Felipe Were

892 citations
10 papers · 545 · h-index 10

Impact in

    • Epigenetics and DNA Methylation
    • Genomics and Chromatin Dynamics
    • Signaling Pathways in Disease
    • Cancer-related gene regulation
    • RNA modifications and cancer

Papers in

Felipe Were

10 papers receiving 538 citations

Peers

Felipe Were
Comparison fields: 5 of 76
  • Aging 10
  • Molecular Biology 376
  • Cancer Research 67
  • Immunology 88
  • Cell Biology 61
Replace Tadaomi Takenawa with:
Tadaomi Takenawa Japan
Patrick Beaudette Germany
Ken-ichi Takita Japan
Charles Morin Canada
Miao Ding China
Michael J. Thomenius United States
Fatimah A. Nahhas United States
Stela S. Palii United States
Xinnong Jiang China
Dorien Haesen Belgium
Felipe Were relative to Tadaomi Takenawa Japan Tadaomi Takenawa's profile →
Citations per field
00.5×7.7×
Tadaomi Takenawa · 1×
Citations per year

Countries citing papers authored by Felipe Were

Since Specialization
Citations

This map shows the geographic impact of Felipe Were's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Felipe Were with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Felipe Were more than expected).

Fields of papers citing papers by Felipe Were

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Felipe Were. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Felipe Were. The network helps show where Felipe Were may publish in the future.

Co-authors

The 25 scholars most cited alongside Felipe Were, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Felipe Were Line = papers co-authored together Felipe Were links everyone, so they are left out of the graph.

All Works

10 of 10 papers shown
#Work
1 1997134
2 202091
3 199963
4 200561
5 200755
6 202049
7 200944
8 199524
9 200614
10 201710

About Felipe Were

Felipe Were is a scholar working on Molecular Biology, Cancer Research, Urology, Cell Biology and Genetics, having authored 10 papers that have together received 545 indexed citations. Recurring topics across this work include Signaling Pathways in Disease (3 papers), Epigenetics and DNA Methylation (2 papers), Hair Growth and Disorders (2 papers), Skin and Cellular Biology Research (2 papers), Advanced Proteomics Techniques and Applications (1 paper), Genetics and Neurodevelopmental Disorders (1 paper), NF-κB Signaling Pathways (1 paper) and Animal Genetics and Reproduction (1 paper). The work is most often cited by research in Aging (10 citations), Molecular Biology (376 citations), Cancer Research (67 citations), Immunology (88 citations) and Cell Biology (61 citations). Felipe Were has collaborated with scholars based in Spain, United States and Netherlands. Frequent co-authors include Miguel Vidal, Juan Miguel Redondo, Camelia V. Marcos-Gutierrez, Rodrigo M. Maza, Arie P. Otte, Jon Schoorlemmer, David Satijn, Jesús Vázquez, Inmaculada Ortega‐Pérez and Margarita Villar. Their work appears in journals such as Nature Communications, Journal of Cell Science, Arteriosclerosis Thrombosis and Vascular Biology, Journal of Biological Chemistry and Journal of Mass Spectrometry.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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