David Curtis

42.2k citations
318 papers · 26.5k · 18 hit papers · h-index 60

Impact in

    • Schizophrenia research and treatment
    • Bipolar Disorder and Treatment
  • Genetics top 0.1%
    • Genetic Associations and Epidemiology
    • Genomic variations and chromosomal abnormalities
    • Genetics and Neurodevelopmental Disorders
    • Genomics and Rare Diseases

Papers in

    • Schizophrenia research and treatment 91
    • Bipolar Disorder and Treatment 49
    • Genetic Associations and Epidemiology 125
    • Genomics and Rare Diseases 14
    • Genomic variations and chromosomal abnormalities 7

David Curtis

308 papers receiving 25.6k citations

David Curtis's Hit Papers

Mapping genomic loci implicates genes and synaptic biology in schizophrenia 2022 · 1.8k citations
1.8k0+4+8Years since publication5001k1.5k

Peers

David Curtis
Comparison fields: 5 of 177
  • Psychiatry and Mental health 9.9k
  • Genetics 10.2k
  • Biological Psychiatry 304
  • Cellular and Molecular Neuroscience 1.6k
  • Clinical Psychology 1.5k
Replace Eric Strengman with:
Eric Strengman Netherlands
Eli Ayumi Stahl United States
Lisa Anne Jones United Kingdom
Masashi Ikeda Japan
Elvira Bramon United Kingdom
Ian Richard Jones United Kingdom
Ivan Nikolov United Kingdom
Thomas Folkmann Hansen Denmark
Thorgeir Elís Thorgeirsson Iceland
Bradley Todd Webb United States
David Curtis relative to Eric Strengman Netherlands Eric Strengman's profile →
Citations per field
00.5×1.7×
Eric Strengman · 1×
Citations per year

Countries citing papers authored by David Curtis

Since Specialization
Citations

This map shows the geographic impact of David Curtis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Curtis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Curtis more than expected).

Fields of papers citing papers by David Curtis

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by David Curtis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Curtis. The network helps show where David Curtis may publish in the future.

Co-authors

The 25 scholars most cited alongside David Curtis, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with David Curtis Line = papers co-authored together David Curtis links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 318 papers — load more, or switch the sort, to bring in the rest.

#Work
1
Genetic relationship between five psychiatric disorders estimated from genome-wide SNPs
Hit paper breakdown →
20131806
2
Mapping genomic loci implicates genes and synaptic biology in schizophrenia
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20221774
3
Analysis of shared heritability in common disorders of the brain
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20181477
4
Rare chromosomal deletions and duplications increase risk of schizophrenia
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20081222
5
Genome-wide association study identifies 30 loci associated with bipolar disorder
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20191100
6
Collaborative genome-wide association analysis supports a role for ANK3 and CACNA1C in bipolar disorder
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20081031
7
Genomic Relationships, Novel Loci, and Pleiotropic Mechanisms across Eight Psychiatric Disorders
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2019992
8
Genome-wide association study of more than 40,000 bipolar disorder cases provides new insights into the underlying biology
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2021977
9
Modeling Linkage Disequilibrium Increases Accuracy of Polygenic Risk Scores
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2015968
10
Monte Carlo tests for associations between disease and alleles at highly polymorphic loci
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1995848
11
Contribution of copy number variants to schizophrenia from a genome-wide study of 41,321 subjects
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2016793
12
The UK10K project identifies rare variants in health and disease
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2015790
13
Psychiatric genome-wide association study analyses implicate neuronal, immune and histone pathways
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2015629
14
Rare coding variants in ten genes confer substantial risk for schizophrenia
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2022581
15 1995545
16
Partitioning Heritability of Regulatory and Cell-Type-Specific Variants across 11 Common Diseases
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2014436
17
Phenotypic characterization of individuals with 30-40 CAG repeats in the Huntington disease (HD) gene reveals HD cases with 36 repeats and apparently normal elderly individuals with 36-39 repeats.
1996419
18 1999377
19
Rare loss-of-function variants in SETD1A are associated with schizophrenia and developmental disorders
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2016341
20 2001317

About David Curtis

David Curtis is a scholar working on Psychiatry and Mental health, Genetics, Clinical Psychology, Gastroenterology and Epidemiology, having authored 318 papers that have together received 26.5k indexed citations. Recurring topics across this work include Genetic Associations and Epidemiology (125 papers), Schizophrenia research and treatment (91 papers), Bipolar Disorder and Treatment (49 papers), Genomics and Rare Diseases (14 papers), Obsessive-Compulsive Spectrum Disorders (9 papers), Celiac Disease Research and Management (8 papers), Genomic variations and chromosomal abnormalities (7 papers) and Alzheimer's disease research and treatments (6 papers). The work is most often cited by research in Psychiatry and Mental health (9.9k citations), Genetics (10.2k citations), Biological Psychiatry (304 citations), Cellular and Molecular Neuroscience (1.6k citations) and Clinical Psychology (1.5k citations). David Curtis has collaborated with scholars based in United Kingdom, United States and Iceland. Frequent co-authors include Pak Chung Sham, Hugh Gurling, Bernard V. North, Jing Hua Zhao, Andrew McQuillin, Gursharan Kalsi, Hannes Pétursson, Jon Brynjolfsson, P. L. Murphy and Peter Brett. Their work appears in journals such as Annals of Human Genetics, Psychiatric Genetics, The British Journal of Psychiatry, The American Journal of Human Genetics and Nature Genetics.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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