John R. Vane

494 papers receiving 61.4k citations

John R. Vane's Hit Papers

The mechanism of action of aspirin 2003 · 943 citations
9430+16+32Years since publication50010001.5k2.0k

Peers

John R. Vane
Comparison fields: 5 of 178
  • Biochemistry 12.9k
  • Pharmacology 26.3k
  • Physiology 18.5k
  • Cardiology and Cardiovascular Medicine 12.5k
  • Pharmacology 5.0k
Replace Salvador Moncada with:
Salvador Moncada United Kingdom
Bengt Samuelsson Sweden
John A. Oates United States
Richard Palmer United Kingdom
Philip Needleman United States
Charles N. Serhan United States
John L. Wallace Canada
Friedrich C. Luft Germany
David G. Harrison United States
Jason D. Morrow United States
John R. Vane relative to Salvador Moncada United Kingdom Salvador Moncada's profile →
Citations per field
00.5×1.5×1.8×
Salvador Moncada · 1×
Citations per year

Countries citing papers authored by John R. Vane

Since Specialization
Citations

This map shows the geographic impact of John R. Vane's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by John R. Vane with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites John R. Vane more than expected).

Fields of papers citing papers by John R. Vane

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by John R. Vane. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by John R. Vane. The network helps show where John R. Vane may publish in the future.

Co-authors

The 25 scholars most cited alongside John R. Vane, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with John R. Vane Line = papers co-authored together John R. Vane links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 506 papers — load more, or switch the sort, to bring in the rest.

#Work
1
Inhibition of Prostaglandin Synthesis as a Mechanism of Action for Aspirin-like Drugs
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19717410
2
An enzyme isolated from arteries transforms prostaglandin endoperoxides to an unstable substance that inhibits platelet aggregation
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19763103
3
CYCLOOXYGENASES 1 AND 2
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19982416
4
Regulatory Functions of the Vascular Endothelium
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19901624
5
Selectivity of nonsteroidal antiinflammatory drugs as inhibitors of constitutive and inducible cyclooxygenase.
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19931452
6
Pharmacology and endogenous roles of prostaglandin endoperoxides, thromboxane A2, and prostacyclin.
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19781414
7
Nonsteroid drug selectivities for cyclo-oxygenase-1 rather than cyclo-oxygenase-2 are associated with human gastrointestinal toxicity: A full in vitro analysis
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19991319
8
Pressor effects of circulating endothelin are limited by its removal in the pulmonary circulation and by the release of prostacyclin and endothelium-derived relaxing factor.
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19881248
9
Arachidonic Acid Metabolites and the Interactions between Platelets and Blood-Vessel Walls
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1979990
10
Inducible isoforms of cyclooxygenase and nitric-oxide synthase in inflammation.
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1994957
11
The mechanism of action of aspirin
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2003943
12
Arterial walls are protected against deposition of platelet thrombi by a substance (prostaglandin X) which they make from prostaglandin endoperoxides
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1976906
13
Arterial walls generate from prostaglandin endoperoxides a substance (prostaglandin X) which relaxes strips of mesenteric and coeliac arteries and inhibits platelet aggregation
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1976859
14
Indomethacin and Aspirin abolish Prostaglandin Release from the Spleen
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1971844
15
Prostaglandins: Their Disappearance from and Release into the Circulation
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1967816
16
Release of Additional Factors in Anaphylaxis and its Antagonism by Anti-inflammatory Drugs
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1969805
17
HUMAN ARTERIAL AND VENOUS TISSUES GENERATE PROSTACYCLIN (PROSTAGLANDIN X), A POTENT INHIBITOR OF PLATELET AGGREGATION
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1977739
18
New insights into the mode of action of anti-inflammatory drugs
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1995722
19
A SENSITIVE METHOD FOR THE ASSAY OF 5‐HYDROXYTRYPTAMINE
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1957721
20
Inhibition of Prostaglandin Synthetase in Brain explains the Anti-pyretic Activity of Paracetamol (4-Acetamidophenol)
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1972672

About John R. Vane

John R. Vane is a scholar working on Physiology, Pharmacology, Biochemistry, Cardiology and Cardiovascular Medicine and Molecular Biology, having authored 506 papers that have together received 69.4k indexed citations. Recurring topics across this work include Nitric Oxide and Endothelin Effects (178 papers), Inflammatory mediators and NSAID effects (171 papers), Eicosanoids and Hypertension Pharmacology (147 papers), Antiplatelet Therapy and Cardiovascular Diseases (48 papers), Renin-Angiotensin System Studies (46 papers), Receptor Mechanisms and Signaling (42 papers), Neuropeptides and Animal Physiology (37 papers) and Cardiac Ischemia and Reperfusion (33 papers). The work is most often cited by research in Biochemistry (12.9k citations), Pharmacology (26.3k citations), Physiology (18.5k citations), Cardiology and Cardiovascular Medicine (12.5k citations) and Pharmacology (5.0k citations). John R. Vane has collaborated with scholars based in United Kingdom, United States and Brazil. Frequent co-authors include Salvador Moncada, R M Botting, Christoph Thiemermann, Ryszard J. Gryglewski, S. H. Ferreira, Jane A. Mitchell, S Bunting, Regina M. Botting, Timothy D. Warner and Y.S. Bakhle. Their work appears in journals such as British Journal of Pharmacology, Nature, Proceedings of the National Academy of Sciences, Journal of Cardiovascular Pharmacology and Prostaglandins.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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