Katja Weckmann
Impact in
- Biological Psychiatry top 5%
- Tryptophan and brain disorders
- Behavioral Neuroscience top 10%
- Stress Responses and Cortisol
Papers in
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- Treatment of Major Depression 4
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- Tryptophan and brain disorders 4
- Co-authors
- John M. Asara (4 shared papers)Christoph W. Turck (4 shared papers)Christiana Labermaier (3 shared papers)Marianne B. Müller (2 shared papers)Parvana Hajieva (2 shared papers)Christian Behl (2 shared papers)Michaela D. Filiou (2 shared papers)Kathryn S. Lilley (2 shared papers)
- Journals
- Scientific Reports (2 papers)Translational Psychiatry (1 paper)Cells (1 paper)PubMed (1 paper)Electronic Theses of LMU Munich (Ludwig-Maximilians-Universität München) (1 paper)
- Partner nations
- GermanyUnited StatesUnited Kingdom
In The Last Decade
Katja Weckmann
6 papers receiving 206 citations
Peers
Comparison fields: 5 of 64
- Biological Psychiatry 94
- Behavioral Neuroscience 32
- Pharmacology 54
- Developmental Neuroscience 8
- Physiology 9
Countries citing papers authored by Katja Weckmann
This map shows the geographic impact of Katja Weckmann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Katja Weckmann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Katja Weckmann more than expected).
Fields of papers citing papers by Katja Weckmann
This network shows the impact of papers produced by Katja Weckmann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Katja Weckmann. The network helps show where Katja Weckmann may publish in the future.
Co-authors
The 16 scholars most cited alongside Katja Weckmann, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2017 | 71 | |
| 2 | 2014 | 43 | |
| 3 | 2020 | 43 | |
| 4 | 2018 | 27 | |
| 5 | 2018 | 21 | |
| 6 | 2017 | 1 |
About Katja Weckmann
Katja Weckmann is a scholar working on Pharmacology, Biological Psychiatry, Molecular Biology, Epidemiology and Physiology, having authored 6 papers that have together received 206 indexed citations. Recurring topics across this work include Tryptophan and brain disorders (4 papers), Treatment of Major Depression (4 papers), Autophagy in Disease and Therapy (2 papers), Diet and metabolism studies (2 papers), Pharmacological Receptor Mechanisms and Effects (1 paper), Mitochondrial Function and Pathology (1 paper), Endoplasmic Reticulum Stress and Disease (1 paper) and Neuroscience and Neuropharmacology Research (1 paper). The work is most often cited by research in Biological Psychiatry (94 citations), Behavioral Neuroscience (32 citations), Pharmacology (54 citations), Developmental Neuroscience (8 citations) and Physiology (9 citations). Katja Weckmann has collaborated with scholars based in Germany, United States and United Kingdom. Frequent co-authors include John M. Asara, Christoph W. Turck, Christiana Labermaier, Marianne B. Müller, Parvana Hajieva, Christian Behl, Michaela D. Filiou, Kathryn S. Lilley, Frederik Dethloff and Giuseppina Maccarrone. Their work appears in journals such as Scientific Reports, Translational Psychiatry, Cells, PubMed and Electronic Theses of LMU Munich (Ludwig-Maximilians-Universität München).
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.