Matthew E. Randolph

427 citations
11 papers · 323 · h-index 8

Impact in

    • Galectins and Cancer Biology
    • Glycosylation and Glycoproteins Research
    • Muscle Physiology and Disorders
    • Wnt/β-catenin signaling in development and cancer
    • Ubiquitin and proteasome pathways

Papers in

    • Glycosylation and Glycoproteins Research 4
    • Muscle Physiology and Disorders 2
    • RNA Research and Splicing 2
    • DNA Repair Mechanisms 1
    • Angiogenesis and VEGF in Cancer 1
    • Galectins and Cancer Biology 3

Matthew E. Randolph

10 papers receiving 319 citations

Peers

Matthew E. Randolph
Comparison fields: 5 of 46
  • Immunology 119
  • Molecular Biology 260
  • Immunology and Allergy 13
  • Cell Biology 36
  • Genetics 19
Replace Martín Enrique Rabassa with:
Martín Enrique Rabassa Argentina
Karen Strenge United States
Natalia Porębska Poland
Nawin Chanthra Japan
Adam Lambert United Kingdom
Karen Matta United States
Karen Kaucic United States
Anne Lamontagne United States
Frank Frissora United States
Christian Colin Brazil
Matthew E. Randolph relative to Martín Enrique Rabassa Argentina Martín Enrique Rabassa's profile →
Citations per field
00.5×3.8×
Martín Enrique Rabassa · 1×
Citations per year

Countries citing papers authored by Matthew E. Randolph

Since Specialization
Citations

This map shows the geographic impact of Matthew E. Randolph's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew E. Randolph with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew E. Randolph more than expected).

Fields of papers citing papers by Matthew E. Randolph

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew E. Randolph. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew E. Randolph. The network helps show where Matthew E. Randolph may publish in the future.

Co-authors

The 25 scholars most cited alongside Matthew E. Randolph, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Matthew E. Randolph Line = papers co-authored together Matthew E. Randolph links everyone, so they are left out of the graph.

All Works

11 of 11 papers shown
#Work
1 200777
2 200972
3 201049
4 201542
5 201527
6 200819
7 201414
8 201714
9 20175
10 20244
11 20250

About Matthew E. Randolph

Matthew E. Randolph is a scholar working on Molecular Biology, Immunology, Genetics, Cellular and Molecular Neuroscience and Oncology, having authored 11 papers that have together received 323 indexed citations. Recurring topics across this work include Glycosylation and Glycoproteins Research (4 papers), Galectins and Cancer Biology (3 papers), Neurogenetic and Muscular Disorders Research (2 papers), Muscle Physiology and Disorders (2 papers), RNA Research and Splicing (2 papers), Sarcoma Diagnosis and Treatment (1 paper), DNA Repair Mechanisms (1 paper) and Angiogenesis and VEGF in Cancer (1 paper). The work is most often cited by research in Immunology (119 citations), Molecular Biology (260 citations), Immunology and Allergy (13 citations), Cell Biology (36 citations) and Genetics (19 citations). Matthew E. Randolph has collaborated with scholars based in United States, Netherlands and Slovakia. Frequent co-authors include Hua‐Bei Guo, Michael Pierce, Grace K. Pavlath, Tamás Nagy, Katherine E. Vest, Hyo‐Jung Choo, Brittany L. Phillips, Gerardo Álvarez-Manilla, Kyle Harris and Kelley W. Moremen. Their work appears in journals such as Journal of Biological Chemistry, iScience, Frontiers in Aging Neuroscience, The Journal of Physiology and FEBS Letters.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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