Éric Meyer

1.8k citations
11 papers · 1.6k · h-index 10

Impact in

Papers in

    • Cell death mechanisms and regulation 9
    • Mitochondrial Function and Pathology 1
    • Cancer-related Molecular Pathways 4
    • CAR-T cell therapy research 2

Éric Meyer

11 papers receiving 1.5k citations

Peers

Éric Meyer
Comparison fields: 5 of 91
  • Cancer Research 280
  • Molecular Biology 1.2k
  • Oncology 403
  • Immunology 316
  • Toxicology 30
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Deborah M. Finucane United States
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Citations per field
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Citations per year

Countries citing papers authored by Éric Meyer

Since Specialization
Citations

This map shows the geographic impact of Éric Meyer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Éric Meyer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Éric Meyer more than expected).

Fields of papers citing papers by Éric Meyer

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Éric Meyer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Éric Meyer. The network helps show where Éric Meyer may publish in the future.

Co-authors

The 24 scholars most cited alongside Éric Meyer, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Éric Meyer Line = papers co-authored together Éric Meyer links everyone, so they are left out of the graph.

All Works

11 of 11 papers shown
#Work
1 2001344
2 2002338
3 2001195
4
Metabolic inhibitors sensitize for CD95 (APO-1/Fas)-induced apoptosis by down-regulating Fas-associated death domain-like interleukin 1-converting enzyme inhibitory protein expression.
2000175
5 1999165
6 2000116
7 2002113
8 199973
9 200559
10 200012
11
Defective CD95-induced apoptosis in children with autoimmune lymphoproliferative syndrome (ALPS)/Canale-Smith-Syndrome (CSS) in the absence of CD95 mutations.
19992

About Éric Meyer

Éric Meyer is a scholar working on Molecular Biology, Oncology, Immunology, Epidemiology and Infectious Diseases, having authored 11 papers that have together received 1.6k indexed citations. Recurring topics across this work include Cell death mechanisms and regulation (9 papers), Cancer-related Molecular Pathways (4 papers), interferon and immune responses (3 papers), CAR-T cell therapy research (2 papers), Systemic Lupus Erythematosus Research (1 paper), Moringa oleifera research and applications (1 paper), Mitochondrial Function and Pathology (1 paper) and Immunodeficiency and Autoimmune Disorders (1 paper). The work is most often cited by research in Cancer Research (280 citations), Molecular Biology (1.2k citations), Oncology (403 citations), Immunology (316 citations) and Toxicology (30 citations). Éric Meyer has collaborated with scholars based in Germany, Switzerland and United States. Frequent co-authors include Klaus‐Michael Debatin, Simone Fulda, Barbara Dockhorn‐Dworniczak, F. van Valen, Claudia Friesen, Guido Kroemer, Santos A. Susín, Gudrun Strauß, Wolfgang Uckert and Christian Beltinger. Their work appears in journals such as Blood, Oncogene, Proceedings of the National Academy of Sciences, Cell Death and Differentiation and Biotechnology and Bioengineering.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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