M.P. Botella
Impact in
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- Genetics and Neurodevelopmental Disorders
- Genomics and Rare Diseases
- Genomic variations and chromosomal abnormalities
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- Autism Spectrum Disorder Research
Papers in
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- RNA regulation and disease 2
- Epigenetics and DNA Methylation 1
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- Folate and B Vitamins Research 1
- Co-authors
- Javier Santoyo‐López (1 shared paper)Gemma Aznar‐Laín (1 shared paper)Blanca Gener (2 shared papers)Ivon Cuscó (1 shared paper)Benjamín Rodríguez‐Santiago (1 shared paper)Miguel Del Campo (1 shared paper)Guillermo Antiñolo (1 shared paper)Luis A. Pérez‐Jurado (1 shared paper)
- Journals
- Frontiers in Genetics (1 paper)Human Mutation (1 paper)Molecular Autism (1 paper)American Journal of Medical Genetics (1 paper)Clinical Dysmorphology (1 paper)
- Partner nations
- SpainUnited KingdomPoland
In The Last Decade
M.P. Botella
6 papers receiving 137 citations
Peers
Comparison fields: 5 of 39
- Genetics 65
- Cognitive Neuroscience 32
- Molecular Biology 69
- Biological Psychiatry 2
- Developmental Neuroscience 3
Countries citing papers authored by M.P. Botella
This map shows the geographic impact of M.P. Botella's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by M.P. Botella with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites M.P. Botella more than expected).
Fields of papers citing papers by M.P. Botella
This network shows the impact of papers produced by M.P. Botella. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by M.P. Botella. The network helps show where M.P. Botella may publish in the future.
Co-authors
The 25 scholars most cited alongside M.P. Botella, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2015 | 94 | |
| 2 | 2018 | 14 | |
| 3 | 2013 | 13 | |
| 4 | 2005 | 12 | |
| 5 | 2000 | 4 | |
| 6 | 2009 | 3 | |
| 7 | 2010 | 0 |
About M.P. Botella
M.P. Botella is a scholar working on Molecular Biology, Rheumatology, Genetics, Neurology and Pediatrics, Perinatology and Child Health, having authored 7 papers that have together received 140 indexed citations. Recurring topics across this work include RNA regulation and disease (2 papers), Autism Spectrum Disorder Research (1 paper), Folate and B Vitamins Research (1 paper), Epigenetics and DNA Methylation (1 paper), Autoimmune Neurological Disorders and Treatments (1 paper), Genetic Syndromes and Imprinting (1 paper), Metabolism and Genetic Disorders (1 paper) and Prenatal Substance Exposure Effects (1 paper). The work is most often cited by research in Genetics (65 citations), Cognitive Neuroscience (32 citations), Molecular Biology (69 citations), Biological Psychiatry (2 citations) and Developmental Neuroscience (3 citations). M.P. Botella has collaborated with scholars based in Spain, United Kingdom and Poland. Frequent co-authors include Javier Santoyo‐López, Gemma Aznar‐Laín, Blanca Gener, Ivon Cuscó, Benjamín Rodríguez‐Santiago, Miguel Del Campo, Guillermo Antiñolo, Luis A. Pérez‐Jurado, Marta Codina‐Solà and Elisabeth Gabau. Their work appears in journals such as Frontiers in Genetics, Human Mutation, Molecular Autism, American Journal of Medical Genetics and Clinical Dysmorphology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.