Michael E. Haws
Impact in
- Rheumatology top 10%
- Bone and Dental Protein Studies
Papers in
-
- FOXO transcription factor regulation 1
- Protein Kinase Regulation and GTPase Signaling 1
- Phosphodiesterase function and regulation 1
- Co-authors
- Sawsan Youssef (1 shared paper)Lawrence Steinman (1 shared paper)Raymond A. Sobel (1 shared paper)Eun Mi Hur (1 shared paper)Craig M. Powell (3 shared papers)Haley E. Speed (1 shared paper)Amelia J. Eisch (1 shared paper)Cory A. Blaiss (1 shared paper)
- Journals
- Toxicological Sciences (1 paper)Journal of Child Neurology (1 paper)The Journal of Pediatrics (1 paper)Genes Brain & Behavior (1 paper)The Journal of Comparative Neurology (1 paper)
- Partner nations
- United States
In The Last Decade
Michael E. Haws
6 papers receiving 391 citations
Peers
Comparison fields: 5 of 71
- Rheumatology 126
- Developmental Neuroscience 24
- Neurology 43
- Immunology 109
- Immunology and Allergy 25
Countries citing papers authored by Michael E. Haws
This map shows the geographic impact of Michael E. Haws's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael E. Haws with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael E. Haws more than expected).
Fields of papers citing papers by Michael E. Haws
This network shows the impact of papers produced by Michael E. Haws. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael E. Haws. The network helps show where Michael E. Haws may publish in the future.
Co-authors
The 25 scholars most cited alongside Michael E. Haws, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2006 | 274 | |
| 2 | 2013 | 46 | |
| 3 | 2011 | 43 | |
| 4 | 2018 | 16 | |
| 5 | 2011 | 10 | |
| 6 | 2016 | 10 |
About Michael E. Haws
Michael E. Haws is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience, Genetics, Pharmacology and Pediatrics, Perinatology and Child Health, having authored 6 papers that have together received 399 indexed citations. Recurring topics across this work include Genetics and Neurodevelopmental Disorders (2 papers), Cholinesterase and Neurodegenerative Diseases (1 paper), FOXO transcription factor regulation (1 paper), Environmental Toxicology and Ecotoxicology (1 paper), Pesticide Exposure and Toxicity (1 paper), Protein Kinase Regulation and GTPase Signaling (1 paper), Phosphodiesterase function and regulation (1 paper) and Fetal and Pediatric Neurological Disorders (1 paper). The work is most often cited by research in Rheumatology (126 citations), Developmental Neuroscience (24 citations), Neurology (43 citations), Immunology (109 citations) and Immunology and Allergy (25 citations). Michael E. Haws has collaborated with scholars based in United States. Frequent co-authors include Sawsan Youssef, Lawrence Steinman, Raymond A. Sobel, Eun Mi Hur, Craig M. Powell, Haley E. Speed, Amelia J. Eisch, Cory A. Blaiss, Felipe Rafael Reyna Espinosa and Shannon M. Standridge. Their work appears in journals such as Toxicological Sciences, Journal of Child Neurology, The Journal of Pediatrics, Genes Brain & Behavior and The Journal of Comparative Neurology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.