Bart Staels

862 papers receiving 92.4k citations

Bart Staels's Hit Papers

Data-driven cluster analysis identifies distinct types of metabolic dysfunction-associated steatotic liver disease 2024 · 90 citations
900+5+11Years since publication10002.0k3.0k4.0k

Peers

Bart Staels
Comparison fields: 5 of 194
  • Endocrinology, Diabetes and Metabolism 13.0k
  • Endocrine and Autonomic Systems 5.3k
  • Biochemistry 5.7k
  • Physiology 18.0k
  • Epidemiology 21.1k
Replace Johan H. Auwerx with:
Johan H. Auwerx Switzerland
Ronald Evans United States
Gerald I. Shulman United States
Frank J. Gonzalez United States
Takashi Kadowaki Japan
Gökhan S. Hotamışlıgil United States
Jerrold M. Olefsky United States
C Ronald Kahn United States
Christopher K. Glass United States
Bruce M. Spiegelman United States
Bart Staels relative to Johan H. Auwerx Switzerland Johan H. Auwerx's profile →
Citations per field
00.5×1.5×1.8×
Johan H. Auwerx · 1×
Citations per year

Countries citing papers authored by Bart Staels

Since Specialization
Citations

This map shows the geographic impact of Bart Staels's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Bart Staels with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Bart Staels more than expected).

Fields of papers citing papers by Bart Staels

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Bart Staels. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Bart Staels. The network helps show where Bart Staels may publish in the future.

Co-authors

The 25 scholars most cited alongside Bart Staels, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Bart Staels Line = papers co-authored together Bart Staels links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 876 papers — load more, or switch the sort, to bring in the rest.

#Work
1
Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
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20164660
2
Mechanism of Action of Fibrates on Lipid and Lipoprotein Metabolism
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19981474
3
Role of Bile Acids and Bile Acid Receptors in Metabolic Regulation
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20091392
4
Molecular mechanism of PPARα action and its impact on lipid metabolism, inflammation and fibrosis in non-alcoholic fatty liver disease
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20141337
5
PPARγ Activation Primes Human Monocytes into Alternative M2 Macrophages with Anti-inflammatory Properties
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20071176
6
Role of the peroxisome proliferator-activated receptor (PPAR) in mediating the effects of fibrates and fatty acids on gene expression
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19961093
7
The Organization, Promoter Analysis, and Expression of the Human PPARγ Gene
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19971043
8
PPARalpha and PPARgamma activators direct a distinct tissue‐specific transcriptional response via a PPRE in the lipoprotein lipase gene.
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19961032
9
PPAR-α and PPAR-γ activators induce cholesterol removal from human macrophage foam cells through stimulation of the ABCA1 pathway
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20011031
10
Activation of human aortic smooth-muscle cells is inhibited by PPARα but not by PPARγ activators
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19981016
11
Transient increase in obese gene expression after food intake or insulin administration
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1995982
12
Peroxisome Proliferator-activated Receptor α Negatively Regulates the Vascular Inflammatory Gene Response by Negative Cross-talk with Transcription Factors NF-κB and AP-1
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1999967
13
The peroxisome proliferator activated receptors (PPARs) and their effects on lipid metabolism and adipocyte differentiation
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1996936
14
Elafibranor, an Agonist of the Peroxisome Proliferator−Activated Receptor−α and −δ, Induces Resolution of Nonalcoholic Steatohepatitis Without Fibrosis Worsening
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2016911
15
International Union of Pharmacology. LXI. Peroxisome Proliferator-Activated Receptors
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2006847
16
Peroxisome proliferator-activated receptors (PPARs): Nuclear receptors at the crossroads between lipid metabolism and inflammation
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2000824
17
Activation of Proliferator-activated Receptors α and γ Induces Apoptosis of Human Monocyte-derived Macrophages
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1998817
18
Bile Acid Control of Metabolism and Inflammation in Obesity, Type 2 Diabetes, Dyslipidemia, and Nonalcoholic Fatty Liver Disease
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2017815
19
PPAR control of metabolism and cardiovascular functions
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2021711
20
Protective Role of Interleukin-10 in Atherosclerosis
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1999701

About Bart Staels

Bart Staels is a scholar working on Molecular Biology, Physiology, Endocrinology, Diabetes and Metabolism, Epidemiology and Oncology, having authored 876 papers that have together received 95.0k indexed citations. Recurring topics across this work include Peroxisome Proliferator-Activated Receptors (324 papers), Adipose Tissue and Metabolism (123 papers), Drug Transport and Resistance Mechanisms (112 papers), Cholesterol and Lipid Metabolism (109 papers), Metabolism, Diabetes, and Cancer (93 papers), Liver Disease Diagnosis and Treatment (88 papers), Adipokines, Inflammation, and Metabolic Diseases (64 papers) and Cancer, Lipids, and Metabolism (49 papers). The work is most often cited by research in Endocrinology, Diabetes and Metabolism (13.0k citations), Endocrine and Autonomic Systems (5.3k citations), Biochemistry (5.7k citations), Physiology (18.0k citations) and Epidemiology (21.1k citations). Bart Staels has collaborated with scholars based in France, United States and Belgium. Frequent co-authors include Johan H. Auwerx, Jean‐Charles Fruchart, Giulia Chinetti, Philippe Lefèbvre, Kristina Schoonjans, Folkert Kuipers, Hélène Duez, Inès Pineda‐Torra, Bertrand Cariou and Jean‐Charles Fruchart. Their work appears in journals such as Journal of Biological Chemistry, Arteriosclerosis Thrombosis and Vascular Biology, Atherosclerosis, Journal of Hepatology and Diabetes.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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