F. Assimacopoulos

978 citations
4 papers · 781 · h-index 3

Impact in

Papers in

    • Peroxisome Proliferator-Activated Receptors 1
    • Ion channel regulation and function 1
    • Receptor Mechanisms and Signaling 1
    • Mitochondrial Function and Pathology 1

F. Assimacopoulos

4 papers receiving 735 citations

Peers

F. Assimacopoulos
Comparison fields: 5 of 71
  • Clinical Biochemistry 83
  • Endocrine and Autonomic Systems 76
  • Physiology 278
  • Aging 17
  • Biochemistry 60
Replace Walter N. Shaw with:
Walter N. Shaw United States
Nancy Kneer United States
Donald O. Allen United States
Yoshiharu Tsubamoto Japan
G van de Werve Canada
Anna Wojtczak Poland
Nenad Nešković France
David Saggerson United Kingdom
Suryaprakash Raichur Australia
L. Hue Belgium
F. Assimacopoulos relative to Walter N. Shaw United States Walter N. Shaw's profile →
Citations per field
00.5×4.3×
Walter N. Shaw · 1×
Citations per year

Countries citing papers authored by F. Assimacopoulos

Since Specialization
Citations

This map shows the geographic impact of F. Assimacopoulos's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by F. Assimacopoulos with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites F. Assimacopoulos more than expected).

Fields of papers citing papers by F. Assimacopoulos

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by F. Assimacopoulos. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by F. Assimacopoulos. The network helps show where F. Assimacopoulos may publish in the future.

Co-authors

The 14 scholars most cited alongside F. Assimacopoulos, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with F. Assimacopoulos Line = papers co-authored together F. Assimacopoulos links everyone, so they are left out of the graph.

All Works

About F. Assimacopoulos

F. Assimacopoulos is a scholar working on Molecular Biology, Physiology, Surgery, Cellular and Molecular Neuroscience and Epidemiology, having authored 4 papers that have together received 781 indexed citations. Recurring topics across this work include Peroxisome Proliferator-Activated Receptors (1 paper), Ion channel regulation and function (1 paper), Receptor Mechanisms and Signaling (1 paper), Metabolism and Genetic Disorders (1 paper), Mitochondrial Function and Pathology (1 paper), Neuroscience and Neuropharmacology Research (1 paper), Pancreatic function and diabetes (1 paper) and Liver Disease Diagnosis and Treatment (1 paper). The work is most often cited by research in Clinical Biochemistry (83 citations), Endocrine and Autonomic Systems (76 citations), Physiology (278 citations), Aging (17 citations) and Biochemistry (60 citations). F. Assimacopoulos has collaborated with scholars based in United States, Switzerland and France. Frequent co-authors include Nancy J. Hutson, J H Exton, Walter Wahli, Béatrice Desvergne, Régis Saladin, Johan Auwerx, Bart Staels, Thomas Lemberger, Manuel Vázquez‐Carrera and John H. Exton. Their work appears in journals such as Journal of Biological Chemistry and The Mouseion at the JAXlibrary (Jackson Laboratory).

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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