Chris Conrad

987 citations
9 papers · 765 · h-index 9

Impact in

  • Neurology top 5%
    • Parkinson's Disease Mechanisms and Treatments
    • Neurological diseases and metabolism
    • Neurological disorders and treatments
  • Physiology top 5%
    • Alzheimer's disease research and treatments

Papers in

    • Alzheimer's disease research and treatments 6
    • Calcium signaling and nucleotide metabolism 1
    • Ubiquitin and proteasome pathways 2
    • Signaling Pathways in Disease 1

Chris Conrad

9 papers receiving 753 citations

Peers

Chris Conrad
Comparison fields: 5 of 64
  • Neurology 341
  • Neurology 182
  • Physiology 438
  • Cellular and Molecular Neuroscience 149
  • Physiology 34
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Chris Conrad relative to Christopher J. Holler United States Christopher J. Holler's profile →
Citations per field
00.5×2.6×
Christopher J. Holler · 1×
Citations per year

Countries citing papers authored by Chris Conrad

Since Specialization
Citations

This map shows the geographic impact of Chris Conrad's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Chris Conrad with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Chris Conrad more than expected).

Fields of papers citing papers by Chris Conrad

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Chris Conrad. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Chris Conrad. The network helps show where Chris Conrad may publish in the future.

Co-authors

The 25 scholars most cited alongside Chris Conrad, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Chris Conrad Line = papers co-authored together Chris Conrad links everyone, so they are left out of the graph.

All Works

9 of 9 papers shown
#Work
1 1997312
2 2017140
3 200296
4 200758
5 200144
6 199835
7 200434
8 199833
9 201713

About Chris Conrad

Chris Conrad is a scholar working on Physiology, Molecular Biology, Neurology, Cellular and Molecular Neuroscience and Pharmacology, having authored 9 papers that have together received 765 indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (6 papers), Parkinson's Disease Mechanisms and Treatments (4 papers), Ubiquitin and proteasome pathways (2 papers), Neurological diseases and metabolism (1 paper), Cholinesterase and Neurodegenerative Diseases (1 paper), Signaling Pathways in Disease (1 paper), Calcium signaling and nucleotide metabolism (1 paper) and Skin and Cellular Biology Research (1 paper). The work is most often cited by research in Neurology (341 citations), Neurology (182 citations), Physiology (438 citations), Cellular and Molecular Neuroscience (149 citations) and Physiology (34 citations). Chris Conrad has collaborated with scholars based in United States, Japan and Germany. Frequent co-authors include Eliezer Masliah, Leon J. Thal, Yu Xia, Peter Davies, Tsunao Saitoh, Athena Andreadis, John Q. Trojanowski, W. C. Wiederholt, Melissa Freeman and Robert Katzman. Their work appears in journals such as Journal of Neurochemistry, Blood, Autophagy, Proceedings of the National Academy of Sciences and Biochemical and Biophysical Research Communications.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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