Massimo Loda

81.0k citations
470 papers · 40.5k · 14 hit papers · h-index 98

Impact in

Papers in

Massimo Loda

460 papers receiving 39.7k citations

Massimo Loda's Hit Papers

Artificial intelligence entering the pathology arena in oncology: current applications and future perspectives 2025 · 26 citations
260+9+19Years since publication50010001.5k

Peers

Massimo Loda
Comparison fields: 5 of 194
  • Cancer Research 9.0k
  • Oncology 10.1k
  • Molecular Biology 20.2k
  • Pulmonary and Respiratory Medicine 8.1k
  • Pathology and Forensic Medicine 4.2k
Replace Nikolaus Schultz with:
Nikolaus Schultz United States
William R. Sellers United States
William L. Gerald United States
Masaki Mori Japan
Matt van de Rijn United States
Olli Kallioniemi Finland
Erik Larsson Sweden
Paul S. Meltzer United States
Gad Getz United States
Carlos L. Arteaga United States
Massimo Loda relative to Nikolaus Schultz United States Nikolaus Schultz's profile →
Citations per field
00.5×1.5×
Nikolaus Schultz · 1×
Citations per year

Countries citing papers authored by Massimo Loda

Since Specialization
Citations

This map shows the geographic impact of Massimo Loda's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Massimo Loda with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Massimo Loda more than expected).

Fields of papers citing papers by Massimo Loda

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Massimo Loda. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Massimo Loda. The network helps show where Massimo Loda may publish in the future.

Co-authors

The 25 scholars most cited alongside Massimo Loda, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Massimo Loda Line = papers co-authored together Massimo Loda links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 470 papers — load more, or switch the sort, to bring in the rest.

#Work
1
Classification of human lung carcinomas by mRNA expression profiling reveals distinct adenocarcinoma subclasses
Hit paper breakdown →
20011954
2
Gene expression correlates of clinical prostate cancer behavior
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20021833
3
Multiclass cancer diagnosis using tumor gene expression signatures
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20011461
4
Methylation of the hMLH1 promoter correlates with lack of expression of hMLH1 in sporadic colon tumors and mismatch repair-defective human tumor cell lines.
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19971203
5
Increased proteasome-dependent degradation of the cyclin-dependent kinase inhibitor p27 in aggressive colorectal carcinomas
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1997911
6
mTOR inhibition reverses Akt-dependent prostate intraepithelial neoplasia through regulation of apoptotic and HIF-1-dependent pathways
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2004781
7
Rb1 and Trp53 cooperate to suppress prostate cancer lineage plasticity, metastasis, and antiandrogen resistance
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2017696
8
Control of TH2 polarization by the chemokine monocyte chemoattractant protein-1
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2000688
9
CpG island methylator phenotype, microsatellite instability, BRAF mutation and clinical outcome in colon cancer
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2008596
10
Prostate stem cell antigen: A cell surface marker overexpressed in prostate cancer
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1998559
11
Essential roles of PI(3)K–p110β in cell growth, metabolism and tumorigenesis
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2008559
12
Inappropriate expression of hepcidin is associated with iron refractory anemia: implications for the anemia of chronic disease
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2002514
13
Loss of PTEN expression in paraffin-embedded primary prostate cancer correlates with high Gleason score and advanced stage.
1999499
14 2000492
15 2000463
16 1995456
17 2010451
18
Lipids and cancer: Emerging roles in pathogenesis, diagnosis and therapeutic intervention
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2020430
19 2005403
20
Prognostic role of the cyclin-dependent kinase inhibitor p27 in non-small cell lung cancer.
1997381

About Massimo Loda

Massimo Loda is a scholar working on Molecular Biology, Pulmonary and Respiratory Medicine, Cancer Research, Oncology and Surgery, having authored 470 papers that have together received 40.5k indexed citations. Recurring topics across this work include Prostate Cancer Treatment and Research (108 papers), Cancer, Lipids, and Metabolism (76 papers), Cancer-related Molecular Pathways (44 papers), Prostate Cancer Diagnosis and Treatment (36 papers), Ubiquitin and proteasome pathways (33 papers), Epigenetics and DNA Methylation (33 papers), Genetic factors in colorectal cancer (28 papers) and Cancer Genomics and Diagnostics (26 papers). The work is most often cited by research in Cancer Research (9.0k citations), Oncology (10.1k citations), Molecular Biology (20.2k citations), Pulmonary and Respiratory Medicine (8.1k citations) and Pathology and Forensic Medicine (4.2k citations). Massimo Loda has collaborated with scholars based in United States, Italy and United Kingdom. Frequent co-authors include Sabina Signoretti, William R. Sellers, Todd R. Golub, Shuji Ogino, Charles S. Fuchs, Eric S. Lander, Phillip G. Febbo, Christine Ladd, Giorgia Zadra and Michele Pagano. Their work appears in journals such as Cancer Research, Journal of Clinical Oncology, Clinical Cancer Research, Molecular Cancer Research and Cancer Epidemiology Biomarkers & Prevention.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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