dévay

418 citations
10 papers · 367 · h-index 8

Impact in

Papers in

    • Nicotinic Acetylcholine Receptors Study 5
    • Receptor Mechanisms and Signaling 2
    • Ion channel regulation and function 1
    • Neuroscience and Neuropharmacology Research 3
    • Neurobiology and Insect Physiology Research 3
    • Photoreceptor and optogenetics research 2

P. Dévay

10 papers receiving 359 citations

Peers

P. Dévay
Comparison fields: 5 of 59
  • Cellular and Molecular Neuroscience 186
  • Behavioral Neuroscience 34
  • Endocrine and Autonomic Systems 38
  • Social Psychology 92
  • Molecular Biology 238
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Citations per field
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Citations per year

Countries citing papers authored by P. Dévay

Since Specialization
Citations

This map shows the geographic impact of P. Dévay's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by P. Dévay with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites P. Dévay more than expected).

Fields of papers citing papers by P. Dévay

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by P. Dévay. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by P. Dévay. The network helps show where P. Dévay may publish in the future.

Co-authors

The 15 scholars most cited alongside P. Dévay, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with P. Dévay Line = papers co-authored together P. Dévay links everyone, so they are left out of the graph.

All Works

10 of 10 papers shown
#Work
1 1991145
2 1999114
3 199431
4 199926
5 198817
6 198612
7 198911
8
Cyclic AMP-induced phosphorylation of 27.5-kDa protein(s) in larval brains of normal and memory-mutant Drosophila melanogaster.
19877
9 19943
10
Protein phosphorylation in dunce memory-mutant Drosophila.
19861

About P. Dévay

P. Dévay is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience, Cell Biology, Pharmacology and Social Psychology, having authored 10 papers that have together received 367 indexed citations. Recurring topics across this work include Nicotinic Acetylcholine Receptors Study (5 papers), Neuroscience and Neuropharmacology Research (3 papers), Neurobiology and Insect Physiology Research (3 papers), Photoreceptor and optogenetics research (2 papers), Receptor Mechanisms and Signaling (2 papers), Amyotrophic Lateral Sclerosis Research (1 paper), Muscle metabolism and nutrition (1 paper) and Ion channel regulation and function (1 paper). The work is most often cited by research in Cellular and Molecular Neuroscience (186 citations), Behavioral Neuroscience (34 citations), Endocrine and Autonomic Systems (38 citations), Social Psychology (92 citations) and Molecular Biology (238 citations). P. Dévay has collaborated with scholars based in United States, Hungary and Netherlands. Frequent co-authors include Arjen B. Brussaard, Lorna W. Role, David Colman, Karel S. Kits, Xueping Qu, P. Friedrich, Daniel S. McGehee, Cheng‐Rong Yu, A.B. Oestreicher and W.H. Gispen. Their work appears in journals such as Nature Neuroscience, The Journal of Physiology, Journal of Neurogenetics, Developmental Biology and Science.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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