Junko Oshima
Impact in
- Aging top 0.2%
- Physiology top 0.5%
- Alzheimer's disease research and treatments
- Telomeres, Telomerase, and Senescence
Papers in
-
- DNA Repair Mechanisms 79
- Nuclear Structure and Function 52
- Genomics and Chromatin Dynamics 33
- RNA Research and Splicing 12
- Mitochondrial Function and Pathology 12
- Physiology 29
- Telomeres, Telomerase, and Senescence 21
- Co-authors
- George M. Martin (56 shared papers)Gerard D. Schellenberg (10 shared papers)Ellen M. Wijsman (5 shared papers)Ying‐Hui Fu (4 shared papers)Chang-En Yu (6 shared papers)Matthew D. Gray (7 shared papers)Fuki M. Hisama (21 shared papers)Lawrence A. Loeb (4 shared papers)
- Journals
- Aging Cell (6 papers)Human Genetics (5 papers)Genomics (5 papers)Human Mutation (5 papers)Human Molecular Genetics (4 papers)
- Partner nations
- United StatesJapanGermany
In The Last Decade
Junko Oshima
125 papers receiving 8.6k citations
Junko Oshima's Hit Papers
Peers
Comparison fields: 5 of 133
- Aging 580
- Physiology 2.9k
- Molecular Biology 6.5k
- Cancer Research 1.1k
- Cell Biology 643
Countries citing papers authored by Junko Oshima
This map shows the geographic impact of Junko Oshima's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Junko Oshima with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Junko Oshima more than expected).
Fields of papers citing papers by Junko Oshima
This network shows the impact of papers produced by Junko Oshima. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Junko Oshima. The network helps show where Junko Oshima may publish in the future.
Co-authors
The 25 scholars most cited alongside Junko Oshima, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
Showing the 20 most-cited of 126 papers — load more, or switch the sort, to bring in the rest.
| # | Work | ||
|---|---|---|---|
| 1 | Candidate Gene for the Chromosome 1 Familial Alzheimer's Disease Locus Hit paper breakdown → | 1995 | 1964 |
| 2 | Positional Cloning of the Werner's Syndrome Gene Hit paper breakdown → | 1996 | 1368 |
| 3 | 1997 | 482 | |
| 4 | 2003 | 322 | |
| 5 | 1998 | 234 | |
| 6 | 2016 | 193 | |
| 7 | 1998 | 180 | |
| 8 | 2000 | 172 | |
| 9 | 2003 | 147 | |
| 10 | 1997 | 139 | |
| 11 | 1998 | 124 | |
| 12 | 2003 | 120 | |
| 13 | Mutations in the consensus helicase domains of the Werner syndrome gene. Werner's Syndrome Collaborative Group. | 1997 | 118 |
| 14 | 1999 | 113 | |
| 15 | 2005 | 111 | |
| 16 | 2017 | 111 | |
| 17 | 2008 | 104 | |
| 18 | 2007 | 103 | |
| 19 | 2000 | 101 | |
| 20 | 1996 | 85 |
About Junko Oshima
Junko Oshima is a scholar working on Molecular Biology, Physiology, Plant Science, Cancer Research and Genetics, having authored 126 papers that have together received 8.8k indexed citations. Recurring topics across this work include DNA Repair Mechanisms (79 papers), Nuclear Structure and Function (52 papers), Genomics and Chromatin Dynamics (33 papers), Telomeres, Telomerase, and Senescence (21 papers), Chromosomal and Genetic Variations (13 papers), RNA Research and Splicing (12 papers), Carcinogens and Genotoxicity Assessment (12 papers) and Mitochondrial Function and Pathology (12 papers). The work is most often cited by research in Aging (580 citations), Physiology (2.9k citations), Molecular Biology (6.5k citations), Cancer Research (1.1k citations) and Cell Biology (643 citations). Junko Oshima has collaborated with scholars based in United States, Japan and Germany. Frequent co-authors include George M. Martin, Gerard D. Schellenberg, Ellen M. Wijsman, Ying‐Hui Fu, Chang-En Yu, Matthew D. Gray, Fuki M. Hisama, Lawrence A. Loeb, Jun Nakura and John Mulligan. Their work appears in journals such as Aging Cell, Human Genetics, Genomics, Human Mutation and Human Molecular Genetics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.